Martin Schröder
- Molecular Biology
- Oncology top 10%
- Genetics top 5%
- Pathology and Forensic Medicine top 10%
- Organic Chemistry
- Co-authors
- Stefan KnappPeter FriedlA. ChaikuadStephan StilgenbauerHartmut DöhnerKonstanze FischerPeter LichterMartin Bentz
- Topics
- Chronic Lymphocytic Leukemia Research (7 papers)Melanoma and MAPK Pathways (5 papers)Protein Kinase Regulation and GTPase Signaling (5 papers)
- Journals
- Proceedings of the National Academy of SciencesAngewandte Chemie International EditionNature Communications
- Partner nations
- GermanyUnited KingdomUnited States
In The Last Decade
Martin Schröder
44 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 87
- Molecular Biology 678
- Oncology 284
- Genetics 240
- Pathology and Forensic Medicine 191
- Organic Chemistry 142
Countries citing papers authored by Martin Schröder
This map shows the geographic impact of Martin Schröder's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Schröder with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Schröder more than expected).
Fields of papers citing papers by Martin Schröder
This network shows the impact of papers produced by Martin Schröder. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Schröder. The network helps show where Martin Schröder may publish in the future.
Co-authorship network of co-authors of Martin Schröder
This figure shows the co-authorship network connecting the top 25 collaborators of Martin Schröder. A scholar is included among the top collaborators of Martin Schröder based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Schröder. Martin Schröder is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 7 | |
| 4 | 16 | |
| 5 | 5 | |
| 6 | 2 | |
| 7 | 50 | |
| 8 | 35 | |
| 9 | 20 | |
| 10 | 23 | |
| 11 | 14 | |
| 12 | 9 | |
| 13 | 19 | |
| 14 | 32 | |
| 15 | 51 | |
| 16 | 89 | |
| 17 | 29 | |
| 18 | 46 | |
| 19 | 112 | |
| 20 | 16 |
About Martin Schröder
Martin Schröder is a scholar working on Immunology and Allergy, Genetics and Biophysics, having authored 45 papers that have together received 1.3k indexed citations. Recurring topics across this work include Chronic Lymphocytic Leukemia Research (7 papers), Melanoma and MAPK Pathways (5 papers) and Protein Kinase Regulation and GTPase Signaling (5 papers). The work is most often cited by research in Genetics (240 citations), Pathology and Forensic Medicine (191 citations) and Oncology (284 citations). Martin Schröder has collaborated with scholars based in Germany, United Kingdom and United States. Frequent co-authors include Stefan Knapp, Peter Friedl, A. Chaikuad, Stephan Stilgenbauer, Hartmut Döhner, Konstanze Fischer, Peter Lichter, Martin Bentz, Rolf Schäfer and Ulrike Lehmkuhl. Their work appears in journals such as Proceedings of the National Academy of Sciences, Angewandte Chemie International Edition and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.