Marie Regairaz

1.6k total citations
15 papers, 1.2k citations indexed

About

Marie Regairaz is a scholar working on Molecular Biology, Oncology and Neurology. According to data from OpenAlex, Marie Regairaz has authored 15 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 8 papers in Oncology and 4 papers in Neurology. Recurrent topics in Marie Regairaz's work include DNA Repair Mechanisms (7 papers), Cancer therapeutics and mechanisms (4 papers) and Neuroblastoma Research and Treatments (4 papers). Marie Regairaz is often cited by papers focused on DNA Repair Mechanisms (7 papers), Cancer therapeutics and mechanisms (4 papers) and Neuroblastoma Research and Treatments (4 papers). Marie Regairaz collaborates with scholars based in France, United States and Japan. Marie Regairaz's co-authors include Yves Pommier, James H. Doroshow, Keli Agama, Yongwei Zhang, Gabriele Zoppoli, Elisabetta Leo, William C. Reinhold, Sudhir Varma, Alberto Ballestrero and Mirit I. Aladjem and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Nature Communications.

In The Last Decade

Marie Regairaz

15 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Marie Regairaz France 13 897 495 150 112 81 15 1.2k
Xing-Jun Cao United States 12 793 0.9× 255 0.5× 184 1.2× 102 0.9× 42 0.5× 14 1.1k
Corey Lourenco Canada 12 827 0.9× 283 0.6× 189 1.3× 94 0.8× 45 0.6× 13 1.1k
Nancy Wainman Canada 18 686 0.8× 296 0.6× 79 0.5× 166 1.5× 41 0.5× 26 944
Kathleen A. Scorsone United States 15 674 0.8× 350 0.7× 178 1.2× 153 1.4× 20 0.2× 18 1.0k
Yan Degenhardt United States 16 1.1k 1.2× 663 1.3× 223 1.5× 362 3.2× 107 1.3× 23 1.6k
Ahamed Saleem United States 22 1.2k 1.4× 578 1.2× 178 1.2× 183 1.6× 83 1.0× 32 1.5k
B A Teicher United States 11 588 0.7× 407 0.8× 249 1.7× 66 0.6× 136 1.7× 11 902
Eric C. McGary United States 15 628 0.7× 480 1.0× 205 1.4× 70 0.6× 55 0.7× 22 1.1k
David E. MacCallum United Kingdom 11 739 0.8× 472 1.0× 96 0.6× 181 1.6× 44 0.5× 21 1.1k
Ana Slipicevic Norway 15 623 0.7× 311 0.6× 152 1.0× 64 0.6× 61 0.8× 37 823

Countries citing papers authored by Marie Regairaz

Since Specialization
Citations

This map shows the geographic impact of Marie Regairaz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marie Regairaz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marie Regairaz more than expected).

Fields of papers citing papers by Marie Regairaz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marie Regairaz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marie Regairaz. The network helps show where Marie Regairaz may publish in the future.

Co-authorship network of co-authors of Marie Regairaz

This figure shows the co-authorship network connecting the top 25 collaborators of Marie Regairaz. A scholar is included among the top collaborators of Marie Regairaz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marie Regairaz. Marie Regairaz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Musiani, Daniele, Hatice Yücel, Rania El Botty, et al.. (2025). Uracil processing by SMUG1 in the absence of UNG triggers homologous recombination and selectively kills BRCA1/2-deficient tumors. Molecular Cell. 85(6). 1072–1084.e10. 3 indexed citations
2.
Marangoni, Elisabetta, Sophie Vacher, Franck Assayag, et al.. (2018). PKD1 is a potential biomarker and therapeutic target in triple-negative breast cancer. Oncotarget. 9(33). 23208–23219. 13 indexed citations
3.
Poruchynsky, Marianne S., Edina Komlódi-Pásztor, Shana Y. Trostel, et al.. (2015). Microtubule-targeting agents augment the toxicity of DNA-damaging agents by disrupting intracellular trafficking of DNA repair proteins. Proceedings of the National Academy of Sciences. 112(5). 1571–1576. 150 indexed citations
4.
Fu, Haiqing, Melvenia M. Martin, Marie Regairaz, et al.. (2015). The DNA repair endonuclease Mus81 facilitates fast DNA replication in the absence of exogenous damage. Nature Communications. 6(1). 6746–6746. 53 indexed citations
5.
Lindén, Stina, K. David Wegner, Marie Regairaz, et al.. (2015). Terbium-based time-gated Förster resonance energy transfer imaging for evaluating protein–protein interactions on cell membranes. Dalton Transactions. 44(11). 4994–5003. 26 indexed citations
6.
Regairaz, Marie, Hervé Sartelet, Marine Castaing, et al.. (2015). Mutation-Independent Activation of the Anaplastic Lymphoma Kinase in Neuroblastoma. American Journal Of Pathology. 186(2). 435–445. 12 indexed citations
7.
Tasso, Mariana, Emerson Giovanelli, Alexandra Fragola, et al.. (2015). Oriented Bioconjugation of Unmodified Antibodies to Quantum Dots Capped with Copolymeric Ligands as Versatile Cellular Imaging Tools. ACS Applied Materials & Interfaces. 7(48). 26904–26913. 39 indexed citations
8.
Petitprez, Amélie, Virginie Poindessous, Djamila Ouaret, et al.. (2013). Acquired irinotecan resistance is accompanied by stable modifications of cell cycle dynamics independent of MSI status. International Journal of Oncology. 42(5). 1644–1653. 29 indexed citations
9.
Ceccaldi, Raphaël, Kalindi Parmar, Enguerran Mouly, et al.. (2012). Bone Marrow Failure in Fanconi Anemia Is Triggered by an Exacerbated p53/p21 DNA Damage Response that Impairs Hematopoietic Stem and Progenitor Cells. Cell stem cell. 11(1). 36–49. 231 indexed citations
10.
Zoppoli, Gabriele, Marie Regairaz, Elisabetta Leo, et al.. (2012). Putative DNA/RNA helicase Schlafen-11 (SLFN11) sensitizes cancer cells to DNA-damaging agents. Proceedings of the National Academy of Sciences. 109(37). 15030–15035. 245 indexed citations
11.
Regairaz, Marie, Yongwei Zhang, Haiqing Fu, et al.. (2011). Mus81-mediated DNA cleavage resolves replication forks stalled by topoisomerase I–DNA complexes. The Journal of Cell Biology. 195(5). 739–749. 136 indexed citations
12.
Zhang, Yongwei, et al.. (2011). Poly(ADP-ribose) polymerase and XPF–ERCC1 participate in distinct pathways for the repair of topoisomerase I-induced DNA damage in mammalian cells. Nucleic Acids Research. 39(9). 3607–3620. 127 indexed citations
13.
Sartelet, Hervé, Shigeru Ohta, Stéphane Barrette, et al.. (2010). High level of apoptosis and low AKT activation in mass screening as opposed to standard neuroblastoma. Histopathology. 56(5). 607–616. 4 indexed citations
14.
Bax, Dorine A., Nathalie Gaspar, Suzanne E. Little, et al.. (2009). EGFRvIII Deletion Mutations in Pediatric High-Grade Glioma and Response to Targeted Therapy in Pediatric Glioma Cell Lines. Clinical Cancer Research. 15(18). 5753–5761. 71 indexed citations
15.
Calvet, Loreley, Birgit Geoerger, Marie Regairaz, et al.. (2006). Pleiotrophin, a candidate gene for poor tumor vasculature and in vivo neuroblastoma sensitivity to irinotecan. Oncogene. 25(22). 3150–3159. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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