Marlies Cortés

509 total citations
8 papers, 337 citations indexed

About

Marlies Cortés is a scholar working on Immunology, Molecular Biology and Surgery. According to data from OpenAlex, Marlies Cortés has authored 8 papers receiving a total of 337 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 4 papers in Molecular Biology and 2 papers in Surgery. Recurrent topics in Marlies Cortés's work include Immune cells in cancer (4 papers), Phagocytosis and Immune Regulation (2 papers) and Immunotherapy and Immune Responses (2 papers). Marlies Cortés is often cited by papers focused on Immune cells in cancer (4 papers), Phagocytosis and Immune Regulation (2 papers) and Immunotherapy and Immune Responses (2 papers). Marlies Cortés collaborates with scholars based in Spain, United States and Chile. Marlies Cortés's co-authors include Antonio Postigo, Douglas S. Darling, Douglas C. Dean, Lidia Sánchez-Moral, María Jesús Fernández‐Aceñero, Oriol de Barrios, Anna Esteve‐Codina, Laura Siles, Balázs Győrffy and Toby Lawrence and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and The EMBO Journal.

In The Last Decade

Marlies Cortés

8 papers receiving 333 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Marlies Cortés Spain 6 176 127 102 78 31 8 337
Nahoko Tomonobu Japan 9 184 1.0× 104 0.8× 104 1.0× 80 1.0× 40 1.3× 24 350
Sara Lamorte Canada 7 226 1.3× 186 1.5× 110 1.1× 83 1.1× 32 1.0× 12 459
Tzu-Sheng Hsu Taiwan 9 128 0.7× 143 1.1× 86 0.8× 84 1.1× 24 0.8× 21 314
Jae Ho Lee South Korea 10 187 1.1× 65 0.5× 98 1.0× 71 0.9× 21 0.7× 20 343
Michelle Somerville United Kingdom 8 142 0.8× 123 1.0× 106 1.0× 62 0.8× 33 1.1× 14 395
Markus Linder Austria 7 190 1.1× 73 0.6× 76 0.7× 61 0.8× 44 1.4× 10 309
Ran Yan United States 6 145 0.8× 131 1.0× 173 1.7× 72 0.9× 38 1.2× 8 378
Xiangdong Yang China 9 129 0.7× 148 1.2× 97 1.0× 46 0.6× 35 1.1× 13 342
Xiangbin Wu China 6 146 0.8× 64 0.5× 120 1.2× 105 1.3× 25 0.8× 9 287

Countries citing papers authored by Marlies Cortés

Since Specialization
Citations

This map shows the geographic impact of Marlies Cortés's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marlies Cortés with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marlies Cortés more than expected).

Fields of papers citing papers by Marlies Cortés

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marlies Cortés. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marlies Cortés. The network helps show where Marlies Cortés may publish in the future.

Co-authorship network of co-authors of Marlies Cortés

This figure shows the co-authorship network connecting the top 25 collaborators of Marlies Cortés. A scholar is included among the top collaborators of Marlies Cortés based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marlies Cortés. Marlies Cortés is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Cortés, Marlies, Lu Han, Verónica Domínguez, et al.. (2023). Atherosclerotic plaque development in mice is enhanced by myeloid ZEB1 downregulation. Nature Communications. 14(1). 8316–8316. 4 indexed citations
2.
Cortés, Marlies, Verónica Domínguez, Sara Fernández, et al.. (2023). Inflammatory macrophages reprogram to immunosuppression by reducing mitochondrial translation. Nature Communications. 14(1). 7471–7471. 39 indexed citations
3.
Cortés, Marlies, Verónica Domínguez, Lu Han, et al.. (2023). The adaptive antioxidant response during fasting-induced muscle atrophy is oppositely regulated by ZEB1 and ZEB2. Proceedings of the National Academy of Sciences. 120(46). e2301120120–e2301120120. 3 indexed citations
4.
Lu, Xiaoqin, Yao Chen, Beatriz E. Rendon, et al.. (2021). Zeb1 induces immune checkpoints to form an immunosuppressive envelope around invading cancer cells. Science Advances. 7(21). 69 indexed citations
5.
Barrios, Oriol de, Lidia Sánchez-Moral, Marlies Cortés, et al.. (2019). ZEB1 promotes inflammation and progression towards inflammation-driven carcinoma through repression of the DNA repair glycosylase MPG in epithelial cells. Gut. 68(12). 2129–2141. 42 indexed citations
6.
Siles, Laura, et al.. (2019). ZEB1 protects skeletal muscle from damage and is required for its regeneration. Nature Communications. 10(1). 1364–1364. 41 indexed citations
7.
Cortés, Marlies, Lidia Sánchez-Moral, Oriol de Barrios, et al.. (2017). Tumor‐associated macrophages (TAMs) depend on ZEB1 for their cancer‐promoting roles. The EMBO Journal. 36(22). 3336–3355. 114 indexed citations
8.
Sáenz, Leonardo, Andrónico Neira‐Carrillo, Rodolfo Paredes, et al.. (2008). Chitosan formulations improve the immunogenicity of a GnRH-I peptide-based vaccine. International Journal of Pharmaceutics. 369(1-2). 64–71. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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