Marc S. Lamphier

4.5k total citations · 1 hit paper
19 papers, 3.7k citations indexed

About

Marc S. Lamphier is a scholar working on Immunology, Oncology and Cancer Research. According to data from OpenAlex, Marc S. Lamphier has authored 19 papers receiving a total of 3.7k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Immunology, 6 papers in Oncology and 4 papers in Cancer Research. Recurrent topics in Marc S. Lamphier's work include Immune Response and Inflammation (5 papers), Cytokine Signaling Pathways and Interactions (5 papers) and interferon and immune responses (4 papers). Marc S. Lamphier is often cited by papers focused on Immune Response and Inflammation (5 papers), Cytokine Signaling Pathways and Interactions (5 papers) and interferon and immune responses (4 papers). Marc S. Lamphier collaborates with scholars based in United States, Japan and Canada. Marc S. Lamphier's co-authors include Nobuyuki Tanaka, John Capodici, Houping Ni, Drew Weissman, Katalin Karikó, Tadatsugu Taniguchi, Tak W. Mak, T. Matsuyama, Douglas T. Golenbock and Hisashi Harada and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Marc S. Lamphier

19 papers receiving 3.6k citations

Hit Papers

mRNA Is an Endogenous Ligand for Toll-like Receptor 3 2004 2026 2011 2018 2004 250 500 750

Peers

Marc S. Lamphier
Irene S. Figari United States
Phillip Wong United States
Roger Kurlander United States
Pin‐Yu Perera United States
Cheong‐Hee Chang United States
Irene S. Figari United States
Marc S. Lamphier
Citations per year, relative to Marc S. Lamphier Marc S. Lamphier (= 1×) peers Irene S. Figari

Countries citing papers authored by Marc S. Lamphier

Since Specialization
Citations

This map shows the geographic impact of Marc S. Lamphier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marc S. Lamphier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marc S. Lamphier more than expected).

Fields of papers citing papers by Marc S. Lamphier

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marc S. Lamphier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marc S. Lamphier. The network helps show where Marc S. Lamphier may publish in the future.

Co-authorship network of co-authors of Marc S. Lamphier

This figure shows the co-authorship network connecting the top 25 collaborators of Marc S. Lamphier. A scholar is included among the top collaborators of Marc S. Lamphier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marc S. Lamphier. Marc S. Lamphier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Lamphier, Marc S., Wanjun Zheng, Eicke Latz, et al.. (2013). Novel Small Molecule Inhibitors of TLR7 and TLR9: Mechanism of Action and Efficacy In Vivo. Molecular Pharmacology. 85(3). 429–440. 107 indexed citations
2.
Franklin, Bernardo S., Sally T. Ishizaka, Marc S. Lamphier, et al.. (2011). Therapeutical targeting of nucleic acid-sensing Toll-like receptors prevents experimental cerebral malaria. Proceedings of the National Academy of Sciences. 108(9). 3689–3694. 93 indexed citations
3.
Muramoto, Kazuyo, Yinlu Ding, Hua Yang, et al.. (2010). A novel antagonist of the prostaglandin E2 EP4 receptor inhibits Th1 differentiation and Th17 expansion and is orally active in arthritis models. British Journal of Pharmacology. 160(2). 292–310. 84 indexed citations
4.
Parroche, Peggy, Fanny N. Lauw, Nadège Goutagny, et al.. (2007). Malaria hemozoin is immunologically inert but radically enhances innate responses by presenting malaria DNA to Toll-like receptor 9. Proceedings of the National Academy of Sciences. 104(6). 1919–1924. 426 indexed citations
5.
Latz, Eicke, Anjali Verma, Alberto Visintin, et al.. (2007). Ligand-induced conformational changes allosterically activate Toll-like receptor 9. Nature Immunology. 8(7). 772–779. 375 indexed citations
6.
Lamphier, Marc S., Cherilyn M. Sirois, Anjali Verma, Douglas T. Golenbock, & Eicke Latz. (2006). TLR9 and the Recognition of Self and Non‐Self Nucleic Acids. Annals of the New York Academy of Sciences. 1082(1). 31–43. 78 indexed citations
7.
Karikó, Katalin, Houping Ni, John Capodici, Marc S. Lamphier, & Drew Weissman. (2004). mRNA Is an Endogenous Ligand for Toll-like Receptor 3. Journal of Biological Chemistry. 279(13). 12542–12550. 854 indexed citations breakdown →
8.
Tanaka, Nobuyuki, Mitsuharu Sato, Marc S. Lamphier, et al.. (1998). Type I interferons are essential mediators of apoptotic death in virally infected cells. Genes to Cells. 3(1). 29–37. 144 indexed citations
9.
Taniguchi, Tadatsugu, Marc S. Lamphier, & Nobuyuki Tanaka. (1997). IRF-1: the transcription factor linking the interferon response and oncogenesis. Biochimica et Biophysica Acta (BBA) - Reviews on Cancer. 1333(1). M9–M17. 108 indexed citations
10.
Tamura, Takeshi, Masahiko Ishihara, Marc S. Lamphier, et al.. (1997). DNA damage-induced apoptosis and Ice gene induction in mitogenically activated T lymphocytes require IRF-1.. PubMed. 11 Suppl 3. 439–40. 32 indexed citations
11.
Tanaka, Nobuyuki, Masahiko Ishihara, Marc S. Lamphier, et al.. (1996). Cooperation of the tumour suppressors IRF-1 and p53 in response to DNA damage. Nature. 382(6594). 816–818. 284 indexed citations
12.
Lamphier, Marc S.. (1996). Risky Career Move Pays Off. Science. 274(5284). 54–54. 2 indexed citations
13.
Taniguchi, Tadatsugu, Hisashi Harada, & Marc S. Lamphier. (1995). Regulation of the interferon system and cell growth by the IRF transcription factors. Journal of Cancer Research and Clinical Oncology. 121(9-10). 516–520. 104 indexed citations
14.
Tamura, Tomohiro, Marc S. Lamphier, Nobuyuki Tanaka, et al.. (1995). An IRF-1-dependent pathway of DNA damage-induced apoptosis in mitogen-activated T lymphocytes. Nature. 376(6541). 596–599. 354 indexed citations
15.
Tanaka, Nobuyuki, Masahiko Ishihara, Motoo Kitagawa, et al.. (1994). Cellular commitment to oncogene-induced transformation or apoptosis is dependent on the transcription factor IRF-1. Cell. 77(6). 829–839. 437 indexed citations
16.
Yamamoto, Hiroyasu, Marc S. Lamphier, Toshitsugu Fujita, Toshiyasu Taniguchi, & Hisashi Harada. (1994). The oncogenic transcription factor IRF-2 possesses a transcriptional repression and a latent activation domain.. PubMed. 9(5). 1423–8. 83 indexed citations
17.
Harada, Hisashi, Takahiro Kondo, Sayaka Ogawa, et al.. (1994). Accelerated exon skipping of IRF-1 mRNA in human myelodysplasia/leukemia; a possible mechanism of tumor suppressor inactivation.. PubMed. 9(11). 3313–20. 101 indexed citations
18.
Lamphier, Marc S. & Mark Ptashne. (1992). Multiple mechanisms mediate glucose repression of the yeast GAL1 gene.. Proceedings of the National Academy of Sciences. 89(13). 5922–5926. 48 indexed citations
19.
Lamphier, Marc S. & Kaichiro Yanagisawa. (1983). Induction of Macrocyst Formation by Factors Secreted by Giant Cells in Dictyostelium discoideum. Development Growth & Differentiation. 25(5). 495–501. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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