Hirotaka Osada

10.8k total citations · 2 hit papers
73 papers, 8.5k citations indexed

About

Hirotaka Osada is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Hirotaka Osada has authored 73 papers receiving a total of 8.5k indexed citations (citations by other indexed papers that have themselves been cited), including 52 papers in Molecular Biology, 22 papers in Oncology and 20 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Hirotaka Osada's work include RNA modifications and cancer (20 papers), Epigenetics and DNA Methylation (14 papers) and Cancer-related gene regulation (12 papers). Hirotaka Osada is often cited by papers focused on RNA modifications and cancer (20 papers), Epigenetics and DNA Methylation (14 papers) and Cancer-related gene regulation (12 papers). Hirotaka Osada collaborates with scholars based in Japan, United States and China. Hirotaka Osada's co-authors include Takashi Takahashi, Yasushi Yatabe, Kiyoshi Yanagisawa, Shuta Tomida, Tetsuya Mitsudomi, Yoshitaka Sekido, Yoshio Tatematsu, Junichi Takamizawa, Hiroyuki Konishi and Yuji Nimura and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Experimental Medicine.

In The Last Decade

Hirotaka Osada

73 papers receiving 8.3k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Reduced Expression of the let-7 MicroRNAs in Human Lung Cancers in Association with Shortened Postoperative Survival

2004 1.9k citations Junichi Takamizawa, Hiroyuki Konishi et al. Cancer Research profile →
A Polycistronic MicroRNA Cluster, miR-17-92 , Is Overexpressed in Human Lung Cancers and Enhances Cell Proliferation

2005 1.3k citations Yoji Hayashita, Hirotaka Osada et al. Cancer Research profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Hirotaka Osada Japan	44	6.5k	4.2k	1.7k	1.4k	782	73	8.5k
Longbang Chen China	49	5.9k 0.9×	4.7k 1.1×	1.7k 1.0×	771 0.5×	336 0.4×	155	8.1k
Andreas G. Bader United States	30	7.2k 1.1×	5.3k 1.2×	1.6k 0.9×	792 0.6×	262 0.3×	58	8.9k
M. James You United States	36	4.5k 0.7×	2.5k 0.6×	2.1k 1.2×	1.1k 0.7×	488 0.6×	129	7.3k
Yongkun Wei United States	35	4.9k 0.8×	2.2k 0.5×	3.3k 1.9×	1.6k 1.1×	517 0.7×	56	7.6k
Xiulan Zhao China	44	3.8k 0.6×	2.3k 0.6×	2.0k 1.2×	718 0.5×	372 0.5×	135	5.5k
James R. Gnarra United States	38	3.8k 0.6×	2.5k 0.6×	1.2k 0.7×	1.4k 1.0×	376 0.5×	68	5.8k
Paolo Nucíforo Spain	42	4.1k 0.6×	1.8k 0.4×	3.4k 2.0×	1.2k 0.9×	699 0.9×	175	7.3k
Michail Shipitsin United States	15	5.1k 0.8×	2.8k 0.7×	5.5k 3.2×	1.0k 0.7×	991 1.3×	21	8.5k
Pierre Savagner France	28	5.0k 0.8×	1.8k 0.4×	3.8k 2.2×	841 0.6×	1.1k 1.4×	47	7.7k
Andrew G. Bert Australia	29	5.7k 0.9×	4.3k 1.0×	1.7k 1.0×	450 0.3×	446 0.6×	43	7.5k

Countries citing papers authored by Hirotaka Osada

Since Specialization

Citations

This map shows the geographic impact of Hirotaka Osada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hirotaka Osada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hirotaka Osada more than expected).

Fields of papers citing papers by Hirotaka Osada

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hirotaka Osada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hirotaka Osada. The network helps show where Hirotaka Osada may publish in the future.

Co-authorship network of co-authors of Hirotaka Osada

This figure shows the co-authorship network connecting the top 25 collaborators of Hirotaka Osada. A scholar is included among the top collaborators of Hirotaka Osada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hirotaka Osada. Hirotaka Osada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Shinjo, Keiko, Yasuyuki Okamoto, Ichiro Takeuchi, et al.. (2012). Abstract 5003: Integrated analysis of genetic and epigenetic alterations reveals CpG island methylator phenotype associated with distinct clinical characters of lung adenocarcinoma. Cancer Research. 72(8_Supplement). 5003–5003. 1 indexed citations

Ishiguro, Futoshi, Hideki Murakami, Tetsuya Mizuno, et al.. (2012). Membranous expression of activated leukocyte cell adhesion molecule contributes to poor prognosis and malignant phenotypes of non–small-cell lung cancer. Journal of Surgical Research. 179(1). 24–32. 19 indexed citations

Murakami, Hideki, Tetsuya Mizuno, Tetsuo Taniguchi, et al.. (2011). LATS2 Is a Tumor Suppressor Gene of Malignant Mesothelioma. Cancer Research. 71(3). 873–883. 181 indexed citations

Suda, Kenichi, Isao Murakami, Tatsuya Katayama, et al.. (2010). Reciprocal and Complementary Role of MET Amplification and EGFR T790M Mutation in Acquired Resistance to Kinase Inhibitors in Lung Cancer. Clinical Cancer Research. 16(22). 5489–5498. 170 indexed citations

Yanagisawa, Kiyoshi, Hiroyuki Konishi, Chinatsu Arima, et al.. (2010). Novel Metastasis-Related Gene CIM Functions in the Regulation of Multiple Cellular Stress–Response Pathways. Cancer Research. 70(23). 9949–9958. 22 indexed citations

Huang, Qin, Shuta Tomida, Yuji Masuda, et al.. (2010). Regulation of DNA Polymerase POLD4 Influences Genomic Instability in Lung Cancer. Cancer Research. 70(21). 8407–8416. 40 indexed citations

An, Byonggu, Yutaka Kondo, Yasuyuki Okamoto, et al.. (2010). Characteristic methylation profile in CpG island methylator phenotype‐negative distal colorectal cancers. International Journal of Cancer. 127(9). 2095–2105. 33 indexed citations

Osada, Hirotaka, Shuta Tomida, Yasushi Yatabe, et al.. (2008). Roles of Achaete-Scute Homologue 1 in DKK1 and E-cadherin Repression and Neuroendocrine Differentiation in Lung Cancer. Cancer Research. 68(6). 1647–1655. 83 indexed citations

Tanaka, Hisaaki, Kiyoshi Yanagisawa, Keiko Shinjo, et al.. (2007). Lineage-Specific Dependency of Lung Adenocarcinomas on the Lung Development Regulator TTF-1. Cancer Research. 67(13). 6007–6011. 165 indexed citations

10.

Horio, Yoshitsugu, Junichi Shimizu, Hirotaka Osada, et al.. (2007). The mRNA expression of RRM1, ERCC1 and ERCC2 is not associated with chemosensitivity of cisplatin, carboplatin and gemcitabine in human lung cancer cell lines. Molecular Cancer Therapeutics. 6. 1 indexed citations

11.

Osada, Hirotaka, Yoshio Tatematsu, Yasushi Yatabe, Yoshitsugu Horio, & Takashi Takahashi. (2005). ASH1 Gene Is a Specific Therapeutic Target for Lung Cancers with Neuroendocrine Features. Cancer Research. 65(23). 10680–10685. 103 indexed citations

12.

Hayashita, Yoji, Hirotaka Osada, Yoshio Tatematsu, et al.. (2005). A Polycistronic MicroRNA Cluster, miR-17-92 , Is Overexpressed in Human Lung Cancers and Enhances Cell Proliferation. Cancer Research. 65(21). 9628–9632. 1274 indexed citations breakdown →

13.

Karube, Yoko, Hisaaki Tanaka, Hirotaka Osada, et al.. (2005). Reduced expression of Dicer associated with poor prognosis in lung cancer patients. Cancer Science. 96(2). 111–115. 476 indexed citations

14.

Takamizawa, Junichi, Hiroyuki Konishi, Kiyoshi Yanagisawa, et al.. (2004). Reduced Expression of the let-7 MicroRNAs in Human Lung Cancers in Association with Shortened Postoperative Survival. Cancer Research. 64(11). 3753–3756. 1938 indexed citations breakdown →

15.

Mizuno, Kotaro, Hirotaka Osada, Hiroyuki Konishi, et al.. (2002). Aberrant hypermethylation of the CHFR prophase checkpoint gene in human lung cancers. Oncogene. 21(15). 2328–2333. 108 indexed citations

16.

Haruki, Nobuhiro, Hiroko Saito, Shuji Nomoto, et al.. (2001). Molecular analysis of the mitotic checkpoint genes BUB1, BUBR1 and BUB3 in human lung cancers. Cancer Letters. 162(2). 201–205. 54 indexed citations

17.

Masuda, Akira, Hirotaka Osada, Yasushi Yatabe, et al.. (2001). Protective Function of p27KIP1 against Apoptosis in Small Cell Lung Cancer Cells in Unfavorable Microenvironments. American Journal Of Pathology. 158(1). 87–96. 32 indexed citations

18.

Kondo, Masashi, Hirotaka Osada, Kosaku Uchida, et al.. (1998). Molecular cloning of human TAK1 and its mutational analysis in human lung cancer. International Journal of Cancer. 75(4). 559–563. 23 indexed citations

19.

Takahashi, Takao, Takao Takahashi, Hiroyuki Konishi, et al.. (1998). Molecular Analysis of a Myc Antagonist, ROX/Mnt, at 17p13.3 in Human Lung Cancers. Japanese Journal of Cancer Research. 89(4). 347–351. 15 indexed citations

20.

Uchida, Kosaku, Masashi Kondo, Shin Takeda, et al.. (1997). Altered transcriptional regulation of the insulin-like growth factor 2 gene in human hepatocellular carcinoma. Molecular Carcinogenesis. 18(4). 193–198. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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