Matthew C. Madison

873 total citations · 1 hit paper
16 papers, 529 citations indexed

About

Matthew C. Madison is a scholar working on Immunology, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Matthew C. Madison has authored 16 papers receiving a total of 529 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 7 papers in Molecular Biology and 6 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Matthew C. Madison's work include IL-33, ST2, and ILC Pathways (5 papers), Extracellular vesicles in disease (4 papers) and Chronic Obstructive Pulmonary Disease (COPD) Research (4 papers). Matthew C. Madison is often cited by papers focused on IL-33, ST2, and ILC Pathways (5 papers), Extracellular vesicles in disease (4 papers) and Chronic Obstructive Pulmonary Disease (COPD) Research (4 papers). Matthew C. Madison collaborates with scholars based in United States, Netherlands and France. Matthew C. Madison's co-authors include Farrah Kheradmand, David B. Corry, Cameron Landers, Hui-Ying Tung, Brian E. Gilbert, M J Hong, Ran You, Paul Porter, Cheng‐Yen Chang and Matthew J. Campen and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and The Journal of Immunology.

In The Last Decade

Matthew C. Madison

14 papers receiving 526 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Electronic cigarettes disrupt lung lipid homeostasis and innate immunity independent of nicotine

2019 254 citations Matthew C. Madison, Cameron Landers et al. Journal of Clinical Investigation profile →

Peers

Matthew C. Madison

PHYSI

PRM

IMMUN

HTM

Lisa Jolly United Kingdom

Monica Goldklang United States

Loubna Akhabir Canada

Susan Waserman Canada

Amir Bar‐Shai Israel

Jianmiao Wang China

A.P. McLaughlin United States

Lei Putney United States

Alexandra M. Nanzer United Kingdom

Waldo Mattos Brazil

Lisa Jolly United Kingdom

Matthew C. Madison

295 ×1.4

PHYSI

267 ×1.5

PRM

145 ×1.2

145 ×1.7

IMMUN

36 ×0.5

HTM

Citations per year, relative to Matthew C. Madison Matthew C. Madison (= 1×) peers Lisa Jolly

Countries citing papers authored by Matthew C. Madison

Since Specialization

Citations

This map shows the geographic impact of Matthew C. Madison's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew C. Madison with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew C. Madison more than expected).

Fields of papers citing papers by Matthew C. Madison

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew C. Madison. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew C. Madison. The network helps show where Matthew C. Madison may publish in the future.

Co-authorship network of co-authors of Matthew C. Madison

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew C. Madison. A scholar is included among the top collaborators of Matthew C. Madison based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew C. Madison. Matthew C. Madison is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Blalock, J. Edwin, et al.. (2025). E-cigarette vapor amplifies neutrophilic inflammation and proteolytic EVs in response to LPS. Frontiers in Immunology. 16. 1665936–1665936.

Madison, Matthew C., Camilla Margaroli, Kristopher R. Genschmer, et al.. (2023). Protease-armed, Pathogenic Extracellular Vesicles Link Smoking and Chronic Obstructive Pulmonary Disease. American Journal of Respiratory and Critical Care Medicine. 208(10). 1115–1125. 7 indexed citations

Margaroli, Camilla, Nirmal Sharma, Siddharth Patel, et al.. (2023). Interferon-dependent signaling is critical for viral clearance in airway neutrophils. JCI Insight. 8(10). 6 indexed citations

Genschmer, Kristopher R., Matthew C. Madison, Liliana Viera, et al.. (2023). Therapeutic effect of two strategies directed at disruption of pathogenic neutrophil extracellular vesicles in a murine emphysema model. American Journal of Physiology-Lung Cellular and Molecular Physiology. 324(5). L694–L699. 3 indexed citations

Margaroli, Camilla, Matthew C. Madison, Liliana Viera, et al.. (2022). An in vivo model for extracellular vesicle–induced emphysema. JCI Insight. 7(4). 24 indexed citations

Margaroli, Camilla, Nirmal Sharma, Matthew C. Madison, et al.. (2021). Spatial mapping of SARS-CoV-2 and H1N1 lung injury identifies differential transcriptional signatures. Cell Reports Medicine. 2(4). 100242–100242. 30 indexed citations

Margaroli, Camilla, Nirmal Sharma, Matthew C. Madison, et al.. (2020). Spatial Mapping of SARS-CoV-2 and H1N1 Influenza Lung Injury Identifies Differential Transcriptional Signatures. SSRN Electronic Journal.

Madison, Matthew C., M J Hong, Xiaoyi Yuan, et al.. (2019). A Novel Animal Model of Emphysema Induced by Anti-Elastin Autoimmunity. The Journal of Immunology. 203(2). 349–359. 6 indexed citations

Yuan, Xiaoyi, Cheng-Yen Chang, Ran You, et al.. (2019). Cigarette smoke–induced reduction of C1q promotes emphysema. JCI Insight. 4(13). 30 indexed citations

10.

Madison, Matthew C., Cameron Landers, Cheng‐Yen Chang, et al.. (2019). Electronic cigarettes disrupt lung lipid homeostasis and innate immunity independent of nicotine. Journal of Clinical Investigation. 129(10). 4290–4304. 254 indexed citations breakdown →

11.

Landers, Cameron, Hui-Ying Tung, John Knight, et al.. (2019). Selective cleavage of fibrinogen by diverse proteinases initiates innate allergic and antifungal immunity through CD11b. Journal of Biological Chemistry. 294(22). 8834–8847. 30 indexed citations

12.

Kim, Myunghoo, Bon‐Hee Gu, Matthew C. Madison, et al.. (2019). Cigarette Smoke Induces Intestinal Inflammation via a Th17 Cell-Neutrophil Axis. Frontiers in Immunology. 10. 75–75. 31 indexed citations

13.

Madison, Matthew C., et al.. (2018). Laryngeal inflammatory response to smoke and vape in a murine model. American Journal of Otolaryngology. 40(1). 89–92. 14 indexed citations

14.

Knight, John, Pijus K. Mandal, Pietro Morlacchi, et al.. (2018). Small molecule targeting of the STAT5/6 Src homology 2 (SH2) domains to inhibit allergic airway disease. Journal of Biological Chemistry. 293(26). 10026–10040. 22 indexed citations

15.

Gu, Bon‐Hee, Matthew C. Madison, David B. Corry, & Farrah Kheradmand. (2018). Matrix remodeling in chronic lung diseases. Matrix Biology. 73. 52–63. 37 indexed citations

16.

Hong, M J, Bon‐Hee Gu, Matthew C. Madison, et al.. (2017). Protective role of γδ T cells in cigarette smoke and influenza infection. Mucosal Immunology. 11(3). 894–908. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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