Maysa E. El‐Sayed
- Cancer Research top 0.5%
- Oncology top 1%
- Pathology and Forensic Medicine top 1%
- Molecular Biology top 10%
- Pulmonary and Respiratory Medicine top 5%
- Co-authors
- Emad A. RakhaIan O. EllisAndrew R. GreenAndrew H.S. LeeJ.F.R. RobertsonRoger W. BlameyMatthew J. GraingeDesmond G. Powe
- Topics
- Breast Cancer Treatment Studies (16 papers)Breast Lesions and Carcinomas (11 papers)HER2/EGFR in Cancer Research (6 papers)
- Partner nations
- United KingdomEgyptUnited States
In The Last Decade
Maysa E. El‐Sayed
19 papers receiving 2.9k citations
Hit Papers
Peers
Comparison fields: 5 of 94
- Cancer Research 1.9k
- Oncology 1.8k
- Pathology and Forensic Medicine 821
- Molecular Biology 812
- Pulmonary and Respiratory Medicine 471
Countries citing papers authored by Maysa E. El‐Sayed
This map shows the geographic impact of Maysa E. El‐Sayed's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maysa E. El‐Sayed with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maysa E. El‐Sayed more than expected).
Fields of papers citing papers by Maysa E. El‐Sayed
This network shows the impact of papers produced by Maysa E. El‐Sayed. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maysa E. El‐Sayed. The network helps show where Maysa E. El‐Sayed may publish in the future.
Co-authorship network of co-authors of Maysa E. El‐Sayed
This figure shows the co-authorship network connecting the top 25 collaborators of Maysa E. El‐Sayed. A scholar is included among the top collaborators of Maysa E. El‐Sayed based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maysa E. El‐Sayed. Maysa E. El‐Sayed is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 15 | |
| 2 | 18 | |
| 3 | 21 | |
| 4 | 6 | |
| 5 | 4 | |
| 6 | 394 | |
| 7 | 17 | |
| 8 | 63 | |
| 9 | 39 | |
| 10 | 110 | |
| 11 | 124 | |
| 12 | 32 | |
| 13 | 435 | |
| 14 | 185 | |
| 15 | 141 | |
| 16 | 99 | |
| 17 | 45 | |
| 18 | 238 | |
| 19 | Prognostic markers in triple‐negative breast cancerbreakdown → | 1039 |
About Maysa E. El‐Sayed
Maysa E. El‐Sayed is a scholar working on Cancer Research, Pathology and Forensic Medicine and Dermatology, having authored 19 papers that have together received 3.0k indexed citations. Recurring topics across this work include Breast Cancer Treatment Studies (16 papers), Breast Lesions and Carcinomas (11 papers) and HER2/EGFR in Cancer Research (6 papers). The work is most often cited by research in Cancer Research (1.9k citations), Oncology (1.8k citations) and Pathology and Forensic Medicine (821 citations). Maysa E. El‐Sayed has collaborated with scholars based in United Kingdom, Egypt and United States. Frequent co-authors include Emad A. Rakha, Ian O. Ellis, Andrew R. Green, Andrew H.S. Lee, J.F.R. Robertson, Roger W. Blamey, Matthew J. Grainge, Desmond G. Powe, Zsolt Hodi and C.W. Elston. Their work appears in journals such as Journal of Clinical Oncology, Cancer and Clinical Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.