Maya R. Karta

670 total citations
9 papers, 462 citations indexed

About

Maya R. Karta is a scholar working on Immunology, Physiology and Surgery. According to data from OpenAlex, Maya R. Karta has authored 9 papers receiving a total of 462 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Immunology, 7 papers in Physiology and 2 papers in Surgery. Recurrent topics in Maya R. Karta's work include IL-33, ST2, and ILC Pathways (7 papers), Asthma and respiratory diseases (7 papers) and Immune Cell Function and Interaction (3 papers). Maya R. Karta is often cited by papers focused on IL-33, ST2, and ILC Pathways (7 papers), Asthma and respiratory diseases (7 papers) and Immune Cell Function and Interaction (3 papers). Maya R. Karta collaborates with scholars based in United States, South Korea and Ethiopia. Maya R. Karta's co-authors include David H. Broide, Taylor A. Doherty, Peter Rosenthal, Sudipta Das, Marina Miller, Andrew Beppu, Richard C. Kurten, Christine N. Vuong, Fazila Chouiali and Qutayba Hamid and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Immunology.

In The Last Decade

Maya R. Karta

9 papers receiving 456 citations

Peers

Countries citing papers authored by Maya R. Karta

Since Specialization

Citations

This map shows the geographic impact of Maya R. Karta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maya R. Karta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maya R. Karta more than expected).

Fields of papers citing papers by Maya R. Karta

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maya R. Karta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maya R. Karta. The network helps show where Maya R. Karta may publish in the future.

Co-authorship network of co-authors of Maya R. Karta

This figure shows the co-authorship network connecting the top 25 collaborators of Maya R. Karta. A scholar is included among the top collaborators of Maya R. Karta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maya R. Karta. Maya R. Karta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

#	Work	Indexed citations
1	β2 integrins rather than β1 integrins mediate Alternaria-induced group 2 innate lymphoid cell trafficking to the lung 2017 ·Journal of Allergy and Clinical Immunology ·Maya R. Karta, Peter Rosenthal, Andrew Beppu, (unknown), Marina Miller, Sudipta Das, Richard C. Kurten, Taylor A. Doherty, David H. Broide	61
2	Group 2 innate lymphoid cells are recruited to the nasal mucosa in patients with aspirin-exacerbated respiratory disease 2017 ·Journal of Allergy and Clinical Immunology ·(unknown), Kellen Cavagnero, Adam S. DeConde, (unknown), Maya R. Karta, David H. Broide, Bruce L. Zuraw, Andrew A. White, Sandra C. Christiansen, Taylor A. Doherty	72
3	Rhinovirus Infection of ORMDL3 Transgenic Mice Is Associated with Reduced Rhinovirus Viral Load and Airway Inflammation 2017 ·The Journal of Immunology ·Dae Jin Song, Marina Miller, Andrew Beppu, Peter Rosenthal, Sudipta Das, Maya R. Karta, Christine N. Vuong, Amit Mehta, Michael Croft, David H. Broide	12
4	Insights into Group 2 Innate Lymphoid Cells in Human Airway Disease 2016 ·Current Allergy and Asthma Reports ·Maya R. Karta, David H. Broide, Taylor A. Doherty	63
5	GSDMB induces an asthma phenotype characterized by increased airway responsiveness and remodeling without lung inflammation 2016 ·Proceedings of the National Academy of Sciences ·Sudipta Das, Marina Miller, Andrew Beppu, J. Mueller, Matthew D. McGeough, Christine N. Vuong, Maya R. Karta, Peter Rosenthal, Fazila Chouiali, Taylor A. Doherty, Richard C. Kurten, Qutayba Hamid, Hal M. Hoffman, David H. Broide	147
6	Fstl1 Promotes Asthmatic Airway Remodeling by Inducing Oncostatin M 2015 ·The Journal of Immunology ·Marina Miller, Andrew Beppu, Peter Rosenthal, Alexa Pham, Sudipta Das, Maya R. Karta, Dae Jin Song, Christine N. Vuong, Taylor A. Doherty, Michael Croft, Bruce L. Zuraw, Xu Zhang, Xiang Gao, Seema S. Aceves, Fazila Chouiali, Qutayba Hamid, David H. Broide	41
7	LPS Modulates Rhinovirus-Induced Chemokine Secretion in Monocytes and Macrophages 2014 ·American Journal of Respiratory Cell and Molecular Biology ·Maya R. Karta, Monica L. Gavala, Colleen S. Curran, (unknown), Patricia J. Keely, James E. Gern, Paul J. Bertics	17
8	Simvastatin attenuates rhinovirus-induced interferon and CXCL10 secretion from monocytic cells in vitro 2014 ·Journal of Leukocyte Biology ·(unknown), Maya R. Karta, Anjon Audhya, James E. Gern, Paul J. Bertics	18
9	Activation of the Transcription Factor FosB/Activating Protein-1 (AP-1) Is a Prominent Downstream Signal of the Extracellular Nucleotide Receptor P2RX7 in Monocytic and Osteoblastic Cells* 2010 ·Journal of Biological Chemistry ·Monica L. Gavala, Lindsay M. Hill, Lisa Y. Lenertz, Maya R. Karta, Paul J. Bertics	31

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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