Mathilde Raverdeau

2.7k citations

20 papers · 1.9k indexed · 1 hit paper · h-index 18

Impact in

Immunology top 2%
- Immune Cell Function and Interaction
- IL-33, ST2, and ILC Pathways
- T-cell and B-cell Immunology
- Immune cells in cancer
Reproductive Medicine top 5%
- Sperm and Testicular Function

Papers in

Immunology 13
- Immune Cell Function and Interaction 9
- T-cell and B-cell Immunology 7
- Immunotherapy and Immune Responses 4
- IL-33, ST2, and ILC Pathways 2
Reproductive Medicine 3
- Sperm and Testicular Function 3

Co-authors: Kingston H. G. Mills Alicja Misiak Lydia Lynch Christine Dennefeld Norbert B. Ghyselinck Aurore Gély-Pernot Betty Féret Lydia Dyck
Journals: The Journal of Immunology (3 papers)The Journal of Experimental Medicine (2 papers)Scientific Reports (1 paper)Endocrinology (1 paper)Journal of Autoimmunity (1 paper)
Partner nations: Ireland United States Canada

In The Last Decade

Mathilde Raverdeau

20 papers receiving 1.9k citations

Hit Papers

align trajectories log scale

Metabolic reprogramming of natural killer cells in obesity limits antitumor responses 2018 · 448 citations

Peers

Countries citing papers authored by Mathilde Raverdeau

Since Specialization

Citations

This map shows the geographic impact of Mathilde Raverdeau's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mathilde Raverdeau with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mathilde Raverdeau more than expected).

Fields of papers citing papers by Mathilde Raverdeau

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mathilde Raverdeau. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mathilde Raverdeau. The network helps show where Mathilde Raverdeau may publish in the future.

Co-authors

The 25 scholars most cited alongside Mathilde Raverdeau, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mathilde Raverdeau Line = papers co-authored together Mathilde Raverdeau links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	Suppressive effects of the obese tumor microenvironment on CD8 T cell infiltration and effector function The Journal of Experimental Medicine ·Lydia Dyck, Hannah Prendeville, Mathilde Raverdeau, Mieszko M. Wilk, Róisín M. Loftus, Aaron Douglas, Janet McCormack, Bruce Moran, Michael Wilkinson, Evanna L. Mills, Michael J. Doughty, Aurélie Fabre, Helen Heneghan, Rahul Shivarkar, Andrew E. Hogan, Edward T. Chouchani, Donal O’Shea, Donal J. Brennan, Lydia Lynch	2022	60
2	Lactobacillus salivarius UCC118™ Dampens Inflammation and Promotes Microbiota Recovery to Provide Therapeutic Benefit in a DSS-Induced Colitis Model Microorganisms ·Namrata Iyer, Michelle A. Williams, Amy O’Callaghan, Elaine Dempsey, Raúl Cabrera‐Rubio, Mathilde Raverdeau, Fiona Crispie, Paul D. Cotter, Sinéad C. Corr	2022	17
3	Anti-inflammatory Trained Immunity Mediated by Helminth Products Attenuates the Induction of T Cell-Mediated Autoimmune Disease Frontiers in Immunology ·リチャード E. ウォード, Kyle T. Cunningham, Mathilde Raverdeau, Robert Walsh, Lucy Curham, Anna Malara, Kingston H. G. Mills	2019	50
4	γδ T cells in cancer: a small population of lymphocytes with big implications Clinical & Translational Immunology ·Mathilde Raverdeau, TonyMokBMScMD, Cathal Harmon, Lydia Lynch	2019	64
5	Retinoic acid‐induced autoantigen‐specific type 1 regulatory T cells suppress autoimmunity EMBO Reports ·Mathilde Raverdeau, Reshma Shaheen, Anna Malara, Mieszko M. Wilk, Alicja Misiak, Lucia Kuffová, Yu Tian, Aoife McGinley, リチャード E. ウォード, Chandirasegaran Massilamany, Jay Reddy, John V. Forrester, Kingston H. G. Mills	2019	27
6	Metabolic reprogramming of natural killer cells in obesity limits antitumor responses Nature Immunology ·Xavier Michelet, Lydia Dyck, Andrew E. Hogan, Róisín M. Loftus, Danielle Duquette, Kevin Wei, Semir Beyaz, Ali Tavakkoli, Cathriona Foley, Raymond P. Donnelly, Cliona O’Farrelly, Mathilde Raverdeau, Ashley H. Vernon, Kamil Mülayim, Donal O’Shea, Barbara S. Nikolajczyk, Kingston H. G. Mills, Michael B. Brenner, David K. Finlay, Lydia Lynch Hit paper breakdown →	2018	448
7	Th17 cells, γδ T cells and their interplay in EAE and multiple sclerosis Journal of Autoimmunity ·Aoife McGinley, Sarah C. Edwards, Mathilde Raverdeau, Kingston H. G. Mills	2018	90
8	Loss of MicroRNA-21 Influences the Gut Microbiota, Causing Reduced Susceptibility in a Murine Model of Colitis Journal of Crohn s and Colitis ·Daniel Johnston, Michelle A. Williams, Christoph A. Thaiss, Raúl Cabrera‐Rubio, Mathilde Raverdeau, Craig P. McEntee, Paul D. Cotter, Eran Elinav, Luke O'neill, Sinéad C. Corr	2018	60
9	Loss of the molecular clock in myeloid cells exacerbates T cell-mediated CNS autoimmune disease Nature Communications ·Caroline E. Sutton, Conor M. Finlay, Mathilde Raverdeau, James O. Early, Joseph DeCourcey, Zbigniew Zasłona, Luke O'neill, Kingston H. G. Mills, Annie M. Curtis	2017	97
10	IL-17–Producing Innate and Pathogen-Specific Tissue Resident Memory γδ T Cells Expand in the Lungs of Bordetella pertussis–Infected Mice The Journal of Immunology ·Alicja Misiak, Mieszko M. Wilk, Mathilde Raverdeau, Kingston H. G. Mills	2016	78
11	Anti-PD-1 inhibits Foxp3+ Treg cell conversion and unleashes intratumoural effector T cells thereby enhancing the efficacy of a cancer vaccine in a mouse model Cancer Immunology Immunotherapy ·Lydia Dyck, Mieszko M. Wilk, Mathilde Raverdeau, Alicja Misiak, Louis Boon, Kingston H. G. Mills	2016	53
12	Development of a quantitative fluorescence-based ligand-binding assay Scientific Reports ·Kou‐Hsiung Lee, Mathilde Raverdeau, H. Paul Voorheis	2016	35
13	Retinoic acid suppresses IL‐17 production and pathogenic activity of γδ T cells in CNS autoimmunity Immunology and Cell Biology ·Mathilde Raverdeau, Kou‐Hsiung Lee, Alicja Misiak, Kingston H. G. Mills	2016	33
14	Retinoic Acid Receptors Control Spermatogonia Cell-Fate and Induce Expression of the SALL4A Transcription Factor PLoS Genetics ·Aurore Gély-Pernot, Mathilde Raverdeau, Marius Teletin, Nadège Vernet, Betty Féret, Muriel Klopfenstein, Christine Dennefeld, Irwin Davidson, G Benoît, Manuel Mark, Norbert B. Ghyselinck	2015	55
15	Protective Role for Caspase-11 during Acute Experimental Murine Colitis The Journal of Immunology ·和也下坊, Mathilde Raverdeau, Gabriella Aviello, Siobhan Wade, Valdete Noveli Rhoden, Katherine M. Sheehan, Sinéad C. Corr, Elaine W. Kay, Luke O'neill, Kingston H. G. Mills, Emma M. Creagh	2014	78
16	S-16 Cytokine ·Kingston H. G. Mills, Aideen C. Allen, Sarah C. Edwards, María Alejandra Gómez Paz Fort, Aoife McGinley, Alicja Misiak, Lara S. Dungan, Mathilde Raverdeau, Conor M. Finlay, Caroline E. Sutton	2014	3
17	Modulation of T Cell and Innate Immune Responses by Retinoic Acid The Journal of Immunology ·Mathilde Raverdeau, Kingston H. G. Mills	2014	161
18	Retinoic acid expression associates with enhanced IL-22 production by γδ T cells and innate lymphoid cells and attenuation of intestinal inflammation The Journal of Experimental Medicine ·Lisa A. Mielke, Sarah A. Jones, Mathilde Raverdeau, Rowan Higgs, Anna Stefańska, Joanna R. Groom, Alicja Misiak, Lara S. Dungan, Caroline E. Sutton, Gundula Streubel, Adrian P. Bracken, Kingston H. G. Mills	2013	234
19	Retinoic acid induces Sertoli cell paracrine signals for spermatogonia differentiation but cell autonomously drives spermatocyte meiosis Proceedings of the National Academy of Sciences ·Mathilde Raverdeau, Aurore Gély-Pernot, Betty Féret, Christine Dennefeld, G Benoît, Irwin Davidson, Pierre Chambon, Manuel Mark, Norbert B. Ghyselinck	2012	177
20	Spermatogonia Differentiation Requires Retinoic Acid Receptor γ Endocrinology ·Aurore Gély-Pernot, Mathilde Raverdeau, Catherine Célébi, Christine Dennefeld, Betty Féret, Muriel Klopfenstein, Shosei Yoshida, Norbert B. Ghyselinck, Manuel Mark	2011	100

About Mathilde Raverdeau

Mathilde Raverdeau is a scholar working on Immunology, Reproductive Medicine, Gastroenterology, Microbiology and Endocrine and Autonomic Systems, having authored 20 papers that have together received 1.9k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (9 papers), T-cell and B-cell Immunology (7 papers), Immunotherapy and Immune Responses (4 papers), Sperm and Testicular Function (3 papers), Reproductive Biology and Fertility (2 papers), IL-33, ST2, and ILC Pathways (2 papers), Cancer Immunotherapy and Biomarkers (2 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (2 papers). The work is most often cited by research in Immunology (940 citations), Reproductive Medicine (234 citations), Biological Psychiatry (34 citations), Cancer Research (198 citations) and Endocrine and Autonomic Systems (84 citations). Mathilde Raverdeau has collaborated with scholars based in Ireland, United States and Canada. Frequent co-authors include Kingston H. G. Mills, Alicja Misiak, Lydia Lynch, Christine Dennefeld, Norbert B. Ghyselinck, Aurore Gély-Pernot, Betty Féret, Lydia Dyck, Mieszko M. Wilk and Caroline E. Sutton. Their work appears in journals such as The Journal of Immunology, The Journal of Experimental Medicine, Scientific Reports, Endocrinology and Journal of Autoimmunity.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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