Michael Schwake

6.4k citations

59 papers · 4.4k indexed · h-index 34

Impact in

Physiology top 0.5%
- Lysosomal Storage Disorders Research
- Calcium signaling and nucleotide metabolism
- Alzheimer's disease research and treatments
Cell Biology top 1%
- Cellular transport and secretion

Papers in ⓘ

Physiology 7
- Lysosomal Storage Disorders Research 18
- Calcium signaling and nucleotide metabolism 7
Cell Biology 13
- Cellular transport and secretion 12

Co-authors: Paul Säftig (28 shared papers)Thomas J. Jentsch (6 shared papers)Thomas Friedrich (7 shared papers)Judith Blanz (8 shared papers)W. C. Gunther (1 shared paper)Michael R. Bösl (1 shared paper)Michael Pusch (2 shared papers)Bernd Schröder (1 shared paper)
Journals: Journal of Biological Chemistry (6 papers)British Journal of Pharmacology (4 papers)Journal of Neuroscience (4 papers)Nature Communications (3 papers)Proceedings of the National Academy of Sciences (3 papers)
Partner nations: Germany United States United Kingdom

In The Last Decade

Michael Schwake

58 papers receiving 4.4k citations

Peers

Countries citing papers authored by Michael Schwake

Since Specialization

Citations

This map shows the geographic impact of Michael Schwake's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Schwake with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Schwake more than expected).

Fields of papers citing papers by Michael Schwake

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Schwake. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Schwake. The network helps show where Michael Schwake may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael Schwake, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael Schwake Line = papers co-authored together Michael Schwake links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 59 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	ClC-5 Cl--channel disruption impairs endocytosis in a mouse model for Dent's disease Nature ·Nils Piwon, W. C. Gunther, Michael Schwake, Michael R. Bösl, Thomas J. Jentsch	2000	443
2	LIMP-2 Is a Receptor for Lysosomal Mannose-6-Phosphate-Independent Targeting of β-Glucocerebrosidase Cell ·David Reczek, Michael Schwake, J Schröder, Heather Hughes, Judith Blanz, Xiaoying Jin, William Brondyk, Scott Van Patten, Tim Edmunds, Paul Säftig	2007	420
3	Phenotypic and Biochemical Analyses of BACE1- and BACE2-deficient Mice Journal of Biological Chemistry ·Diana Dominguez, Jos Tournoy, Dieter Hartmann, Tobias Huth, Kim Cryns, Siska Deforce, Lutgarde Serneels, Ira Espuny-Camacho, Els Marjaux, Katleen Craessaerts, Anton Roebroek, Michael Schwake, Rudi D’Hooge, Patricia Bach, Ulrich Kalinke, Dieder Moechars, Christian Alzheimer, Karina Reiß, Paul Säftig, Bart De Strooper	2005	271
4	Structure of LIMP-2 provides functional insights with implications for SR-BI and CD36 Nature ·Dante Neculai, Michael Schwake, Mani Ravichandran, Friederike Zunke, Richard F. Collins, Judith Peters, M. Neculai, Jonathan Plumb, P. Loppnau, J.C. Pizarro, Alma Seitova, William S. Trimble, Paul Säftig, Sergio Grinstein, Sirano Dhe‐Paganon	2013	224
5	Molecular Determinants of KCNQ (K_v7) K⁺Channel Sensitivity to the Anticonvulsant Retigabine Journal of Neuroscience ·Anne Schenzer, Thomas Friedrich, Michael Pusch, Paul Säftig, Thomas J. Jentsch, Joachim Grötzinger, Michael Schwake	2005	219
6	Lysosomal Membrane Proteins and Their Central Role in Physiology Traffic ·Michael Schwake, Bernd Schröder, Paul Säftig	2013	185
7	GRIN2B mutations in west syndrome and intellectual disability with focal epilepsy Annals of Neurology ·Johannes R. Lemke, Rik Hendrickx, Kirsten Geider, Bodo Laube, Michael Schwake, Victoria L. Harvey, Victoria M. James, Alex Pepler, Isabelle Steiner, Konstanze Hörtnagel, John Neidhardt, Susanne Ruf, Markus Wolff, Deborah Bartholdi, Roberto Caraballo, Konrad Platzer, Arvid Suls, Peter De Jonghe, Saskia Biskup, Sarah Weckhuysen	2013	158
8	Molecular expression and pharmacological identification of a role for K_v7 channels in murine vascular reactivity British Journal of Pharmacology ·Shuk Yin M. Yeung, Vladimı́r Pucovský, James D. Moffatt, Luiz Balthazar Saldanha, Michael Schwake, Susumu Ohya, Iain A. Greenwood	2007	156
9	Surface Expression and Single Channel Properties of KCNQ2/KCNQ3, M-type K+ Channels Involved in Epilepsy Journal of Biological Chemistry ·Michael Schwake, Michael Pusch, Tatjana Kharkovets, Thomas J. Jentsch	2000	143
10	LRRK2 kinase activity regulates lysosomal glucocerebrosidase in neurons derived from Parkinson’s disease patients Nature Communications ·Daniel Ysselstein, Maria Nguyen, Tiffany Young, Alex Severino, Michael Schwake, Kalpana Merchant, Dimitri Krainc	2019	140
11	A carboxy‐terminal domain determines the subunit specificity of KCNQ K ⁺ channel assembly EMBO Reports ·Michael Schwake, Thomas J. Jentsch, Thomas Friedrich	2003	117
12	An Internalization Signal in ClC-5, an Endosomal Cl−Channel Mutated in Dent's Disease Journal of Biological Chemistry ·Michael Schwake, Thomas Friedrich, Thomas J. Jentsch	2001	117
13	Lysosomal integral membrane protein-2 (LIMP-2/SCARB2) is involved in lysosomal cholesterol export Nature Communications ·Saskia Heybrock, Kristiina Kanerva, Ying Meng, Chris Ing, Anna Liang, Zi-Jian Xiong, Xialian Weng, Young Ah Kim, Richard F. Collins, William S. Trimble, Régis Pomès, Gilbert G. Privé, Wim Annaert, Michael Schwake, Jöerg Heeren, Renate Lüllmann‐Rauch, Sergio Grinstein, Elina Ikonen, Paul Säftig, Dante Neculai	2019	115
14	Refinement of the Binding Site and Mode of Action of the Anticonvulsant Retigabine on KCNQ K+ Channels Molecular Pharmacology ·Wienke Lange, Jan Geißendörfer, Anne Schenzer, Joachim Grötzinger, Guiscard Seebohm, Thomas Friedrich, Michael Schwake	2008	103
15	LIMP-2 expression is critical for β-glucocerebrosidase activity and α-synuclein clearance Proceedings of the National Academy of Sciences ·Michelle Rothaug, Friederike Zunke, Joseph R. Mazzulli, Michaela Schweizer, Hermann C. Altmeppen, Renate Lüllmann‐Rauch, Wouter W. Kallemeijn, Paulo Gaspar, Johannes M. F. G. Aerts, Markus Glatzel, Paul Säftig, Dimitri Krainc, Michael Schwake, Judith Blanz	2014	102
16	Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients Human Molecular Genetics ·Clarissa Valdez, Yvette C. Wong, Michael Schwake, Guojun Bu, Zbigniew K. Wszołek, Dimitri Krainc	2017	101
17	A DNA-based voltmeter for organelles Nature Nanotechnology ·Anand Saminathan, John Devany, Aneesh Tazhe Veetil, Bhavyashree Suresh, Kavya Smitha Pillai, Michael Schwake, Yamuna Krishnan	2020	92
18	Tetraspanin15 regulates cellular trafficking and activity of the ectodomain sheddase ADAM10 Cellular and Molecular Life Sciences ·Johannes Prox, Michael Willenbrock, Silvio Weber, Tobias Lehmann, Dirk Schmidt‐Arras, Ralf Schwanbeck, Paul Säftig, Michael Schwake	2012	89
19	A Mutation in the Golgi Qb-SNARE Gene GOSR2 Causes Progressive Myoclonus Epilepsy with Early Ataxia The American Journal of Human Genetics ·Mark Corbett, Michael Schwake, Melanie Bahlo, Leanne M. Dibbens, Meng Lin, Luke C. Gandolfo, Danya F. Vears, John D. O’Sullivan, Thomas Robertson, Marta A. Bayly, Alison Gardner, Annemarie Vlaar, Georg Christoph Korenke, Bastiaan R. Bloem, I.F.M. de Coo, Judith M.A. Verhagen, Anna‐Elina Lehesjoki, Jozef Gécz, Samuel F. Berkovic	2011	84
20	Structural Determinants of M-Type KCNQ (K_v7) K⁺Channel Assembly Journal of Neuroscience ·Michael Schwake, Despina Athanasiadu, Christian Beimgraben, Judith Blanz, Christian Beck, Thomas J. Jentsch, Paul Säftig, Thomas Friedrich	2006	80

About Michael Schwake

Michael Schwake is a scholar working on Physiology, Physiology, Cell Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience, having authored 59 papers that have together received 4.4k indexed citations. Recurring topics across this work include Lysosomal Storage Disorders Research (18 papers), Ion channel regulation and function (17 papers), Cardiac electrophysiology and arrhythmias (14 papers), Cellular transport and secretion (12 papers), Neuroscience and Neuropharmacology Research (10 papers), Calcium signaling and nucleotide metabolism (7 papers), Carbohydrate Chemistry and Synthesis (5 papers) and Genetics and Neurodevelopmental Disorders (4 papers). The work is most often cited by research in Physiology (416 citations), Cell Biology (936 citations), Physiology (1.4k citations), Cellular and Molecular Neuroscience (986 citations) and Cardiology and Cardiovascular Medicine (878 citations). Michael Schwake has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include Paul Säftig, Thomas J. Jentsch, Thomas Friedrich, Judith Blanz, W. C. Gunther, Michael R. Bösl, Michael Pusch, Bernd Schröder, Dimitri Krainc and J Schröder. Their work appears in journals such as Journal of Biological Chemistry, British Journal of Pharmacology, Journal of Neuroscience, Nature Communications and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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