Michal Caspi

1.6k total citations
31 papers, 1.3k citations indexed

About

Michal Caspi is a scholar working on Molecular Biology, Cell Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Michal Caspi has authored 31 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 6 papers in Cell Biology and 5 papers in Pathology and Forensic Medicine. Recurrent topics in Michal Caspi's work include Wnt/β-catenin signaling in development and cancer (13 papers), Cancer-related gene regulation (8 papers) and Genetic factors in colorectal cancer (5 papers). Michal Caspi is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (13 papers), Cancer-related gene regulation (8 papers) and Genetic factors in colorectal cancer (5 papers). Michal Caspi collaborates with scholars based in Israel, United States and France. Michal Caspi's co-authors include Rina Rosin‐Arbesfeld, Orly Reiner, Alona Zilberberg, Tamar Sapir, Michael Elbaum, Sharon G. Wolf, Jamel Chelly, Fiona Francis, Robert Atlas and Jan R. De Mey and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Molecular Biology and Molecular and Cellular Biology.

In The Last Decade

Michal Caspi

30 papers receiving 1.2k citations

Peers

Michal Caspi

GENET

ONCOL

Sachiko Kamakura Japan

Shin‐ichiro Hiraga Japan

Emily M. Hatch United States

Catherine L. Keck United States

Steven C. Pruitt United States

Nicolas Rama France

Shalom Guy Slutsky Israel

Pierre Close Belgium

Alexandre A. S. F. Raposo Portugal

Yongsu Jeong South Korea

Sachiko Kamakura Japan

Michal Caspi

1.2k ×1.4

338 ×0.8

131 ×0.7

113 ×0.8

GENET

194 ×1.4

ONCOL

Citations per year, relative to Michal Caspi Michal Caspi (= 1×) peers Sachiko Kamakura

Countries citing papers authored by Michal Caspi

Since Specialization

Citations

This map shows the geographic impact of Michal Caspi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michal Caspi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michal Caspi more than expected).

Fields of papers citing papers by Michal Caspi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michal Caspi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michal Caspi. The network helps show where Michal Caspi may publish in the future.

Co-authorship network of co-authors of Michal Caspi

This figure shows the co-authorship network connecting the top 25 collaborators of Michal Caspi. A scholar is included among the top collaborators of Michal Caspi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michal Caspi. Michal Caspi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Caspi, Michal, et al.. (2024). Wnt signaling regulates chemokine production and cell migration of circulating human monocytes. Cell Communication and Signaling. 22(1). 229–229. 4 indexed citations

Caspi, Michal, et al.. (2023). Nonsense mutation suppression is enhanced by targeting different stages of the protein synthesis process. PLoS Biology. 21(11). e3002355–e3002355. 5 indexed citations

Caspi, Michal, et al.. (2023). The C-terminal Amphipathic Helix of Carboxypeptidase E Mediates Export from the ER and Secretion via Lysosomes. Journal of Molecular Biology. 435(15). 168171–168171. 1 indexed citations

Caspi, Michal, et al.. (2020). Therapeutic targeting of the oncogenic Wnt signaling pathway for treating colorectal cancer and other colonic disorders. Advanced Drug Delivery Reviews. 169. 118–136. 66 indexed citations

Taiber, Shahar, Michal Caspi, Amiel A. Dror, et al.. (2020). Striatin Is Required for Hearing and Affects Inner Hair Cells and Ribbon Synapses. Frontiers in Cell and Developmental Biology. 8. 615–615. 4 indexed citations

Caspi, Michal, et al.. (2019). Serum starvation enhances nonsense mutation readthrough. Journal of Molecular Medicine. 97(12). 1695–1710. 7 indexed citations

Kariv, Revital, et al.. (2018). Erythromycin readthrough of APC nonsense stop codon mutation in Familial adenomatous polyposis. Annals of Oncology. 29. iii29–iii29. 1 indexed citations

Caspi, Michal, et al.. (2017). High-Temperature Requirement A1 (Htra1) - A Novel Regulator of Canonical Wnt Signaling. Scientific Reports. 7(1). 17995–17995. 21 indexed citations

Skalka, Nir, et al.. (2016). Carboxypeptidase E (CPE) inhibits the secretion and activity of Wnt3a. Oncogene. 35(50). 6416–6428. 7 indexed citations

10.

Kalma, Yael, Mira Malcov, Ami Amit, et al.. (2016). The effect of a germline mutation in the APC gene on β-catenin in human embryonic stem cells. BMC Cancer. 16(1). 952–952. 13 indexed citations

11.

Caspi, Michal, Nir Skalka, Ariel Munitz, et al.. (2015). A flow cytometry-based reporter assay identifies macrolide antibiotics as nonsense mutation read-through agents. Journal of Molecular Medicine. 94(4). 469–482. 21 indexed citations

12.

Caspi, Michal, et al.. (2014). 14‐3‐3 and β‐catenin are secreted on extracellular vesicles to activate the oncogenic Wnt pathway. Molecular Oncology. 8(5). 894–911. 48 indexed citations

13.

Skalka, Nir, Michal Caspi, E Caspi, Y. Peng Loh, & Rina Rosin‐Arbesfeld. (2012). Carboxypeptidase E: a negative regulator of the canonical Wnt signaling pathway. Oncogene. 32(23). 2836–2847. 41 indexed citations

14.

Caspi, Michal, et al.. (2010). The EDD E3 ubiquitin ligase ubiquitinates and up-regulates β-catenin. Molecular Biology of the Cell. 22(3). 399–411. 94 indexed citations

15.

Zilberberg, Alona, et al.. (2008). The armadillo repeat domain of the APC tumor suppressor protein interacts with Striatin family members. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1783(10). 1792–1802. 47 indexed citations

16.

Caspi, Michal, Alona Zilberberg, Hagit Eldar-Finkelman, & Rina Rosin‐Arbesfeld. (2008). Nuclear GSK-3β inhibits the canonical Wnt signalling pathway in a β-catenin phosphorylation-independent manner. Oncogene. 27(25). 3546–3555. 78 indexed citations

17.

Caspi, Michal, Frédéric M. Coquelle, Talia Levy, et al.. (2003). LIS1 Missense Mutations. Journal of Biological Chemistry. 278(40). 38740–38748. 26 indexed citations

18.

Coquelle, Frédéric M., Michal Caspi, Fabrice P. Cordelières, et al.. (2002). LIS1, CLIP-170's Key to the Dynein/Dynactin Pathway. Molecular and Cellular Biology. 22(9). 3089–3102. 213 indexed citations

19.

Sheffield, Peter, Sarah Garrard, Michal Caspi, et al.. (2000). Homologs of the ?- and ?-subunits of mammalian brain platelet-activating factor acetylhydrolase Ib in theDrosophila melanogaster genome. Proteins Structure Function and Bioinformatics. 39(1). 1–8. 24 indexed citations

20.

Heard, Jacqueline E., Michal Caspi, & Kathleen Dunn. (1997). Evolutionary Diversity of Symbiotically Induced Nodule MADS Box Genes: Characterization of nmhC5, a Member of a Novel Subfamily. Molecular Plant-Microbe Interactions. 10(5). 665–676. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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