Maja Tomičić

3.7k total citations
83 papers, 3.0k citations indexed

About

Maja Tomičić is a scholar working on Molecular Biology, Oncology and Hematology. According to data from OpenAlex, Maja Tomičić has authored 83 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 46 papers in Molecular Biology, 22 papers in Oncology and 14 papers in Hematology. Recurrent topics in Maja Tomičić's work include DNA Repair Mechanisms (27 papers), Cancer-related Molecular Pathways (15 papers) and Cell death mechanisms and regulation (10 papers). Maja Tomičić is often cited by papers focused on DNA Repair Mechanisms (27 papers), Cancer-related Molecular Pathways (15 papers) and Cell death mechanisms and regulation (10 papers). Maja Tomičić collaborates with scholars based in Germany, Croatia and United States. Maja Tomičić's co-authors include Bernd Kaina, Markus Christmann, Wynand P. Roos, Beate Köberle, Dorthe Aasland, R Thust, Thomas Efferth, Mirza Bojić, Rolf Rauh and Sabine Grösch and has published in prestigious journals such as Nucleic Acids Research, Cancer Research and Oncogene.

In The Last Decade

Maja Tomičić

80 papers receiving 2.9k citations

Peers

Countries citing papers authored by Maja Tomičić

Since Specialization

Citations

This map shows the geographic impact of Maja Tomičić's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maja Tomičić with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maja Tomičić more than expected).

Fields of papers citing papers by Maja Tomičić

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maja Tomičić. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maja Tomičić. The network helps show where Maja Tomičić may publish in the future.

Co-authorship network of co-authors of Maja Tomičić

This figure shows the co-authorship network connecting the top 25 collaborators of Maja Tomičić. A scholar is included among the top collaborators of Maja Tomičić based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maja Tomičić. Maja Tomičić is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Therapy-induced senescence of glioblastoma cells is determined by the p21CIP1-CDK1/2 axis and does not require activation of DREAM 2025 ·Cell Death and Disease ·(unknown), Jenny Rinke, (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Bernd Kaina, Maja Tomičić, Markus Christmann	3
2	The cell cycle inhibitor p21CIP1 is essential for irinotecan-induced senescence and plays a decisive role in re-sensitization of temozolomide-resistant glioblastoma cells to irinotecan 2024 ·Biomedicine & Pharmacotherapy ·(unknown), (unknown), (unknown), Markus Christmann, Maja Tomičić	3
3	Case report: Suspected transfusion-related acute lung injury type II in a child with refractory systemic juvenile idiopathic arthritis complicated by macrophage activation syndrome 2024 ·Frontiers in Pediatrics ·Alenka Gagro, Maja Tomičić, (unknown), (unknown)	1
4	The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence 2024 ·Nucleic Acids Research ·(unknown), (unknown), (unknown), (unknown), Jia‐Xuan Chen, Georg Nagel, (unknown), (unknown), Petra Beli, Alexander Loewer, Thomas G. Hofmann, Maja Tomičić, Markus Christmann	11
5	Alterations in Molecular Profiles Affecting Glioblastoma Resistance to Radiochemotherapy: Where Does the Good Go? 2022 ·Cancers ·(unknown), Markus Christmann, Maja Tomičić	24
6	Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14ARF-Mediated Sustained p53 Activation 2021 ·Cancers ·Maja Tomičić, Franziska Krämer, (unknown), (unknown), Markus Christmann	19
7	Targeting c-IAP1, c-IAP2, and Bcl-2 Eliminates Senescent Glioblastoma Cells Following Temozolomide Treatment 2021 ·Cancers ·(unknown), (unknown), (unknown), (unknown), (unknown), Bernd Kaina, Maja Tomičić, Markus Christmann	25
8	Benzo[a]pyrene represses DNA repair through altered E2F1/E2F4 function marking an early event in DNA damage-induced cellular senescence 2020 ·Nucleic Acids Research ·(unknown), Laura Mayer, (unknown), Bernd Kaina, Thomas G. Hofmann, Maja Tomičić, Markus Christmann	30
9	Antithrombotic activity of flavonoids and polyphenols rich plant species 2019 ·Acta Pharmaceutica ·Mirza Bojić, Željan Maleš, (unknown), (unknown), Maja Tomičić	51
10	Temozolomide Induces Senescence and Repression of DNA Repair Pathways in Glioblastoma Cells via Activation of ATR–CHK1, p21, and NF-κB 2018 ·Cancer Research ·Dorthe Aasland, (unknown), Laura Hauck, (unknown), (unknown), (unknown), Simon Schneider, (unknown), Wynand P. Roos, Maja Tomičić, Bernd Kaina, Markus Christmann	147
11	Repair gene O⁶‐methylguanine‐DNA methyltransferase is controlled by SP1 and up‐regulated by glucocorticoids, but not by temozolomide and radiation 2017 ·Journal of Neurochemistry ·Dorthe Aasland, (unknown), Maja Tomičić, Olivier J. Switzeny, Bernd Kaina, Markus Christmann	40
12	[Indications and contraindications for platelet transfusions in patients with thrombocytopenia]. 2015 ·PubMed ·Maja Tomičić, Tomislav Vuk, (unknown)	1
13	Translesion Polymerase η Is Upregulated by Cancer Therapeutics and Confers Anticancer Drug Resistance 2014 ·Cancer Research ·Maja Tomičić, Dorthe Aasland, Steffen C. Naumann, (unknown), Christina Barckhausen, Bernd Kaina, Markus Christmann	52
14	INDIKACIJE I KONTRAINDIKACIJE ZA PRIMJENU TROMBOCITNIH TRANSFUZIJA U BOLESNIKA S TROMBOCITOPENIJOM 2014 ·University of Zagreb University Computing Centre (SRCE) ·Maja Tomičić, Tomislav Vuk, (unknown)	1
15	Cisplatin resistance: Preclinical findings and clinical implications 2010 ·Biochimica et Biophysica Acta (BBA) - Reviews on Cancer ·Beate Köberle, Maja Tomičić, (unknown), Bernd Kaina	305
16	Three prime exonuclease I (TREX1) is Fos/AP-1 regulated by genotoxic stress and protects against ultraviolet light and benzo(a)pyrene-induced DNA damage 2010 ·Nucleic Acids Research ·Markus Christmann, Maja Tomičić, Dorthe Aasland, (unknown), Bernd Kaina	54
17	5, 10-Methylenetetrahydrofolate reductase (MTHFR) 677 C --> T genetic polymorphism in 228 Croatian volunteers. 2004 ·PubMed ·(unknown), (unknown), Maja Tomičić, Nada Vrkić, (unknown), (unknown), Zdenko Sonicki, (unknown)	10
18	BER, MGMT, and MMR in defense against alkylation-induced genotoxicity and apoptosis 2001 ·Progress in nucleic acid research and molecular biology ·Bernd Kaina, (unknown), Sabine Grösch, (unknown), (unknown), Maja Tomičić, Torsten Dunkern, Markus Christmann	81
19	Comparison of the genotoxic and apoptosis-inducing properties of ganciclovir and penciclovir in Chinese hamster ovary cells transfected with the thymidine kinase gene of herpes simplex virus-1: Implications for gene therapeutic approaches 2000 ·Cancer Gene Therapy ·R Thust, Maja Tomičić, R. Klöcking, (unknown), P Wutzler, Bernd Kaina	46
20	[Nationally produced 153SmEDTMP in the palliative treatment of metastatic bone cancer pain]. 1996 ·PubMed ·Elena Olea, (unknown), Maja Tomičić, (unknown), (unknown), (unknown), Luísa Pinto, (unknown), (unknown), (unknown)	2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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