Michitaka Masuda
- Molecular Biology top 5%
- Cell Biology top 1%
- Immunology and Allergy top 1%
- Cellular and Molecular Neuroscience top 5%
- Physiology top 10%
- Co-authors
- Keigi FujiwaraNaoki MochizukiMasaki OsawaYuji KamiokaTakashi OhkiYumiko KanoHirofumi OnishiAkiko Yamagishi
- Topics
- Cellular Mechanics and Interactions (8 papers)Angiogenesis and VEGF in Cancer (7 papers)Cell Adhesion Molecules Research (7 papers)
- Partner nations
- JapanUnited States
In The Last Decade
Michitaka Masuda
29 papers receiving 2.1k citations
Peers
Comparison fields: 5 of 101
- Molecular Biology 1.3k
- Cell Biology 1.0k
- Immunology and Allergy 436
- Cellular and Molecular Neuroscience 293
- Physiology 270
Countries citing papers authored by Michitaka Masuda
This map shows the geographic impact of Michitaka Masuda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michitaka Masuda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michitaka Masuda more than expected).
Fields of papers citing papers by Michitaka Masuda
This network shows the impact of papers produced by Michitaka Masuda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michitaka Masuda. The network helps show where Michitaka Masuda may publish in the future.
Co-authorship network of co-authors of Michitaka Masuda
This figure shows the co-authorship network connecting the top 25 collaborators of Michitaka Masuda. A scholar is included among the top collaborators of Michitaka Masuda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michitaka Masuda. Michitaka Masuda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 71 | |
| 2 | 41 | |
| 3 | 48 | |
| 4 | 224 | |
| 5 | 169 | |
| 6 | 5 | |
| 7 | 62 | |
| 8 | 36 | |
| 9 | 61 | |
| 10 | 141 | |
| 11 | 17 | |
| 12 | 88 | |
| 13 | Tyrosine phosphorylation of platelet endothelial cell adhesion molecule-1 (PECAM-1, CD31) in mechanically stimulated vascular endothelial cells. | 146 |
| 14 | 46 | |
| 15 | 2 | |
| 16 | 36 | |
| 17 | 1 | |
| 18 | 26 | |
| 19 | 3 | |
| 20 | 22 |
About Michitaka Masuda
Michitaka Masuda is a scholar working on Immunology and Allergy, Cell Biology and Toxicology, having authored 29 papers that have together received 2.1k indexed citations. Recurring topics across this work include Cellular Mechanics and Interactions (8 papers), Angiogenesis and VEGF in Cancer (7 papers) and Cell Adhesion Molecules Research (7 papers). The work is most often cited by research in Immunology and Allergy (436 citations), Cell Biology (1.0k citations) and Molecular Biology (1.3k citations). Michitaka Masuda has collaborated with scholars based in Japan and United States. Frequent co-authors include Keigi Fujiwara, Naoki Mochizuki, Masaki Osawa, Yuji Kamioka, Takashi Ohki, Yumiko Kano, Hirofumi Onishi, Akiko Yamagishi, Kazuo Katoh and Shigetomo Fukuhara. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Cell Biology and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.