Marta Noa Valcarcel‐Ares
About
Marta Noa Valcarcel‐Ares is a scholar working on Molecular Biology, Neurology and Neurology.
According to data from OpenAlex, Marta Noa Valcarcel‐Ares has authored 31 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 7 papers in Neurology and 5 papers in Neurology. Recurrent topics in Marta Noa Valcarcel‐Ares's work include Neurological Disease Mechanisms and Treatments (5 papers), Growth Hormone and Insulin-like Growth Factors (4 papers) and Genomics, phytochemicals, and oxidative stress (3 papers). Marta Noa Valcarcel‐Ares is often cited by papers focused on Neurological Disease Mechanisms and Treatments (5 papers), Growth Hormone and Insulin-like Growth Factors (4 papers) and Genomics, phytochemicals, and oxidative stress (3 papers). Marta Noa Valcarcel‐Ares collaborates with scholars based in United States, Hungary and Spain. Marta Noa Valcarcel‐Ares's co-authors include Anna Csiszár, Carlos Vaamonde‐García, R.R. Riveiro-Naveira, M.J. López-Armada, Zoltán Ungvári, Stefano Tarantini, William E. Sonntag, Andriy Yabluchanskiy, Zsuzsanna Tucsek and Tamás Kiss and has published in prestigious journals such as SHILAP Revista de lepidopterología, The FASEB Journal and Annals of the Rheumatic Diseases.
In The Last Decade
Marta Noa Valcarcel‐Ares
31 papers receiving 2.4k citations
Hit Papers
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| Marta Noa Valcarcel‐Ares United States | 23 | 849 | 616 | 543 | 279 | 260 | 31 | 2.4k | ||
| Tripti Gautam United States | 21 | 888 1.0× | 615 1.0× | 639 1.2× | 259 0.9× | 395 1.5× | 35 | 2.5k | ||
| Ádám Nyúl‐Tóth Hungary | 32 | 680 0.8× | 581 0.9× | 857 1.6× | 256 0.9× | 378 1.5× | 68 | 2.6k | ||
| Tamás Csípő Hungary | 30 | 482 0.6× | 577 0.9× | 513 0.9× | 237 0.8× | 309 1.2× | 60 | 2.3k | ||
| Fuzhou Hua China | 29 | 979 1.2× | 368 0.6× | 282 0.5× | 187 0.7× | 103 0.4× | 77 | 2.2k | ||
| Jian‐Quan Shi China | 21 | 612 0.7× | 536 0.9× | 636 1.2× | 309 1.1× | 245 0.9× | 54 | 1.9k | ||
| Kristian P. Doyle United States | 25 | 878 1.0× | 315 0.5× | 1.2k 2.3× | 351 1.3× | 272 1.0× | 45 | 2.6k | ||
| Ján Lehotský Slovakia | 28 | 1.1k 1.3× | 477 0.8× | 272 0.5× | 238 0.9× | 154 0.6× | 121 | 2.7k | ||
| Sookja Kim Chung Hong Kong | 30 | 772 0.9× | 891 1.4× | 469 0.9× | 427 1.5× | 165 0.6× | 67 | 2.7k | ||
| Jenq‐Lin Yang Taiwan | 28 | 1.0k 1.2× | 520 0.8× | 373 0.7× | 242 0.9× | 160 0.6× | 53 | 2.3k | ||
| Budbazar Enkhjargal United States | 28 | 708 0.8× | 222 0.4× | 453 0.8× | 237 0.8× | 746 2.9× | 43 | 1.9k |
Countries citing papers authored by Marta Noa Valcarcel‐Ares
Since
Specialization
Citations
This map shows the geographic impact of Marta Noa Valcarcel‐Ares's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marta Noa Valcarcel‐Ares with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marta Noa Valcarcel‐Ares more than expected).
Fields of papers citing papers by Marta Noa Valcarcel‐Ares
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Marta Noa Valcarcel‐Ares. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marta Noa Valcarcel‐Ares. The network helps show where Marta Noa Valcarcel‐Ares may publish in the future.
Co-authorship network of co-authors of Marta Noa Valcarcel‐Ares
This figure shows the co-authorship network connecting the top 25 collaborators of Marta Noa Valcarcel‐Ares. A scholar is included among the top collaborators of Marta Noa Valcarcel‐Ares based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marta Noa Valcarcel‐Ares. Marta Noa Valcarcel‐Ares is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Valcarcel‐Ares, Marta Noa, et al.. (2023). Integrating Language Instruction into Pharmacy Education: Spanish and Arabic Languages as Examples. SHILAP Revista de lepidopterología. 2(3). 175–187.
2.
Valcarcel‐Ares, Marta Noa, et al.. (2021). Preclinical and clinical evidence of IGF-1 as a prognostic marker and acute intervention with ischemic stroke. Journal of Cerebral Blood Flow & Metabolism. 41(10). 2475–2491.
3.
Kiss, Tamás, Priya Balasubramanian, Marta Noa Valcarcel‐Ares, et al.. (2019). Nicotinamide mononucleotide (NMN) treatment attenuates oxidative stress and rescues angiogenic capacity in aged cerebromicrovascular endothelial cells: a potential mechanism for the prevention of vascular cognitive impairment. GeroScience. 41(5). 619–630.
4.
Tarantini, Stefano, Marta Noa Valcarcel‐Ares, Andriy Yabluchanskiy, et al.. (2017). Nrf2 Deficiency Exacerbates Obesity-Induced Oxidative Stress, Neurovascular Dysfunction, Blood–Brain Barrier Disruption, Neuroinflammation, Amyloidogenic Gene Expression, and Cognitive Decline in Mice, Mimicking the Aging Phenotype. The Journals of Gerontology Series A. 73(7). 853–863.
5.
Csiszár, Anna, Stefano Tarantini, Gábor Fülöp, et al.. (2017). Hypertension impairs neurovascular coupling and promotes microvascular injury: role in exacerbation of Alzheimer’s disease. GeroScience. 39(4). 359–372.
6.
Ungvári, Zoltán, Stefano Tarantini, Péter Hertelendy, et al.. (2017). Cerebromicrovascular dysfunction predicts cognitive decline and gait abnormalities in a mouse model of whole brain irradiation-induced accelerated brain senescence. GeroScience. 39(1). 33–42.
7.
Tarantini, Stefano, Gábor Áron Fülöp, Péter Hertelendy, et al.. (2017). Pharmacologically induced impairment of neurovascular coupling responses alters gait coordination in mice. GeroScience. 39(5-6). 601–614.
8.
Ungvári, Zoltán, Marta Noa Valcarcel‐Ares, Stefano Tarantini, et al.. (2017). Connective tissue growth factor (CTGF) in age-related vascular pathologies. GeroScience. 39(5-6). 491–498.
9.
Tarantini, Stefano, Zsuzsanna Tucsek, Marta Noa Valcarcel‐Ares, et al.. (2016). Circulating IGF-1 deficiency exacerbates hypertension-induced microvascular rarefaction in the mouse hippocampus and retrosplenial cortex: implications for cerebromicrovascular and brain aging. AGE. 38(4). 273–289.
10.
Riveiro-Naveira, R.R., Marta Noa Valcarcel‐Ares, Maylin Almonte‐Becerril, et al.. (2016). Resveratrol lowers synovial hyperplasia, inflammatory markers and oxidative damage in an acute antigen-induced arthritis model. Lara D. Veeken. 55(10). 1889–1900.
11.
Tarantini, Stefano, Cory B. Giles, Jonathan D. Wren, et al.. (2016). IGF-1 deficiency in a critical period early in life influences the vascular aging phenotype in mice by altering miRNA-mediated post-transcriptional gene regulation: implications for the developmental origins of health and disease hypothesis. AGE. 38(4). 239–258.
12.
Tarantini, Stefano, Péter Hertelendy, Zsuzsanna Tucsek, et al.. (2015). Pharmacologically-Induced Neurovascular Uncoupling is Associated with Cognitive Impairment in Mice. Journal of Cerebral Blood Flow & Metabolism. 35(11). 1871–1881.
13.
Tóth, Péter, Stefano Tarantini, Antonio Dávila, et al.. (2015). Purinergic glio-endothelial coupling during neuronal activity: role of P2Y1 receptors and eNOS in functional hyperemia in the mouse somatosensory cortex. American Journal of Physiology-Heart and Circulatory Physiology. 309(11). H1837–H1845.
14.
Valcarcel‐Ares, Marta Noa, R.R. Riveiro-Naveira, Carlos Vaamonde‐García, et al.. (2014). Mitochondrial dysfunction promotes and aggravates the inflammatory response in normal human synoviocytes. Lara D. Veeken. 53(7). 1332–1343.
15.
Riveiro-Naveira, R.R., José Loureiro-Amigo, Marta Noa Valcarcel‐Ares, et al.. (2014). Anti-inflammatory effect of resveratrol as a dietary supplement in an antigen-induced arthritis rat model.. Osteoarthritis and Cartilage. 22. S290–S290.
16.
Valcarcel‐Ares, Marta Noa, T. Gautam, Junie P. Warrington, et al.. (2012). Disruption of Nrf2 Signaling Impairs Angiogenic Capacity of Endothelial Cells: Implications for Microvascular Aging. The Journals of Gerontology Series A. 67(8). 821–829.
17.
Ungvári, Zoltán, Zsuzsanna Tucsek, Danuta Sosnowska, et al.. (2012). Aging-Induced Dysregulation of Dicer1-Dependent MicroRNA Expression Impairs Angiogenic Capacity of Rat Cerebromicrovascular Endothelial Cells. The Journals of Gerontology Series A. 68(8). 877–891.
18.
Vaamonde‐García, Carlos, et al.. (2012). Mitochondrial dysfunction increases inflammatory responsiveness to cytokines in normal human chondrocytes. Arthritis & Rheumatism. 64(9). 2927–2936.
19.
Bailey‐Downs, Lora C., Matthew Mitschelen, Danuta Sosnowska, et al.. (2011). Liver-Specific Knockdown of IGF-1 Decreases Vascular Oxidative Stress Resistance by Impairing the Nrf2-Dependent Antioxidant Response: A Novel Model of Vascular Aging. The Journals of Gerontology Series A. 67A(4). 313–329.
20.
Valcarcel‐Ares, Marta Noa, et al.. (2010). A novel role for mitochondrial dysfunction in the inflammatory response of rheumatoid arthritis. Annals of the Rheumatic Diseases. 69. A56–A56.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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