Morena Cadaldini
- Molecular Biology
- Cardiology and Cardiovascular Medicine
- Cellular and Molecular Neuroscience
- Genetics
- Physiology
- Co-authors
- C. AngeliniElena PegoraroLibero VitielloEric P. HoffmanGian Antonio DanieliFrancesco MariMarta MiorinHarold G. Marks
- Topics
- Muscle Physiology and Disorders (4 papers)Ion channel regulation and function (2 papers)Genetic Neurodegenerative Diseases (2 papers)
- Journals
- NeurologyJournal of Neurology Neurosurgery & PsychiatryJournal of the Neurological Sciences
- Partner nations
- ItalyUnited StatesAustria
In The Last Decade
Morena Cadaldini
11 papers receiving 298 citations
Peers
Comparison fields: 5 of 53
- Molecular Biology 221
- Cardiology and Cardiovascular Medicine 87
- Cellular and Molecular Neuroscience 77
- Genetics 75
- Physiology 50
Countries citing papers authored by Morena Cadaldini
This map shows the geographic impact of Morena Cadaldini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Morena Cadaldini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Morena Cadaldini more than expected).
Fields of papers citing papers by Morena Cadaldini
This network shows the impact of papers produced by Morena Cadaldini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Morena Cadaldini. The network helps show where Morena Cadaldini may publish in the future.
Co-authorship network of co-authors of Morena Cadaldini
This figure shows the co-authorship network connecting the top 25 collaborators of Morena Cadaldini. A scholar is included among the top collaborators of Morena Cadaldini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Morena Cadaldini. Morena Cadaldini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 14 | |
| 2 | 14 | |
| 3 | 16 | |
| 4 | 7 | |
| 5 | Muscle fatigue evaluation in the follow-up of children affected by Duchenne muscular dystrophy | 6 |
| 6 | 52 | |
| 7 | 33 | |
| 8 | 5 | |
| 9 | 53 | |
| 10 | 66 | |
| 11 | 40 |
About Morena Cadaldini
Morena Cadaldini is a scholar working on Speech and Hearing, Clinical Biochemistry and Rheumatology, having authored 11 papers that have together received 306 indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (4 papers), Ion channel regulation and function (2 papers) and Genetic Neurodegenerative Diseases (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (77 citations), Cardiology and Cardiovascular Medicine (87 citations) and Molecular Biology (221 citations). Morena Cadaldini has collaborated with scholars based in Italy, United States and Austria. Frequent co-authors include C. Angelini, Elena Pegoraro, Libero Vitiello, Eric P. Hoffman, Gian Antonio Danieli, Francesco Mari, Marta Miorin, Harold G. Marks, M.P. Freda and Flávia Martinello. Their work appears in journals such as Neurology, Journal of Neurology Neurosurgery & Psychiatry and Journal of the Neurological Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.