Michael W. Baughn

4.5k total citations · 2 hit papers
11 papers, 1.2k citations indexed

About

Michael W. Baughn is a scholar working on Neurology, Molecular Biology and Genetics. According to data from OpenAlex, Michael W. Baughn has authored 11 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Neurology, 7 papers in Molecular Biology and 4 papers in Genetics. Recurrent topics in Michael W. Baughn's work include Amyotrophic Lateral Sclerosis Research (8 papers), Neurogenetic and Muscular Disorders Research (4 papers) and Prion Diseases and Protein Misfolding (3 papers). Michael W. Baughn is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (8 papers), Neurogenetic and Muscular Disorders Research (4 papers) and Prion Diseases and Protein Misfolding (3 papers). Michael W. Baughn collaborates with scholars based in United States, Singapore and United Kingdom. Michael W. Baughn's co-authors include John Ravits, C. Frank Bennett, Frank Rigo, María José Gómez-Rodríguez, Matthew B. Harms, Robert H. Baloh, Takuya Ohkubo, Don W. Cleveland, Anais Sahabian and Leonard Petrucelli and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nature Neuroscience.

In The Last Decade

Michael W. Baughn

9 papers receiving 1.2k citations

Hit Papers

Targeting RNA Foci in iPSC-Derived Motor Neurons from ALS... 2013 2026 2017 2021 2013 2019 100 200 300 400
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Targeting RNA Foci in iPSC-Derived Motor Neurons from ALS Patients with a C9ORF72 Repeat Expansion
    2013 493 citations Dhruv Sareen, Jacqueline G. O’Rourke et al. Science Translational Medicine profile →
  2. Premature polyadenylation-mediated loss of stathmin-2 is a hallmark of TDP-43-dependent neurodegeneration
    2019 339 citations Ze’ev Melamed, Jone López‐Erauskin et al. Nature Neuroscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael W. Baughn United States 7 889 734 619 221 124 11 1.2k
Sharon Carmona United States 4 652 0.7× 512 0.7× 422 0.7× 221 1.0× 153 1.2× 4 977
Wendy Scheveneels Belgium 11 591 0.7× 456 0.6× 358 0.6× 221 1.0× 131 1.1× 13 915
Jeannie Chew United States 8 615 0.7× 491 0.7× 342 0.6× 158 0.7× 186 1.5× 8 894
Nicholas J. Maragakis United States 8 590 0.7× 660 0.9× 434 0.7× 275 1.2× 134 1.1× 11 1.1k
Xu‐Gang Xia United States 16 668 0.8× 568 0.8× 389 0.6× 173 0.8× 134 1.1× 23 1.1k
Nisha M. Badders United States 9 761 0.9× 989 1.3× 542 0.9× 275 1.2× 116 0.9× 10 1.5k
Anna M. Blokhuis Netherlands 7 655 0.7× 451 0.6× 412 0.7× 151 0.7× 152 1.2× 7 929
Yalda Sedaghat Canada 7 698 0.8× 736 1.0× 439 0.7× 152 0.7× 77 0.6× 10 1.1k
Elizabeth L. Daley United States 7 593 0.7× 513 0.7× 393 0.6× 167 0.8× 93 0.8× 7 863
Thomas Westergard United States 7 532 0.6× 425 0.6× 311 0.5× 197 0.9× 107 0.9× 8 779

Countries citing papers authored by Michael W. Baughn

Since Specialization
Citations

This map shows the geographic impact of Michael W. Baughn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael W. Baughn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael W. Baughn more than expected).

Fields of papers citing papers by Michael W. Baughn

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael W. Baughn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael W. Baughn. The network helps show where Michael W. Baughn may publish in the future.

Co-authorship network of co-authors of Michael W. Baughn

This figure shows the co-authorship network connecting the top 25 collaborators of Michael W. Baughn. A scholar is included among the top collaborators of Michael W. Baughn based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael W. Baughn. Michael W. Baughn is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Krupina, Ksenia, Alexander Goginashvili, Michael W. Baughn, et al.. (2025). Chromothripsis and ecDNA initiated by N4BP2 nuclease fragmentation of cytoplasm-exposed chromosomes. Science. 390(6778). 1156–1163.
2.
Beccari, Melinda S., Michael W. Baughn, Jonathan W. Artates, et al.. (2025). Stathmin-2 enhances motor axon regeneration after injury independent of its binding to tubulin. Proceedings of the National Academy of Sciences. 122(21). e2502294122–e2502294122.
3.
McMahon, Moira A., Meghdad Rahdar, Swagatam Mukhopadhyay, et al.. (2023). GOLGA8 increases bulk antisense oligonucleotide uptake and activity in mammalian cells. Molecular Therapy — Nucleic Acids. 32. 289–301. 5 indexed citations
4.
Smith, Andrew M., et al.. (2021). Strong, Non-specific Adhesion Using C-Lectin Heterotrimers in a Molluscan Defensive Secretion. Integrative and Comparative Biology. 61(4). 1440–1449. 7 indexed citations
5.
Melamed, Ze’ev, Jone López‐Erauskin, Michael W. Baughn, et al.. (2019). Premature polyadenylation-mediated loss of stathmin-2 is a hallmark of TDP-43-dependent neurodegeneration. Nature Neuroscience. 22(2). 180–190. 339 indexed citations breakdown →
6.
Krach, Florian, Ranjan Batra, Emily C. Wheeler, et al.. (2018). Transcriptome–pathology correlation identifies interplay between TDP-43 and the expression of its kinase CK1E in sporadic ALS. Acta Neuropathologica. 136(3). 405–423. 60 indexed citations
7.
8.
Saberi, Shahram, Jennifer E. Stauffer, Jie Jiang, et al.. (2017). Sense-encoded poly-GR dipeptide repeat proteins correlate to neurodegeneration and uniquely co-localize with TDP-43 in dendrites of repeat-expanded C9orf72 amyotrophic lateral sclerosis. Acta Neuropathologica. 135(3). 459–474. 135 indexed citations
9.
Sareen, Dhruv, Jacqueline G. O’Rourke, Pratap Meera, et al.. (2013). Targeting RNA Foci in iPSC-Derived Motor Neurons from ALS Patients with a C9ORF72 Repeat Expansion. Science Translational Medicine. 5(208). 208ra149–208ra149. 493 indexed citations breakdown →
10.
Harms, Matthew B., Janet Cady, Craig M. Zaidman, et al.. (2013). Lack of C9ORF72 coding mutations supports a gain of function for repeat expansions in amyotrophic lateral sclerosis. Neurobiology of Aging. 34(9). 2234.e13–2234.e19. 58 indexed citations
11.
Rabin, Stuart J., Jae Mun Kim, Michael W. Baughn, et al.. (2009). Sporadic ALS has compartment-specific aberrant exon splicing and altered cell–matrix adhesion biology. Human Molecular Genetics. 19(2). 313–328. 106 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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