John F. Harper
About
In The Last Decade
John F. Harper
9 papers receiving 324 citations
Peers
Comparison fields: 5 of 57
- Molecular Biology 255
- Genetics 94
- Cardiology and Cardiovascular Medicine 72
- Genetics 64
- Cellular and Molecular Neuroscience 50
Countries citing papers authored by John F. Harper
This map shows the geographic impact of John F. Harper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John F. Harper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John F. Harper more than expected).
Fields of papers citing papers by John F. Harper
This network shows the impact of papers produced by John F. Harper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John F. Harper. The network helps show where John F. Harper may publish in the future.
Co-authorship network of co-authors of John F. Harper
This figure shows the co-authorship network connecting the top 25 collaborators of John F. Harper. A scholar is included among the top collaborators of John F. Harper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John F. Harper. John F. Harper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 12 | |
| 2 | 14 | |
| 3 | 24 | |
| 4 | 1 | |
| 5 | Intragenic deletions in 21 Duchenne muscular dystrophy (DMD)/Becker muscular dystrophy (BMD) families studied with the dystrophin cDNA: location of breakpoints on HindIII and BglII exon-containing fragment maps, meiotic and mitotic origin of the mutations. | 131 |
| 6 | Congenital adrenal hypoplasia, myopathy, and glycerol kinase deficiency: molecular genetic evidence for deletions. | 78 |
| 7 | Effect of aerobic capacity on circulatory responses to static, dynamic, and combination work. | 0 |
| 8 | 61 | |
| 9 | 1 | |
| 10 | 1 | |
| 11 | 9 |
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.