Mehdi Aliomrani

633 citations
44 papers · 499 indexed · h-index 13
Topics
Multiple Sclerosis Research Studies (9 papers)Heavy Metal Exposure and Toxicity (6 papers)Glycosylation and Glycoproteins Research (5 papers)
Partner nations
IranCanadaBrazil

In The Last Decade

Mehdi Aliomrani

42 papers receiving 492 citations

Peers

Mehdi Aliomrani
Comparison fields: 5 of 94
  • Molecular Biology 175
  • Health, Toxicology and Mutagenesis 105
  • Plant Science 78
  • Pathology and Forensic Medicine 72
  • Pharmacology 64
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Countries citing papers authored by Mehdi Aliomrani

Since Specialization
Citations

This map shows the geographic impact of Mehdi Aliomrani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mehdi Aliomrani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mehdi Aliomrani more than expected).

Fields of papers citing papers by Mehdi Aliomrani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mehdi Aliomrani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mehdi Aliomrani. The network helps show where Mehdi Aliomrani may publish in the future.

Co-authorship network of co-authors of Mehdi Aliomrani

This figure shows the co-authorship network connecting the top 25 collaborators of Mehdi Aliomrani. A scholar is included among the top collaborators of Mehdi Aliomrani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mehdi Aliomrani. Mehdi Aliomrani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 1
2 0
3 4
4 0
5 8
6 5
7 9
8 9
9 1
10 1
11 23
12 8
13 11
14 13
15 34
16
The Prevalence of the Genetic Polymorphism of GSTM1, GSTT1 and GSTP1 and Its Relationship with Clinical Criteria of Multiple Sclerosis (MS) Patients in Tehran
3
17 25
18 48
19 38
20
Cytotoxicity of Euphorbia bungei on HeLa and HT29 tumor cells
1

About Mehdi Aliomrani

Mehdi Aliomrani is a scholar working on Developmental Neuroscience, Pathology and Forensic Medicine and Health, Toxicology and Mutagenesis, having authored 44 papers that have together received 499 indexed citations. Recurring topics across this work include Multiple Sclerosis Research Studies (9 papers), Heavy Metal Exposure and Toxicity (6 papers) and Glycosylation and Glycoproteins Research (5 papers). The work is most often cited by research in Developmental Neuroscience (32 citations), Health, Toxicology and Mutagenesis (105 citations) and Biological Psychiatry (12 citations). Mehdi Aliomrani has collaborated with scholars based in Iran, Canada and Brazil. Frequent co-authors include Mohammad Reza Sepand, Hamid Shirkhanloo, Mina Mirian, Mohammad Ali Sahraian, Jaleh Varshosaz, Mohammad Sharifzadeh, Mohammad Reza Khoshayand, Ali Kazemi, Hamid Reza Mirzaei and Omid Sabzevari. Their work appears in journals such as SHILAP Revista de lepidopterología, International Journal of Pharmaceutics and International Journal of Biological Macromolecules.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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