Massimo Sabatini
- Molecular Biology
- Oncology top 10%
- Rheumatology top 5%
- Hematology top 5%
- Cancer Research
- Co-authors
- Philippe PastoureauChristophe LesurFrédéric De CeuninckBrian G.M. DurieDonald R. BertoliniGregory R. MundyTimothy S. BringmanAlison Gillespie
- Topics
- Osteoarthritis Treatment and Mechanisms (13 papers)Protease and Inhibitor Mechanisms (7 papers)Bone health and treatments (6 papers)
- Cited by
- RheumatologyHematologyOncology
In The Last Decade
Massimo Sabatini
28 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 98
- Molecular Biology 415
- Oncology 342
- Rheumatology 234
- Hematology 164
- Cancer Research 137
Countries citing papers authored by Massimo Sabatini
This map shows the geographic impact of Massimo Sabatini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Massimo Sabatini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Massimo Sabatini more than expected).
Fields of papers citing papers by Massimo Sabatini
This network shows the impact of papers produced by Massimo Sabatini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Massimo Sabatini. The network helps show where Massimo Sabatini may publish in the future.
Co-authorship network of co-authors of Massimo Sabatini
This figure shows the co-authorship network connecting the top 25 collaborators of Massimo Sabatini. A scholar is included among the top collaborators of Massimo Sabatini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Massimo Sabatini. Massimo Sabatini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 17 | |
| 2 | 15 | |
| 3 | 3 | |
| 4 | 25 | |
| 5 | 33 | |
| 6 | 12 | |
| 7 | 20 | |
| 8 | 5 | |
| 9 | 68 | |
| 10 | Cellular and molecular tools | 4 |
| 11 | Structure and in vivo analysis | 3 |
| 12 | 43 | |
| 13 | 2 | |
| 14 | 63 | |
| 15 | 22 | |
| 16 | 11 | |
| 17 | 13 | |
| 18 | 55 | |
| 19 | 13 | |
| 20 | An experimental rat model of local bone cancer invasion and its responsiveness to ethane-1-hydroxy-1,1-bis(phosphonate). | 24 |
About Massimo Sabatini
Massimo Sabatini is a scholar working on Rheumatology, Immunology and Allergy and Oncology, having authored 28 papers that have together received 1.0k indexed citations. Recurring topics across this work include Osteoarthritis Treatment and Mechanisms (13 papers), Protease and Inhibitor Mechanisms (7 papers) and Bone health and treatments (6 papers). The work is most often cited by research in Rheumatology (234 citations), Hematology (164 citations) and Oncology (342 citations). Massimo Sabatini has collaborated with scholars based in France, Italy and Australia. Frequent co-authors include Philippe Pastoureau, Christophe Lesur, Frédéric De Ceuninck, Brian G.M. Durie, Donald R. Bertolini, Gregory R. Mundy, Timothy S. Bringman, Alison Gillespie, Glenn E. Nedwin and I. Ross Garrett. Their work appears in journals such as New England Journal of Medicine, PLoS ONE and Analytical Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.