Michael B. Small
- Oncology top 10%
- Cancer-related Molecular Pathways 4
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- Cell death mechanisms and regulation 5
- RNA regulation and disease 2
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- Neuroblastoma Research and Treatments 2
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- Virus-based gene therapy research 5
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- Telomeres, Telomerase, and Senescence 4
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- Tissue Engineering and Regenerative Medicine 2
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- Microtubule and mitosis dynamics 2
- Co-authors
- Nissim HayAndrew J. WagnerHarvey L. OzerYakov GluzmanJ. Michael BishopM. SchwabManfred SchwabPatrick Brady
- Cited by
- OncologyMolecular BiologyNeurology
- Journals
- Molecular and Cellular Biology (4 papers)Journal of Cellular Physiology (3 papers)Experimental Cell Research (2 papers)
- Partner nations
- United StatesUnited Kingdom
In The Last Decade
Michael B. Small
13 papers receiving 653 citations
Peers
Comparison fields: 5 of 78
- Oncology 237
- Molecular Biology 449
- Neurology 73
- Cancer Research 63
- Genetics 108
Countries citing papers authored by Michael B. Small
This map shows the geographic impact of Michael B. Small's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael B. Small with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael B. Small more than expected).
Fields of papers citing papers by Michael B. Small
This network shows the impact of papers produced by Michael B. Small. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael B. Small. The network helps show where Michael B. Small may publish in the future.
Co-authorship network
The 19 scholars most cited alongside Michael B. Small, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2008 | 25 | |
| 2 | 1997 | 7 | |
| 3 | 1997 | 10 | |
| 4 | 1996 | 19 | |
| 5 | 1996 | 5 | |
| 6 | 1996 | 1 | |
| 7 | 1995 | 15 | |
| 8 | 1993 | 106 | |
| 9 | 1993 | 54 | |
| 10 | 1993 | 195 | |
| 11 | 1987 | 107 | |
| 12 | 1987 | 27 | |
| 13 | 1982 | 106 |
About Michael B. Small
Michael B. Small is a scholar working on Aging, Oncology, Genetics, Physiology and Neurology, having authored 13 papers that have together received 677 indexed citations. Recurring topics across this work include Virus-based gene therapy research (5 papers), Cell death mechanisms and regulation (5 papers), Telomeres, Telomerase, and Senescence (4 papers), Cancer-related Molecular Pathways (4 papers), Tissue Engineering and Regenerative Medicine (2 papers), RNA regulation and disease (2 papers), Neuroblastoma Research and Treatments (2 papers) and Microtubule and mitosis dynamics (2 papers). The work is most often cited by research in Oncology (237 citations), Molecular Biology (449 citations), Neurology (73 citations), Cancer Research (63 citations) and Genetics (108 citations). Michael B. Small has collaborated with scholars based in United States and United Kingdom. Frequent co-authors include Nissim Hay, Andrew J. Wagner, Harvey L. Ozer, Yakov Gluzman, J. Michael Bishop, M. Schwab, Manfred Schwab, Patrick Brady, Haim Pinkas and Karen Hubbard. Their work appears in journals such as Molecular and Cellular Biology, Journal of Cellular Physiology, Experimental Cell Research, Current Opinion in Cell Biology and The Journal of Infectious Diseases.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.