Maria Laura Falchetti
- Molecular Biology
- Genetics top 2%
- Physiology top 10%
- Oncology top 10%
- Cancer Research top 10%
- Co-authors
- Roberto PalliniLuigi Maria LaroccaAndrea LeviGiulio MairaGiulio AlessandriValentina CoccèAugusto PessinaEttore D’Ambrosio
- Topics
- Telomeres, Telomerase, and Senescence (16 papers)Glioma Diagnosis and Treatment (9 papers)Cancer Research and Treatments (8 papers)
- Partner nations
- ItalyUnited StatesMalaysia
In The Last Decade
Maria Laura Falchetti
48 papers receiving 1.4k citations
Peers
Comparison fields: 5 of 89
- Molecular Biology 664
- Genetics 447
- Physiology 295
- Oncology 266
- Cancer Research 233
Countries citing papers authored by Maria Laura Falchetti
This map shows the geographic impact of Maria Laura Falchetti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maria Laura Falchetti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maria Laura Falchetti more than expected).
Fields of papers citing papers by Maria Laura Falchetti
This network shows the impact of papers produced by Maria Laura Falchetti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maria Laura Falchetti. The network helps show where Maria Laura Falchetti may publish in the future.
Co-authorship network of co-authors of Maria Laura Falchetti
This figure shows the co-authorship network connecting the top 25 collaborators of Maria Laura Falchetti. A scholar is included among the top collaborators of Maria Laura Falchetti based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maria Laura Falchetti. Maria Laura Falchetti is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 12 | |
| 2 | 17 | |
| 3 | 7 | |
| 4 | 33 | |
| 5 | 2 | |
| 6 | 21 | |
| 7 | 5 | |
| 8 | 64 | |
| 9 | 44 | |
| 10 | 37 | |
| 11 | 143 | |
| 12 | 17 | |
| 13 | 15 | |
| 14 | 8 | |
| 15 | 80 | |
| 16 | 36 | |
| 17 | Chordoma and chondrosarcoma of the skull base. A series of 24 surgically treated cases and factors predicting early tumor recurrence | 2 |
| 18 | 32 | |
| 19 | 37 | |
| 20 | 27 |
About Maria Laura Falchetti
Maria Laura Falchetti is a scholar working on Genetics, Biotechnology and Physiology, having authored 49 papers that have together received 1.5k indexed citations. Recurring topics across this work include Telomeres, Telomerase, and Senescence (16 papers), Glioma Diagnosis and Treatment (9 papers) and Cancer Research and Treatments (8 papers). The work is most often cited by research in Genetics (447 citations), Developmental Neuroscience (92 citations) and Cancer Research (233 citations). Maria Laura Falchetti has collaborated with scholars based in Italy, United States and Malaysia. Frequent co-authors include Roberto Pallini, Luigi Maria Larocca, Andrea Levi, Giulio Maira, Giulio Alessandri, Valentina Coccè, Augusto Pessina, Ettore D’Ambrosio, Arianna Bonomi and Maria Patrizia Mongiardi. Their work appears in journals such as Nucleic Acids Research, PLoS ONE and Oncogene.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.