Masaaki Nagatake
- Co-authors
- Takashi TakahashiHiroyuki OsadaKosaku UchidaTetsuya MitsudomiKaoru ShimokataY TakagiTakashi KoshikawaMasashi Kondo
- Topics
- Genetic factors in colorectal cancer (2 papers)Cancer-related gene regulation (2 papers)Cancer-related Molecular Pathways (2 papers)
- Journals
- OncogeneLung CancerPubMed
- Partner nations
- JapanUnited States
In The Last Decade
Masaaki Nagatake
7 papers receiving 542 citations
Peers
Comparison fields: 5 of 41
- Molecular Biology 438
- Oncology 282
- Pathology and Forensic Medicine 119
- Genetics 94
- Cancer Research 85
Countries citing papers authored by Masaaki Nagatake
This map shows the geographic impact of Masaaki Nagatake's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masaaki Nagatake with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masaaki Nagatake more than expected).
Fields of papers citing papers by Masaaki Nagatake
This network shows the impact of papers produced by Masaaki Nagatake. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masaaki Nagatake. The network helps show where Masaaki Nagatake may publish in the future.
Co-authorship network of co-authors of Masaaki Nagatake
This figure shows the co-authorship network connecting the top 25 collaborators of Masaaki Nagatake. A scholar is included among the top collaborators of Masaaki Nagatake based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masaaki Nagatake. Masaaki Nagatake is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 41 | |
| 2 | Prognostic significance of cyclin D1 and retinoblastoma expression in combination with p53 abnormalities in primary, resected non-small cell lung cancers. | 56 |
| 3 | 92 | |
| 4 | Somatic in vivo alterations of the DPC4 gene at 18q21 in human lung cancers. | 204 |
| 5 | Aberrant hypermethylation at the bcl-2 locus at 18q21 in human lung cancers. | 25 |
| 6 | Allelic-expression imbalance of the insulin-like growth factor 2 gene in hepatocellular carcinoma and underlying disease. | 45 |
| 7 | In vivo occurrence of p16 (MTS1) and p15 (MTS2) alterations preferentially in non-small cell lung cancers. | 97 |
About Masaaki Nagatake
Masaaki Nagatake is a scholar working on Pathology and Forensic Medicine, Cancer Research and Oncology, having authored 7 papers that have together received 560 indexed citations. Recurring topics across this work include Genetic factors in colorectal cancer (2 papers), Cancer-related gene regulation (2 papers) and Cancer-related Molecular Pathways (2 papers). The work is most often cited by research in Oncology (282 citations), Pathology and Forensic Medicine (119 citations) and Molecular Biology (438 citations). Masaaki Nagatake has collaborated with scholars based in Japan and United States. Frequent co-authors include Takashi Takahashi, Hiroyuki Osada, Kosaku Uchida, Tetsuya Mitsudomi, Kaoru Shimokata, Y Takagi, Takashi Koshikawa, Masashi Kondo, Ryuzo Ueda and Osuke Washimi. Their work appears in journals such as Oncogene, Lung Cancer and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.