Bertrand Ducos
- Molecular Biology top 5%
- Aging top 0.1%
- Physiology top 2%
- Endocrinology, Diabetes and Metabolism top 5%
- Cellular and Molecular Neuroscience top 5%
- Co-authors
- Yves Le BoucMartin HolzenbergerPatricia LeneuveAlain GéloënPascale CerveraJoëlle DupontPatrick C. EvenMarianne Mangeney
- Topics
- Neuropeptides and Animal Physiology (7 papers)Photoreceptor and optogenetics research (5 papers)CRISPR and Genetic Engineering (4 papers)
- Partner nations
- FranceUnited StatesBurundi
In The Last Decade
Bertrand Ducos
45 papers receiving 2.9k citations
Hit Papers
Peers
Comparison fields: 5 of 116
- Molecular Biology 1.4k
- Aging 900
- Physiology 797
- Endocrinology, Diabetes and Metabolism 360
- Cellular and Molecular Neuroscience 358
Countries citing papers authored by Bertrand Ducos
This map shows the geographic impact of Bertrand Ducos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bertrand Ducos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bertrand Ducos more than expected).
Fields of papers citing papers by Bertrand Ducos
This network shows the impact of papers produced by Bertrand Ducos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bertrand Ducos. The network helps show where Bertrand Ducos may publish in the future.
Co-authorship network of co-authors of Bertrand Ducos
This figure shows the co-authorship network connecting the top 25 collaborators of Bertrand Ducos. A scholar is included among the top collaborators of Bertrand Ducos based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bertrand Ducos. Bertrand Ducos is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 1 | |
| 4 | 2 | |
| 5 | 30 | |
| 6 | 6 | |
| 7 | 5 | |
| 8 | 6 | |
| 9 | 4 | |
| 10 | 8 | |
| 11 | 46 | |
| 12 | 21 | |
| 13 | 14 | |
| 14 | 51 | |
| 15 | IGF-1 receptor regulates lifespan and resistance to oxidative stress in micebreakdown → | 1627 |
| 16 | 16 | |
| 17 | 18 | |
| 18 | 29 | |
| 19 | 71 | |
| 20 | 36 |
About Bertrand Ducos
Bertrand Ducos is a scholar working on Cellular and Molecular Neuroscience, Structural Biology and Cell Biology, having authored 46 papers that have together received 3.0k indexed citations. Recurring topics across this work include Neuropeptides and Animal Physiology (7 papers), Photoreceptor and optogenetics research (5 papers) and CRISPR and Genetic Engineering (4 papers). The work is most often cited by research in Aging (900 citations), Endocrine and Autonomic Systems (278 citations) and Physiology (797 citations). Bertrand Ducos has collaborated with scholars based in France, United States and Burundi. Frequent co-authors include Yves Le Bouc, Martin Holzenberger, Patricia Leneuve, Alain Géloën, Pascale Cervera, Joëlle Dupont, Patrick C. Even, Marianne Mangeney, Martial Renard and Thiérry Heidmann. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.