Massimo Mantegazza

6.6k total citations · 1 hit paper
92 papers, 4.5k citations indexed

About

Massimo Mantegazza is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Psychiatry and Mental health. According to data from OpenAlex, Massimo Mantegazza has authored 92 papers receiving a total of 4.5k indexed citations (citations by other indexed papers that have themselves been cited), including 57 papers in Molecular Biology, 56 papers in Cellular and Molecular Neuroscience and 48 papers in Psychiatry and Mental health. Recurrent topics in Massimo Mantegazza's work include Neuroscience and Neuropharmacology Research (50 papers), Epilepsy research and treatment (44 papers) and Ion channel regulation and function (43 papers). Massimo Mantegazza is often cited by papers focused on Neuroscience and Neuropharmacology Research (50 papers), Epilepsy research and treatment (44 papers) and Ion channel regulation and function (43 papers). Massimo Mantegazza collaborates with scholars based in France, Italy and United States. Massimo Mantegazza's co-authors include Silvana Franceschetti, William A. Catterall, Sandrine Cestèle, Todd Scheuer, Frank H. Yu, G. Avanzini, Paolo Scalmani, William J. Spain, G. Stanley McKnight and Kimberly A. Burton and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Journal of Neuroscience.

In The Last Decade

Massimo Mantegazza

89 papers receiving 4.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Reduced sodium current in GABAergic interneurons in a mouse model of severe myoclonic epilepsy in infancy

2006 823 citations Massimo Mantegazza et al. profile →

Peers

Countries citing papers authored by Massimo Mantegazza

Since Specialization

Citations

This map shows the geographic impact of Massimo Mantegazza's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Massimo Mantegazza with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Massimo Mantegazza more than expected).

Fields of papers citing papers by Massimo Mantegazza

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Massimo Mantegazza. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Massimo Mantegazza. The network helps show where Massimo Mantegazza may publish in the future.

Co-authorship network of co-authors of Massimo Mantegazza

This figure shows the co-authorship network connecting the top 25 collaborators of Massimo Mantegazza. A scholar is included among the top collaborators of Massimo Mantegazza based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Massimo Mantegazza. Massimo Mantegazza is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Long-lasting astrocyte remodeling in Dravet Syndrome Scn1a ^+/– mouse model 2026 ·bioRxiv (Cold Spring Harbor Laboratory) ·(unknown), (unknown), (unknown), Marine Blaquière, (unknown), (unknown), (unknown), Massimo Mantegazza, Étienne Audinat, Nicola Andrea Marchi, Noémie Cresto	0
2	Artificial intelligence‐driven closed‐loop devices in sudden unexpected death in epilepsy prediction and prevention: Insights from persons with epilepsy and caregivers 2025 ·Epilepsia ·(unknown), (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Ricardo Matias, Fabrice Duprat, Massimo Mantegazza, Onur Parlak, Philippe Ryvlin, Sándor Beniczky, (unknown), Emilio Perucca, João Claro, Carlos Conde	0
3	The role of electroencephalography in epilepsy research—From seizures to interictal activity and comorbidities 2025 ·Epilepsia ·(unknown), Liset Menéndez de la Prida, Edward H. Bertram, Mark O. Cunningham, David C. Henshall, Anli Liu, Vadym Gnatkovsky, Simona Balestrini, Marco de Curtis, Aristea S. Galanopoulou, Julia Jacobs, John G. R. Jefferys, Massimo Mantegazza, Cristina R. Reschke, Přemysl Jiruška	4
4	Neurodevelopmental defects in Dravet syndrome Scn1a+/− mice: Targeting GABA-switch rescues behavioral dysfunctions but not seizures and mortality 2025 ·Neurobiology of Disease ·Lara Pizzamiglio, (unknown), (unknown), G Fossati, Elisabetta Menna, Isabelle Léna, Flavia Antonucci, Massimo Mantegazza	4
5	Preictal dysfunctions of inhibitory interneurons paradoxically lead to their rebound hyperactivity and to low-voltage-fast onset seizures in Dravet syndrome 2024 ·Proceedings of the National Academy of Sciences ·(unknown), Mathieu Kuchenbuch, (unknown), (unknown), María A. Zuluaga, Marco Lorenzi, Rima Nabbout, Massimo Mantegazza	6
6	Are we there yet? A critical evaluation of sudden and unexpected death in epilepsy models 2023 ·Epilepsia ·(unknown), George B. Richerson, (unknown), Fabrice Duprat, Massimo Mantegazza, Laurent Bezin, Sylvain Rheims	14
7	Involvement of GABAergic Interneuron Subtypes in 4-Aminopyridine-Induced Seizure-Like Events in Mouse Entorhinal Cortexin Vitro 2023 ·Journal of Neuroscience ·Paolo Scalmani, Rosina Paterra, Massimo Mantegazza, Massimo Avoli, Marco de Curtis	15
8	Rescuing epileptic and behavioral alterations in a Dravet syndrome mouse model by inhibiting eukaryotic elongation factor 2 kinase (eEF2K) 2022 ·Molecular Autism ·(unknown), Laura Gritti, Luisa Ponzoni, Paolo Scalmani, Massimo Mantegazza, Mariaelvina Sala, Chiara Verpelli, Carlo Sala	10
9	DNA damage and transcriptional regulation in iPSC-derived neurons from Ataxia Telangiectasia patients 2019 ·Scientific Reports ·Alessandro Corti, (unknown), Matteo Dugo, Raffaele Calogero, Benedetta Terragni, Massimo Mantegazza, Silvana Franceschetti, (unknown), Patrizia Gasparini, Daniele Lecis, Krystyńa Chrzańowska, Domenico Delia	13
10	A two-hit story: Seizures and genetic mutation interaction sets phenotype severity in SCN1A epilepsies 2019 ·Neurobiology of Disease ·(unknown), Fabrice Duprat, Paula A. Pousinha, Alexandre Loucif, Vincent Douchamps, Maria Cristina Regondi, Marion Ayrault, (unknown), Marion I. Stunault, Andrew Escayg, Romain Goutagny, Vadym Gnatkovsky, Carolina Frassoni, Hélène Marie, Ingrid Bethus, Massimo Mantegazza	45
11	The X-Linked Intellectual Disability Protein IL1RAPL1 Regulates Dendrite Complexity 2017 ·Journal of Neuroscience ·Caterina Montani, Mariana Ramos-Brossier, Luisa Ponzoni, Laura Gritti, Andrzej Cwetsch, Daniela Braida, Yoann Saillour, Benedetta Terragni, Massimo Mantegazza, Mariaelvina Sala, Chiara Verpelli, Pierre Billuart, Carlo Sala	34
12	The impact of genetic and experimental studies on classification and therapy of the epilepsies 2017 ·Neuroscience Letters ·G. Avanzini, Massimo Mantegazza, Benedetta Terragni, Laura Canafoglia, Paolo Scalmani, Silvana Franceschetti	8
13	Impaired Action Potential Initiation in GABAergic Interneurons Causes Hyperexcitable Networks in an Epileptic Mouse Model Carrying a Human Na(V)1.1 Mutation 2014 ·Journal of Neuroscience Nursing ·Ulrike B. S. Hedrich, (unknown), Daniel S. Kirschenbaum, (unknown), (unknown), Yuanyuan Liu, Stephan Theiss, (unknown), Andrew Escayg, Marcel Dihné, Heinz Beck, Massimo Mantegazza, Holger Lerche	4
14	Nonfunctional Na _V 1.1 familial hemiplegic migraine mutant transformed into gain of function by partial rescue of folding defects 2013 ·Proceedings of the National Academy of Sciences ·Sandrine Cestèle, Emanuele Schiavon, Raffaella Rusconi, Silvana Franceschetti, Massimo Mantegazza	79
15	Dravet syndrome: Insights from in vitro experimental models 2011 ·Epilepsia ·Massimo Mantegazza	13
16	Na⁺channelopathies and epilepsy: recent advances and new perspectives 2010 ·Expert Review of Clinical Pharmacology ·Carla Marini, Massimo Mantegazza	22
17	A rescuable folding defective Na_v1.1 (SCN1A) sodium channel mutant causes GEFS+: Common mechanism in Na_v1.1 related epilepsies? 2009 ·Human Mutation ·(unknown), Romina Combi, Sandrine Cestèle, Daniele Grioni, Silvana Franceschetti, (unknown), Massimo Mantegazza	52
18	Identification of an Na _v 1.1 sodium channel (SCN1A) loss-of-function mutation associated with familial simple febrile seizures 2005 ·Proceedings of the National Academy of Sciences ·Massimo Mantegazza, Antonio Gambardella, Raffaella Rusconi, Emanuele Schiavon, Ferdinanda Annesi, Rita Restano‐Cassulini, Angelo Labate, Sara Carrideo, R. Chifari, Maria Paola Canevini, R. Canger, Silvana Franceschetti, Grazia Annesi, Enzo Wanke, Aldo Quattrone	154
19	First chemical synthesis of a scorpion α‐toxin affecting sodium channels: The Aah I toxin of Androctonus australis hector 2004 ·Journal of Peptide Science ·Sarrah Ben M’Barek, Ziad Fajloun, Sandrine Cestèle, Christiane Devaux, Pascal Mansuelle, Amor Mosbah, Besma Jouirou, Massimo Mantegazza, J. Van Rietschoten, Mohamed El Ayeb, Hervé Rochat, Jean‐Marc Sabatier, Franĉois Sampiéri	12
20	Valproate selectively reduces the persistent fraction of Na+ current in neocortical neurons 1998 ·Epilepsy Research ·Stefano Taverna, Massimo Mantegazza, Silvana Franceschetti, G. Avanzini	75

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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