Matthew I. Wahl

6.6k total citations · 2 hit papers
32 papers, 5.3k citations indexed

About

Matthew I. Wahl is a scholar working on Molecular Biology, Immunology and Genetics. According to data from OpenAlex, Matthew I. Wahl has authored 32 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 14 papers in Immunology and 13 papers in Genetics. Recurrent topics in Matthew I. Wahl's work include Protein Kinase Regulation and GTPase Signaling (15 papers), Chronic Lymphocytic Leukemia Research (12 papers) and Immunodeficiency and Autoimmune Disorders (9 papers). Matthew I. Wahl is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (15 papers), Chronic Lymphocytic Leukemia Research (12 papers) and Immunodeficiency and Autoimmune Disorders (9 papers). Matthew I. Wahl collaborates with scholars based in United States, Japan and United Kingdom. Matthew I. Wahl's co-authors include Sue Goo Rhee, S Nishibe, Graham Carpenter, Owen N. Witte, G Carpenter, Thomas O. Daniel, R. Kato, David J. Rawlings, Andrew M. Scharenberg and S.M. Teresa Hernández‐Sotomayor and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Matthew I. Wahl

32 papers receiving 5.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Increase of the Catalytic Activity of Phospholipase C-γ1 by Tyrosine Phosphorylation

1990 576 citations S Nishibe, Matthew I. Wahl et al. Science profile →
Prostate stem cell antigen: A cell surface marker overexpressed in prostate cancer

1998 556 citations Robert E. Reiter, Tetsuro Watabe et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Matthew I. Wahl United States	29	3.3k	1.7k	873	845	683	32	5.3k
J. Frederic Mushinski United States	36	3.3k 1.0×	1.4k 0.8×	285 0.3×	636 0.8×	493 0.7×	117	4.7k
Andreas Batzer United States	24	5.7k 1.7×	1.1k 0.6×	466 0.5×	1.7k 2.1×	976 1.4×	26	7.4k
Brian Duckworth United States	12	3.4k 1.0×	926 0.5×	272 0.3×	914 1.1×	1.2k 1.7×	15	4.9k
Ritu Dhand United Kingdom	21	4.8k 1.5×	865 0.5×	437 0.5×	1.0k 1.2×	1.5k 2.2×	26	6.1k
Christine Ellis Canada	20	4.2k 1.3×	796 0.5×	272 0.3×	1.2k 1.4×	1.1k 1.6×	31	5.5k
Rosana Kapeller United States	26	4.1k 1.2×	1.0k 0.6×	271 0.3×	1.1k 1.3×	1.0k 1.5×	41	5.9k
Kurt R. Auger United States	22	4.1k 1.2×	661 0.4×	320 0.4×	1.3k 1.5×	1.0k 1.5×	33	5.4k
Jaime A. Escobedo United States	26	5.4k 1.6×	771 0.5×	313 0.4×	1.2k 1.5×	998 1.5×	38	7.0k
Daniel Afar United States	33	2.0k 0.6×	835 0.5×	822 0.9×	1.1k 1.3×	266 0.4×	68	4.0k
Jane McGlade Canada	23	5.0k 1.5×	1.2k 0.7×	166 0.2×	1.4k 1.7×	1.1k 1.6×	26	6.6k

Countries citing papers authored by Matthew I. Wahl

Since Specialization

Citations

This map shows the geographic impact of Matthew I. Wahl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew I. Wahl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew I. Wahl more than expected).

Fields of papers citing papers by Matthew I. Wahl

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew I. Wahl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew I. Wahl. The network helps show where Matthew I. Wahl may publish in the future.

Co-authorship network of co-authors of Matthew I. Wahl

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew I. Wahl. A scholar is included among the top collaborators of Matthew I. Wahl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew I. Wahl. Matthew I. Wahl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Guo, Shuling, Matthew I. Wahl, & Owen N. Witte. (2005). Mutational analysis of the SH2-kinase linker region of Bruton's tyrosine kinase defines alternative modes of regulation for cytoplasmic tyrosine kinase families. International Immunology. 18(1). 79–87. 16 indexed citations

Fujimoto, Manabu, Jonathan C. Poe, Anne B. Satterthwaite, et al.. (2002). Complementary Roles for CD19 and Bruton’s Tyrosine Kinase in B Lymphocyte Signal Transduction. The Journal of Immunology. 168(11). 5465–5476. 41 indexed citations

Tomlinson, Michael G., Douglas Woods, Martin McMahon, et al.. (2001). A conditional form of Bruton's tyrosine kinase is sufficient to activate multiple downstream signaling pathways via PLC Gamma 2 in B cells. BMC Immunology. 2(1). 4–4. 53 indexed citations

Kouro, Taku, Kisaburo Nagata, Satoshi Takaki, et al.. (2001). Bruton's tyrosine kinase is required for signaling the CD79b-mediated pro-B to pre-B cell transition. International Immunology. 13(4). 485–493. 26 indexed citations

Guo, Beichu, R. Kato, Maria Garcia-Lloret, Matthew I. Wahl, & David J. Rawlings. (2000). Engagement of the Human Pre-B Cell Receptor Generates a Lipid Raft–Dependent Calcium Signaling Complex. Immunity. 13(2). 243–253. 196 indexed citations

Nisitani, Sazuku, R. Kato, David J. Rawlings, Owen N. Witte, & Matthew I. Wahl. (1999). In situdetection of activated Bruton’s tyrosine kinase in the Ig signaling complex by phosphopeptide-specific monoclonal antibodies. Proceedings of the National Academy of Sciences. 96(5). 2221–2226. 46 indexed citations

Weng, Zhigang, Sazuku Nisitani, Matthew I. Wahl, et al.. (1998). A DNA damage and stress inducible G protein-coupled receptor blocks cells in G₂/M. Proceedings of the National Academy of Sciences. 95(21). 12334–12339. 129 indexed citations

Li, Zuomei, R. Kato, Matthew I. Wahl, et al.. (1998). Btk/Tec kinases regulate sustained increases in intracellular Ca2+ following B-cell receptor activation. The EMBO Journal. 17(7). 1973–1985. 350 indexed citations

Reiter, Robert E., Tetsuro Watabe, George Thomas, et al.. (1998). Prostate stem cell antigen: A cell surface marker overexpressed in prostate cancer. Proceedings of the National Academy of Sciences. 95(4). 1735–1740. 556 indexed citations breakdown →

10.

Wahl, Matthew I., Anne-Catherine Fluckiger, R. Kato, et al.. (1997). Phosphorylation of two regulatory tyrosine residues in the activation of Bruton’s tyrosine kinase via alternative receptors. Proceedings of the National Academy of Sciences. 94(21). 11526–11533. 116 indexed citations

11.

Rawlings, David J., Andrew M. Scharenberg, Hyunsun Park, et al.. (1996). Activation of BTK by a Phosphorylation Mechanism Initiated by SRC Family Kinases. Science. 271(5250). 822–825. 386 indexed citations

12.

Park, Hyunsun, Matthew I. Wahl, Daniel Afar, et al.. (1996). Regulation of Btk Function by a Major Autophosphorylation Site Within the SH3 Domain. Immunity. 4(5). 515–525. 262 indexed citations

13.

Wahl, Matthew I. & Graham Carpenter. (1991). Selective phospholipase C activation. BioEssays. 13(3). 107–113. 42 indexed citations

14.

Wahl, Matthew I., et al.. (1990). Identification of two epidermal growth factor-sensitive tyrosine phosphorylation sites of phospholipase C-gamma in intact HSC-1 cells.. Journal of Biological Chemistry. 265(7). 3944–3948. 117 indexed citations

15.

Nishibe, S, Matthew I. Wahl, S.M. Teresa Hernández‐Sotomayor, et al.. (1990). Increase of the Catalytic Activity of Phospholipase C-γ1 by Tyrosine Phosphorylation. Science. 250(4985). 1253–1256. 576 indexed citations breakdown →

16.

Sim, Soyeong, et al.. (1990). Tyrosine residues in bovine phospholipase C-gamma phosphorylated by the epidermal growth factor receptor in vitro.. Journal of Biological Chemistry. 265(7). 3940–3943. 151 indexed citations

17.

Wahl, Matthew I., S Nishibe, Pann‐Ghill Suh, Sue Goo Rhee, & Guy H. Carpenter. (1989). Epidermal growth factor stimulates tyrosine phosphorylation of phospholipase C-II independently of receptor internalization and extracellular calcium.. Proceedings of the National Academy of Sciences. 86(5). 1568–1572. 340 indexed citations

18.

Wahl, Matthew I., et al.. (1989). Platelet-derived growth factor (PDGF) binding promotes physical association of PDGF receptor with phospholipase C.. Proceedings of the National Academy of Sciences. 86(21). 8232–8236. 207 indexed citations

19.

Wahl, Matthew I. & G Carpenter. (1988). Regulation of epidermal growth factor-stimulated formation of inositol phosphates in A-431 cells by calcium and protein kinase C.. Journal of Biological Chemistry. 263(16). 7581–7590. 116 indexed citations

20.

Wahl, Matthew I., J. David Sweatt, & Graham Carpenter. (1987). Epidermal growth factor (EGF) stimulates inositol trisphosphate formation in cells which overexpress the EGF receptor. Biochemical and Biophysical Research Communications. 142(3). 688–695. 92 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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