Masayoshi Takeuchi

14.8k total citations · 1 hit paper
256 papers, 12.4k citations indexed

About

Masayoshi Takeuchi is a scholar working on Clinical Biochemistry, Endocrinology, Diabetes and Metabolism and Pathology and Forensic Medicine. According to data from OpenAlex, Masayoshi Takeuchi has authored 256 papers receiving a total of 12.4k indexed citations (citations by other indexed papers that have themselves been cited), including 201 papers in Clinical Biochemistry, 106 papers in Endocrinology, Diabetes and Metabolism and 54 papers in Pathology and Forensic Medicine. Recurrent topics in Masayoshi Takeuchi's work include Advanced Glycation End Products research (198 papers), Natural Antidiabetic Agents Studies (48 papers) and Alcohol Consumption and Health Effects (48 papers). Masayoshi Takeuchi is often cited by papers focused on Advanced Glycation End Products research (198 papers), Natural Antidiabetic Agents Studies (48 papers) and Alcohol Consumption and Health Effects (48 papers). Masayoshi Takeuchi collaborates with scholars based in Japan, United States and Romania. Masayoshi Takeuchi's co-authors include Sho‐ichi Yamagishi, Takanori Matsui, Zenji Makita, Sho‐ichi Yamagishi, Yosuke Inagaki, Tsutomu Imaizumi, Kazuo Nakamura, Shinjiro Amano, Tamami Okamoto and Hiroshi Yamamoto and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and PLoS ONE.

In The Last Decade

Masayoshi Takeuchi

255 papers receiving 12.1k citations

Hit Papers

Novel splice variants of the receptor for advanced glycat... 2003 2026 2010 2018 2003 100 200 300 400 500

Peers

Masayoshi Takeuchi
Michael Brownlee United States
Ravichandran Ramasamy United States
Diane Edelstein United States
Shi Du Yan United States
Masayoshi Takeuchi
Citations per year, relative to Masayoshi Takeuchi Masayoshi Takeuchi (= 1×) peers Takanori Matsui

Countries citing papers authored by Masayoshi Takeuchi

Since Specialization
Citations

This map shows the geographic impact of Masayoshi Takeuchi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masayoshi Takeuchi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masayoshi Takeuchi more than expected).

Fields of papers citing papers by Masayoshi Takeuchi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Masayoshi Takeuchi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masayoshi Takeuchi. The network helps show where Masayoshi Takeuchi may publish in the future.

Co-authorship network of co-authors of Masayoshi Takeuchi

This figure shows the co-authorship network connecting the top 25 collaborators of Masayoshi Takeuchi. A scholar is included among the top collaborators of Masayoshi Takeuchi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masayoshi Takeuchi. Masayoshi Takeuchi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sakai, Akiko, Kenji Takeda, Hirokazu Suzuki, & Masayoshi Takeuchi. (2024). Structures of Toxic Advanced Glycation End-Products Derived from Glyceraldehyde, A Sugar Metabolite. Biomolecules. 14(2). 202–202. 12 indexed citations
2.
Takeuchi, Masayoshi. (2024). Toxic AGEs (TAGE) Cause Lifestyle-Related Diseases. Antioxidants. 13(11). 1372–1372. 2 indexed citations
3.
Furukawa, Ayako, et al.. (2024). Toxic Advanced Glycation End-Products Inhibit Axonal Elongation Mediated by β-Tubulin Aggregation in Mice Optic Nerves. International Journal of Molecular Sciences. 25(13). 7409–7409. 1 indexed citations
5.
Sakai, Akiko, Kenji Takeda, & Masayoshi Takeuchi. (2023). Involvement of Intracellular TAGE and the TAGE–RAGE–ROS Axis in the Onset and Progression of NAFLD/NASH. Antioxidants. 12(3). 748–748. 12 indexed citations
6.
Takeuchi, Masayoshi, Akiko Sakai, Takanobu Takata, Jun-ichi Takino, & Yoshiki Koriyama. (2022). Effects of Toxic AGEs (TAGE) on Human Health. Cells. 11(14). 2178–2178. 27 indexed citations
7.
Takeuchi, Masayoshi, Akiko Sakai, Takanobu Takata, et al.. (2021). Intracellular Toxic AGEs (TAGE) Triggers Numerous Types of Cell Damage. Biomolecules. 11(3). 387–387. 36 indexed citations
9.
Takeuchi, Masayoshi, et al.. (2015). Assessment of the Concentrations of Various Advanced Glycation End-Products in Beverages and Foods That Are Commonly Consumed in Japan. PLoS ONE. 10(3). e0118652–e0118652. 69 indexed citations
11.
Takino, Jun-ichi, Hiroyuki Tsuchiya, Tomohiko Sakabe, et al.. (2013). Presence of Glyceraldehyde-Derived Advanced Glycation End-Products in the Liver of Insulin-Resistant Mice. International Journal for Vitamin and Nutrition Research. 83(2). 137–141. 7 indexed citations
12.
Ishibashi, Yuji, Takanori Matsui, Masayoshi Takeuchi, & Sho‐ichi Yamagishi. (2009). Glucagon-like peptide-1 (GLP-1) inhibits advanced glycation end product (AGE)-induced up-regulation of VCAM-1 mRNA levels in endothelial cells by suppressing AGE receptor (RAGE) expression. Biochemical and Biophysical Research Communications. 391(3). 1405–1408. 144 indexed citations
13.
Yamagishi, Sho‐ichi, et al.. (2007). Pigment Epithelium-derived Factor (PEDF) Blocks Advanced Glycation End Product (AGE)-induced Angiogenesis In Vitro. Hormone and Metabolic Research. 39(3). 233–235. 15 indexed citations
14.
Yoshida, Takafumi, Sho‐ichi Yamagishi, Kazuo Nakamura, et al.. (2006). Pigment epithelium‐derived factor (PEDF) inhibits advanced glycation end product (AGE)‐induced C‐reactive protein expression in hepatoma cells by suppressing Rac‐1 activation. FEBS Letters. 580(11). 2788–2796. 34 indexed citations
15.
Yamagishi, Sho‐ichi, Yuji Inagaki, Masayoshi Takeuchi, & Nobuhito Sasaki. (2004). Is pigment epithelium-derived factor level in cerebrospinal fluid a promising biomarker for early diagnosis of Alzheimer's disease?. Medical Hypotheses. 63(1). 115–117. 24 indexed citations
16.
Yamagishi, Sho‐ichi, Yosuke Inagaki, Tamami Okamoto, et al.. (2003). Advanced glycation end products inhibit de novo protein synthesis and induce TGF-β overexpression in proximal tubular cells. Kidney International. 63(2). 464–473. 136 indexed citations
17.
Inagaki, Yuji, et al.. (2002). Up-regulation of vascular endothelial growth factor and down-regulation of pigment epithelium-derived factor messenger ribonucleic acid levels in leptin-exposed cultured retinal pericytes.. PubMed. 24(4). 137–42. 20 indexed citations
18.
Yamamoto, Yasuhiko, Ichiro Kato, Toshio Doi, et al.. (2001). Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice. Journal of Clinical Investigation. 108(2). 261–268. 40 indexed citations
19.
Yamamoto, Yasuhiko, Ichiro Kato, Toshio Doi, et al.. (2001). Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice. Journal of Clinical Investigation. 108(2). 261–268. 409 indexed citations
20.
Kameda, Yukihiko, et al.. (1997). Preparation and biological activity of manno- and galacto-validamines, new 5a-carba-glycosylamines as α-glycosidase inhibitors. Carbohydrate Research. 300(3). 259–264. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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