Hajime Karasuyama

23.2k total citations · 6 hit papers
214 papers, 17.3k citations indexed

About

Hajime Karasuyama is a scholar working on Immunology, Immunology and Allergy and Physiology. According to data from OpenAlex, Hajime Karasuyama has authored 214 papers receiving a total of 17.3k indexed citations (citations by other indexed papers that have themselves been cited), including 154 papers in Immunology, 65 papers in Immunology and Allergy and 50 papers in Physiology. Recurrent topics in Hajime Karasuyama's work include Immune Cell Function and Interaction (60 papers), Mast cells and histamine (60 papers) and T-cell and B-cell Immunology (57 papers). Hajime Karasuyama is often cited by papers focused on Immune Cell Function and Interaction (60 papers), Mast cells and histamine (60 papers) and T-cell and B-cell Immunology (57 papers). Hajime Karasuyama collaborates with scholars based in Japan, United States and Switzerland. Hajime Karasuyama's co-authors include Fritz Melchers, Keisuke Kuida, Richard A. Flavell, Pasko Rakić, Chia‐Yi Kuan, Yoshiyuki Minegishi, Kaori Mukai, Kazushige Obata‐Ninomiya, Kensuke Miyake and Akira Kudō and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Hajime Karasuyama

212 papers receiving 17.0k citations

Hit Papers

Decreased apoptosis in the brain and premature lethality ... 1988 2026 2000 2013 1996 1998 1988 1990 2007 500 1000 1.5k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice
    1996 1.6k citations Keisuke Kuida, Chia‐Yi Kuan et al. Nature profile →
  2. Reduced Apoptosis and Cytochrome c–Mediated Caspase Activation in Mice Lacking Caspase 9
    1998 1.4k citations Keisuke Kuida, Tarik F. Haydar et al. Cell profile →
  3. Establishment of mouse cell lines which constitutively secrete large quantities of interleukin 2, 3, 4 or 5, using modified cDNA expression vectors
    1988 1.0k citations Hajime Karasuyama, Fritz Melchers European Journal of Immunology profile →
  4. Interleukin-2 production by tumor cells bypasses T helper function in the generation of an antitumor response
    1990 769 citations Hajime Karasuyama et al. Cell profile →
  5. Dominant-negative mutations in the DNA-binding domain of STAT3 cause hyper-IgE syndrome
    2007 704 citations Yoshiyuki Minegishi, Masako Saitō et al. Nature profile →
  6. Treatment of Established Renal Cancer by Tumor Cells Engineered to Secrete Interleukin-4
    1991 605 citations Hajime Karasuyama et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hajime Karasuyama Japan 64 10.4k 5.4k 2.6k 2.4k 2.2k 214 17.3k
Clifford A. Lowell United States 85 11.8k 1.1× 6.5k 1.2× 4.1k 1.6× 2.7k 1.1× 1.9k 0.9× 227 20.7k
Angel F. López Australia 63 6.9k 0.7× 3.6k 0.7× 1.5k 0.6× 2.9k 1.2× 2.3k 1.0× 275 13.7k
Carlos Martı́nez-A Spain 81 9.4k 0.9× 8.6k 1.6× 2.1k 0.8× 5.3k 2.3× 1.6k 0.7× 331 20.5k
Hitoshi Kikutani Japan 65 9.2k 0.9× 4.4k 0.8× 1.3k 0.5× 3.9k 1.7× 1.1k 0.5× 186 17.0k
Kazuo Sugamura Japan 78 12.2k 1.2× 4.9k 0.9× 894 0.3× 4.0k 1.7× 1.8k 0.8× 304 20.8k
J. Fernando Bazán United States 58 10.5k 1.0× 8.1k 1.5× 1.1k 0.4× 4.8k 2.0× 1.6k 0.7× 95 22.1k
Chikao Morimoto United States 74 8.4k 0.8× 5.1k 0.9× 2.7k 1.0× 5.6k 2.4× 914 0.4× 339 18.5k
David R. Greaves United Kingdom 67 7.1k 0.7× 7.1k 1.3× 1.0k 0.4× 3.5k 1.5× 1.6k 0.7× 183 16.9k
Steven Dower United States 72 10.7k 1.0× 5.5k 1.0× 1.2k 0.5× 2.1k 0.9× 1.1k 0.5× 192 17.5k
Nancy H. Ruddle United States 67 9.1k 0.9× 2.7k 0.5× 1.2k 0.5× 2.9k 1.3× 925 0.4× 174 14.2k

Countries citing papers authored by Hajime Karasuyama

Since Specialization
Citations

This map shows the geographic impact of Hajime Karasuyama's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hajime Karasuyama with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hajime Karasuyama more than expected).

Fields of papers citing papers by Hajime Karasuyama

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hajime Karasuyama. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hajime Karasuyama. The network helps show where Hajime Karasuyama may publish in the future.

Co-authorship network of co-authors of Hajime Karasuyama

This figure shows the co-authorship network connecting the top 25 collaborators of Hajime Karasuyama. A scholar is included among the top collaborators of Hajime Karasuyama based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hajime Karasuyama. Hajime Karasuyama is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Leyva-Castillo, Juan Manuel, Mrinmoy Das, Christy Kam, et al.. (2024). IL-4 acts on skin-derived dendritic cells to promote the TH2 response to cutaneous sensitization and the development of allergic skin inflammation. Journal of Allergy and Clinical Immunology. 154(6). 1462–1471.e3. 10 indexed citations
2.
Yoshikawa, Soichiro, Kensuke Miyake, Atsunori Kamiya, & Hajime Karasuyama. (2020). The role of basophils in acquired protective immunity to tick infestation. Parasite Immunology. 43(5). 12 indexed citations
3.
Kawano, Yohei, Georg Petkau, Christina Stehle, et al.. (2018). Stable lines and clones of long-term proliferating normal, genetically unmodified murine common lymphoid progenitors. Blood. 131(18). 2026–2035. 8 indexed citations
4.
Miyake, Kensuke, et al.. (2017). Trogocytosis of peptide–MHC class II complexes from dendritic cells confers antigen-presenting ability on basophils. Proceedings of the National Academy of Sciences. 114(5). 1111–1116. 102 indexed citations
5.
Sato, Shingo, et al.. (2016). Basophil tryptase mMCP-11 plays a crucial role in IgE-mediated, delayed-onset allergic inflammation in mice. Blood. 128(25). 2909–2918. 22 indexed citations
6.
Obata‐Ninomiya, Kazushige, Kenji Ishiwata, Yuichiro Nei, et al.. (2013). The skin is an important bulwark of acquired immunity against intestinal helminths. The Journal of Experimental Medicine. 210(12). 2583–2595. 112 indexed citations
7.
Torrero, Marina N., Marc P. Hübner, David Larson, Hajime Karasuyama, & Edward Mitre. (2010). Basophils Amplify Type 2 Immune Responses, but Do Not Serve a Protective Role, during Chronic Infection of Mice with the Filarial Nematode Litomosoides sigmodontis. The Journal of Immunology. 185(12). 7426–7434. 41 indexed citations
8.
Ugajin, Tsukasa, Toshiyuki Kojima, Kaori Mukai, et al.. (2009). Basophils preferentially express mouse mast cell protease 11 among the mast cell tryptase family in contrast to mast cells. Journal of Leukocyte Biology. 86(6). 1417–1425. 87 indexed citations
9.
Obata‐Ninomiya, Kazushige, Kaori Mukai, Yusuke Tsujimura, et al.. (2007). Basophils are essential initiators of a novel type of chronic allergic inflammation. Blood. 110(3). 913–920. 239 indexed citations
10.
Minegishi, Yoshiyuki, Masako Saitō, Shigeru Tsuchiya, et al.. (2007). Dominant-negative mutations in the DNA-binding domain of STAT3 cause hyper-IgE syndrome. Nature. 448(7157). 1058–1062. 704 indexed citations breakdown →
11.
12.
Toyama‐Sorimachi, Noriko, Yoshiki Omatsu, Yusuke Tsujimura, et al.. (2005). Inhibitory NK Receptor Ly49Q Is Expressed on Subsets of Dendritic Cells in a Cellular Maturation- and Cytokine Stimulation-Dependent Manner. The Journal of Immunology. 174(8). 4621–4629. 38 indexed citations
13.
Taguchi, Tomoko, Nobutaka Kiyokawa, Toyo Suzuki, et al.. (2003). Pre-B Cell Antigen Receptor-Mediated Signal Inhibits CD24-Induced Apoptosis in Human Pre-B Cells. The Journal of Immunology. 170(1). 252–260. 46 indexed citations
14.
Toyama‐Sorimachi, Noriko, Hideo Yagita∥, Fujiko Kitamura, et al.. (2001). Mouse CD94 Participates in Qa-1-Mediated Self Recognition by NK Cells and Delivers Inhibitory Signals Independent of Ly-49. The Journal of Immunology. 166(6). 3771–3779. 17 indexed citations
15.
Stephan, Robert P., et al.. (2001). Analysis of VpreB Expression During B Lineage Differentiation in λ5-Deficient Mice. The Journal of Immunology. 167(7). 3734–3739. 13 indexed citations
16.
Kuida, Keisuke, Tarik F. Haydar, Chia‐Yi Kuan, et al.. (1998). Reduced Apoptosis and Cytochrome c–Mediated Caspase Activation in Mice Lacking Caspase 9. Cell. 94(3). 325–337. 1369 indexed citations breakdown →
17.
Karasuyama, Hajime, et al.. (1996). Surrogate Light Chain in B Cell Development. Advances in immunology. 63. 1–41. 90 indexed citations
18.
Rolink, Antonius, Hajime Karasuyama, Ulf Grawunder, et al.. (1993). B cell development in mice with a defective λ5 gene. European Journal of Immunology. 23(6). 1284–1288. 84 indexed citations
19.
Karasuyama, Hajime & Fritz Melchers. (1988). Establishment of mouse cell lines which constitutively secrete large quantities of interleukin 2, 3, 4 or 5, using modified cDNA expression vectors. European Journal of Immunology. 18(1). 97–104. 1032 indexed citations breakdown →
20.
Karasuyama, Hajime. (1988). [Establishment of mouse cell lines which constitutively secrete large quantities of interleukin 2, 3, 4 or 5, using high-copy cDNA expression vectors].. PubMed. 33(14). 2527–32. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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