M.B. Bennink

565 total citations
16 papers, 432 citations indexed

About

M.B. Bennink is a scholar working on Immunology, Oncology and Rheumatology. According to data from OpenAlex, M.B. Bennink has authored 16 papers receiving a total of 432 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 5 papers in Oncology and 5 papers in Rheumatology. Recurrent topics in M.B. Bennink's work include Osteoarthritis Treatment and Mechanisms (5 papers), Cytokine Signaling Pathways and Interactions (4 papers) and Immune Response and Inflammation (3 papers). M.B. Bennink is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (5 papers), Cytokine Signaling Pathways and Interactions (4 papers) and Immune Response and Inflammation (3 papers). M.B. Bennink collaborates with scholars based in Netherlands, Germany and United States. M.B. Bennink's co-authors include Wim B. van den Berg, Fons A. J. van de Loo, Onno J. Arntz, Leo A. B. Joosten, Ruben L. Smeets, Sharon Veenbergen, E.L. Vitters, E.N. Blaney Davidson, P.M. van der Kraan and A. van Caam and has published in prestigious journals such as Annals of the Rheumatic Diseases, Osteoarthritis and Cartilage and Gene Therapy.

In The Last Decade

M.B. Bennink

15 papers receiving 425 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
M.B. Bennink Netherlands 10 176 176 173 71 69 16 432
E.U. Sumer Denmark 10 244 1.4× 54 0.3× 169 1.0× 57 0.8× 96 1.4× 12 497
Zrinka Jajić Croatia 10 194 1.1× 68 0.4× 104 0.6× 69 1.0× 32 0.5× 43 339
Claire Deligne France 8 88 0.5× 173 1.0× 97 0.6× 142 2.0× 33 0.5× 18 415
Fumihiko Miyoshi Japan 10 104 0.6× 152 0.9× 204 1.2× 68 1.0× 18 0.3× 25 454
A. DiFrancesco United States 8 259 1.5× 139 0.8× 126 0.7× 111 1.6× 137 2.0× 12 522
Elizabeth Hutto United States 8 91 0.5× 149 0.8× 151 0.9× 105 1.5× 24 0.3× 10 569
David H. Goddard United Kingdom 13 114 0.6× 65 0.4× 95 0.5× 40 0.6× 31 0.4× 25 372
Claudia J. Calder United Kingdom 10 73 0.4× 385 2.2× 147 0.8× 44 0.6× 13 0.2× 15 606
Ana P. Lopes Netherlands 14 137 0.8× 214 1.2× 156 0.9× 68 1.0× 7 0.1× 27 500
B. Stott United Kingdom 8 167 0.9× 144 0.8× 76 0.4× 104 1.5× 76 1.1× 13 461

Countries citing papers authored by M.B. Bennink

Since Specialization
Citations

This map shows the geographic impact of M.B. Bennink's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M.B. Bennink with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M.B. Bennink more than expected).

Fields of papers citing papers by M.B. Bennink

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M.B. Bennink. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M.B. Bennink. The network helps show where M.B. Bennink may publish in the future.

Co-authorship network of co-authors of M.B. Bennink

This figure shows the co-authorship network connecting the top 25 collaborators of M.B. Bennink. A scholar is included among the top collaborators of M.B. Bennink based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M.B. Bennink. M.B. Bennink is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
2.
Broeren, Mathijs G A, et al.. (2015). AB0101 Viral Expression of TSG-6 Can Stimulate Osteophyte Formation in Experimental Osteoarthritis. Annals of the Rheumatic Diseases. 74. 924–924. 1 indexed citations
3.
Davidson, E.N. Blaney, E.L. Vitters, M.B. Bennink, et al.. (2014). Inducible chondrocyte-specific overexpression of BMP2 in young mice results in severe aggravation of osteophyte formation in experimental OA without altering cartilage damage. Annals of the Rheumatic Diseases. 74(6). 1257–1264. 42 indexed citations
4.
Davidson, E.N. Blaney, E.L. Vitters, M.B. Bennink, et al.. (2014). BMP2 requires TGF-beta to induce osteophytes during experimental OA. Osteoarthritis and Cartilage. 22. S16–S17.
5.
Davidson, E.N. Blaney, A. van Caam, E.L. Vitters, et al.. (2014). TGF-β is a potent inducer of Nerve Growth Factor in articular cartilage via the ALK5-Smad2/3 pathway. Potential role in OA related pain?. Osteoarthritis and Cartilage. 23(3). 478–486. 65 indexed citations
6.
Davidson, E.N. Blaney, E.L. Vitters, A. van Caam, et al.. (2014). A5.18 BMP2 requires TGF-BETA to induce osteophytes during experimental osteoarthritis. Annals of the Rheumatic Diseases. 73. A70–A70. 1 indexed citations
7.
Davidson, E.N. Blaney, E.L. Vitters, M.B. Bennink, et al.. (2013). Inducible chondrocyte-specific overexpression of BMP2 in young mice results in severe aggravation of osteophyte formation in experimental OA without altering cartilage damage. Osteoarthritis and Cartilage. 21. S67–S67. 5 indexed citations
8.
Abdollahi‐Roodsaz, Shahla, M.B. Bennink, Onno J. Arntz, et al.. (2012). Therapeutic efficacy of Tyro3, Axl, and Mer tyrosine kinase agonists in collagen‐induced arthritis. Arthritis & Rheumatism. 65(3). 671–680. 61 indexed citations
9.
Veenbergen, Sharon, M.B. Bennink, Alsya J. Affandi, et al.. (2011). A pivotal role for antigen-presenting cells overexpressing SOCS3 in controlling invariant NKT cell responses during collagen-induced arthritis. Annals of the Rheumatic Diseases. 70(12). 2167–2175. 20 indexed citations
10.
Veenbergen, Sharon, Ruben L. Smeets, M.B. Bennink, et al.. (2009). The natural soluble form of IL-18 receptor β exacerbates collagen-induced arthritis via modulation of T-cell immune responses. Annals of the Rheumatic Diseases. 69(1). 276–283. 27 indexed citations
11.
Geurts, Jeroen, Onno J. Arntz, M.B. Bennink, et al.. (2007). Application of a disease-regulated promoter is a safer mode of local IL-4 gene therapy for arthritis. Gene Therapy. 14(23). 1632–1638. 20 indexed citations
12.
Smeets, Ruben L., Sharon Veenbergen, Onno J. Arntz, et al.. (2006). A novel role for suppressor of cytokine signaling 3 in cartilage destruction via induction of chondrocyte desensitization toward insulin‐like growth factor. Arthritis & Rheumatism. 54(5). 1518–1528. 41 indexed citations
13.
Smeets, Ruben L., Leo A. B. Joosten, Onno J. Arntz, et al.. (2005). Soluble interleukin‐1 receptor accessory protein ameliorates collagen‐induced arthritis by a different mode of action from that of interleukin‐1 receptor antagonist. Arthritis & Rheumatism. 52(7). 2202–2211. 35 indexed citations
14.
Smeets, Ruben L., Fons A. J. van de Loo, Onno J. Arntz, et al.. (2003). Adenoviral delivery of IL-18 binding protein C ameliorates Collagen-Induced Arthritis in mice. Gene Therapy. 10(12). 1004–1011. 71 indexed citations
15.
Smeets, Ruben L., Fons A. J. van de Loo, Leo A. B. Joosten, et al.. (2003). Effectiveness of the soluble form of the interleukin‐1 receptor accessory protein as an inhibitor of interleukin‐1 in collagen‐induced arthritis. Arthritis & Rheumatism. 48(10). 2949–2958. 39 indexed citations
16.
Smeets, Ruben L., Fons A. J. van de Loo, Leo A. B. Joosten, et al.. (2003). Modulation of Arthritis through overexpression of soluble inter-leukin-1 receptor accessory protein (sIL-1RAcP): a novel inhibitor of interleukin-1, distinct from IL-1Ra. Inflammation Research. 52(S2). S197–S199. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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