Max A. Thorwald

598 total citations
37 papers, 390 citations indexed

About

Max A. Thorwald is a scholar working on Physiology, Health, Toxicology and Mutagenesis and Molecular Biology. According to data from OpenAlex, Max A. Thorwald has authored 37 papers receiving a total of 390 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Physiology, 10 papers in Health, Toxicology and Mutagenesis and 9 papers in Molecular Biology. Recurrent topics in Max A. Thorwald's work include Air Quality and Health Impacts (10 papers), Alzheimer's disease research and treatments (8 papers) and Genomics, phytochemicals, and oxidative stress (4 papers). Max A. Thorwald is often cited by papers focused on Air Quality and Health Impacts (10 papers), Alzheimer's disease research and treatments (8 papers) and Genomics, phytochemicals, and oxidative stress (4 papers). Max A. Thorwald collaborates with scholars based in United States, Japan and China. Max A. Thorwald's co-authors include Rudy M. Ortiz, Caleb E. Finch, Akira Nishiyama, Daisuke Nakano, Rubén Rodríguez, Todd E. Morgan, Henry Jay Forman, Amin Haghani, Constantinos Sioutas and János Peti‐Peterdi and has published in prestigious journals such as Stroke, Free Radical Biology and Medicine and Environmental Pollution.

In The Last Decade

Max A. Thorwald

33 papers receiving 387 citations

Peers

Max A. Thorwald

HTM

PHYSI

EDM

CCM

Daniele La Russa Italy

Marcos André Soares Leal Brazil

Ai-Ling Ji China

Mohammad Kazem Gharib-Naseri Iran

Sufang Chen China

Yu‐Wan Yang Taiwan

Francesco Torres Italy

M Endo Japan

Yali Xu China

Daniele La Russa Italy

Max A. Thorwald

70 ×0.6

HTM

127 ×1.3

123 ×1.5

PHYSI

49 ×0.8

EDM

31 ×0.7

CCM

Citations per year, relative to Max A. Thorwald Max A. Thorwald (= 1×) peers Daniele La Russa

Countries citing papers authored by Max A. Thorwald

Since Specialization

Citations

This map shows the geographic impact of Max A. Thorwald's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Max A. Thorwald with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Max A. Thorwald more than expected).

Fields of papers citing papers by Max A. Thorwald

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Max A. Thorwald. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Max A. Thorwald. The network helps show where Max A. Thorwald may publish in the future.

Co-authorship network of co-authors of Max A. Thorwald

This figure shows the co-authorship network connecting the top 25 collaborators of Max A. Thorwald. A scholar is included among the top collaborators of Max A. Thorwald based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Max A. Thorwald. Max A. Thorwald is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Shkirkova, Kristina, Selena Chen, Constantinos Sioutas, et al.. (2025). Air pollution decreases postsynaptic PSD-95 and NMDA receptor subunits in synaptosomes from mouse cerebral cortex. Environmental Pollution. 383. 126845–126845. 1 indexed citations

Arevalo, José A., Max A. Thorwald, Kaitlin Allen, et al.. (2025). Age-related declines in mitochondrial Prdx6 contribute to dysregulated muscle bioenergetics. Redox Biology. 86. 103808–103808.

Thorwald, Max A., Gilberto Garcia, Justine Silva, et al.. (2025). Iron‐associated lipid peroxidation in Alzheimer's disease is increased in lipid rafts with decreased ferroptosis suppressors, tested by chelation in mice. Alzheimer s & Dementia. 21(1). e14541–e14541. 14 indexed citations

Hruby, Adam, Gilberto Garcia, Max A. Thorwald, et al.. (2025). Beyond genes and environment: mapping biological stochasticity in aging. GeroScience. 47(3). 2835–2850.

Thorwald, Max A., et al.. (2025). Down syndrome with Alzheimer's disease brains have increased iron and associated lipid peroxidation consistent with ferroptosis. Alzheimer s & Dementia. 21(S1). e105930–e105930.

Thorwald, Max A., Gilberto Garcia, Minhoo Kim, et al.. (2025). Down syndrome with Alzheimer's disease brains have increased iron and associated lipid peroxidation consistent with ferroptosis. Alzheimer s & Dementia. 21(6). e70322–e70322. 3 indexed citations

Thorwald, Max A., et al.. (2024). Deferoxamine treatment decreases amyloid fibrils and lowers iron mediated oxidative damage in ApoEFAD mice. Alzheimer s & Dementia. 20(S8). 1 indexed citations

Thorwald, Max A., Mafalda Cacciottolo, Carla D’Agostino, et al.. (2024). Air pollution amyloidogenesis is attenuated by the gamma‐secretase modulator GSM‐15606. Alzheimer s & Dementia. 20(9). 6107–6114. 3 indexed citations

Thorwald, Max A., et al.. (2024). Activation of the muscle-to-brain axis ameliorates neurocognitive deficits in an Alzheimer’s disease mouse model via enhancing neurotrophic and synaptic signaling. GeroScience. 47(2). 1593–1613. 5 indexed citations

10.

D’Agostino, Carla, Max A. Thorwald, Lindsay Meyerdirk, et al.. (2023). Air pollution nanoparticle and alpha-synuclein fibrils synergistically decrease glutamate receptor A1, depending upon nPM batch activity. Heliyon. 9(4). e15622–e15622. 9 indexed citations

11.

Thorwald, Max A., et al.. (2023). ApoE4 is associated with higher lipid peroxidation but not protein nitration in AD brains. Alzheimer s & Dementia. 19(S13).

12.

Higuchi‐Sanabria, Ryo, et al.. (2023). A tale of two pathways: Regulation of proteostasis by UPRmt and MDPs. Current Opinion in Neurobiology. 78. 102673–102673. 1 indexed citations

13.

Thorwald, Max A., et al.. (2022). Glucose Increases Hepatic Mitochondrial Antioxidant Enzyme Activities in Insulin Resistant Rats Following Chronic Angiotensin Receptor Blockade. International Journal of Molecular Sciences. 23(18). 10897–10897. 2 indexed citations

14.

Thorwald, Max A., Justine Silva, Elizabeth Head, & Caleb E. Finch. (2022). Amyloid futures in the expanding pathology of brain aging and dementia. Alzheimer s & Dementia. 19(6). 2605–2617. 12 indexed citations

15.

Thorwald, Max A., David Y. Hui, Akira Nishiyama, et al.. (2021). Chronic angiotensin receptor activation promotes hepatic triacylglycerol accumulation during an acute glucose challenge in obese-insulin-resistant OLETF rats. Endocrine. 75(1). 92–107. 10 indexed citations

16.

Haghani, Amin, Nikoo Safi, Hongqiao Zhang, et al.. (2020). Toxicity of urban air pollution particulate matter in developing and adult mouse brain: Comparison of total and filter-eluted nanoparticles. Environment International. 136. 105510–105510. 64 indexed citations

17.

Dhillon, Jaapna, et al.. (2020). Comparison of hand-held acoustic Doppler with point-of-care portable color Doppler ultrasound in the assessment of venous reflux disease. Journal of Vascular Surgery Venous and Lymphatic Disorders. 8(5). 831–839.e2. 4 indexed citations

18.

Rodríguez, Rubén, Andrew Lee, Keisa W. Mathis, et al.. (2018). Angiotensin receptor and tumor necrosis factor-α activation contributes to glucose intolerance independent of systolic blood pressure in obese rats. American Journal of Physiology-Renal Physiology. 315(4). F1081–F1090. 5 indexed citations

19.

Thorwald, Max A., Mostofa Jamal, Hiroshi Kinoshita, et al.. (2018). Nrf2-related gene expression is impaired during a glucose challenge in type II diabetic rat hearts. Free Radical Biology and Medicine. 130. 306–317. 15 indexed citations

20.

Thorwald, Max A., Rubén Rodríguez, Andrew Lee, et al.. (2017). Angiotensin receptor blockade improves cardiac mitochondrial activity in response to an acute glucose load in obese insulin resistant rats. Redox Biology. 14. 371–378. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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