Michail V. Sitkovsky
- Physiology top 0.01%
- Immunology top 0.2%
- Molecular Biology top 2%
- Oncology top 1%
- Cancer Research top 0.5%
- Co-authors
- Akio OhtaDmitriy LukashevSergey ApasovM. KoshibaCharles C. CaldwellStephen HatfieldJon A. BurasHidefumi Kojima
- Topics
- Adenosine and Purinergic Signaling (88 papers)Immune Cell Function and Interaction (55 papers)Cancer, Hypoxia, and Metabolism (17 papers)
- Cited by
- PhysiologyImmunologyNeurology
- Partner nations
- United StatesGermanyJapan
In The Last Decade
Michail V. Sitkovsky
131 papers receiving 13.7k citations
Hit Papers
Peers
Comparison fields: 5 of 132
- Physiology 6.6k
- Immunology 6.1k
- Molecular Biology 3.7k
- Oncology 2.4k
- Cancer Research 1.8k
Countries citing papers authored by Michail V. Sitkovsky
This map shows the geographic impact of Michail V. Sitkovsky's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michail V. Sitkovsky with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michail V. Sitkovsky more than expected).
Fields of papers citing papers by Michail V. Sitkovsky
This network shows the impact of papers produced by Michail V. Sitkovsky. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michail V. Sitkovsky. The network helps show where Michail V. Sitkovsky may publish in the future.
Co-authorship network of co-authors of Michail V. Sitkovsky
This figure shows the co-authorship network connecting the top 25 collaborators of Michail V. Sitkovsky. A scholar is included among the top collaborators of Michail V. Sitkovsky based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michail V. Sitkovsky. Michail V. Sitkovsky is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 34 | |
| 3 | 13 | |
| 4 | 33 | |
| 5 | 1 | |
| 6 | 164 | |
| 7 | 76 | |
| 8 | 67 | |
| 9 | 80 | |
| 10 | 27 | |
| 11 | A2A adenosine receptor protects tumors from antitumor T cellsbreakdown → | 817 |
| 12 | 33 | |
| 13 | 143 | |
| 14 | 5 | |
| 15 | 132 | |
| 16 | 64 | |
| 17 | 137 | |
| 18 | 336 | |
| 19 | 85 | |
| 20 | 67 |
About Michail V. Sitkovsky
Michail V. Sitkovsky is a scholar working on Physiology, Immunology and Neurology, having authored 132 papers that have together received 13.9k indexed citations. Recurring topics across this work include Adenosine and Purinergic Signaling (88 papers), Immune Cell Function and Interaction (55 papers) and Cancer, Hypoxia, and Metabolism (17 papers). The work is most often cited by research in Physiology (6.6k citations), Immunology (6.1k citations) and Neurology (1.1k citations). Michail V. Sitkovsky has collaborated with scholars based in United States, Germany and Japan. Frequent co-authors include Akio Ohta, Dmitriy Lukashev, Akio Ohta, Sergey Apasov, M. Koshiba, Charles C. Caldwell, Stephen Hatfield, Jon A. Buras, Hidefumi Kojima and Bernhard Holzmann. Their work appears in journals such as Nature, New England Journal of Medicine and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.