Magda Giòrdano

3.1k total citations
77 papers, 2.6k citations indexed

About

Magda Giòrdano is a scholar working on Cellular and Molecular Neuroscience, Cognitive Neuroscience and Social Psychology. According to data from OpenAlex, Magda Giòrdano has authored 77 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Cellular and Molecular Neuroscience, 20 papers in Cognitive Neuroscience and 14 papers in Social Psychology. Recurrent topics in Magda Giòrdano's work include Neuroscience and Neuropharmacology Research (26 papers), Neurotransmitter Receptor Influence on Behavior (22 papers) and Genetic Neurodegenerative Diseases (8 papers). Magda Giòrdano is often cited by papers focused on Neuroscience and Neuropharmacology Research (26 papers), Neurotransmitter Receptor Influence on Behavior (22 papers) and Genetic Neurodegenerative Diseases (8 papers). Magda Giòrdano collaborates with scholars based in Mexico, United States and Argentina. Magda Giòrdano's co-authors include Andrew B. Norman, Verónica M. Rodríguez, Michael Bunsey, Paul R. Sanberg, Paul R. Sanberg, Leticia Carrizales, Jorge H. Limón-Pacheco, Ulises Bardullas, L Dufour and María E. Jiménez‐Capdeville and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Brain Research.

In The Last Decade

Magda Giòrdano

73 papers receiving 2.5k citations

Peers

Magda Giòrdano

CMN

NEURO

HTM

W. Michael Caudle United States

Jason R. Cannon United States

Robert C. MacPhail United States

H.A. Tilson United States

Richard M. Kostrzewa United States

Daniela M. Barros Brazil

Ruth M. Empson United Kingdom

Małgorzata Kajta Poland

R.J. Boegman Canada

Gerrit Wolterink Netherlands

W. Michael Caudle United States

Magda Giòrdano

1.3k ×1.1

CMN

746 ×1.1

1.5k ×3.6

NEURO

624 ×1.7

HTM

215 ×0.6

Citations per year, relative to Magda Giòrdano Magda Giòrdano (= 1×) peers W. Michael Caudle

Countries citing papers authored by Magda Giòrdano

Since Specialization

Citations

This map shows the geographic impact of Magda Giòrdano's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Magda Giòrdano with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Magda Giòrdano more than expected).

Fields of papers citing papers by Magda Giòrdano

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Magda Giòrdano. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Magda Giòrdano. The network helps show where Magda Giòrdano may publish in the future.

Co-authorship network of co-authors of Magda Giòrdano

This figure shows the co-authorship network connecting the top 25 collaborators of Magda Giòrdano. A scholar is included among the top collaborators of Magda Giòrdano based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Magda Giòrdano. Magda Giòrdano is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Giòrdano, Magda, et al.. (2024). Hypoactivity and neurochemical alterations in the basal ganglia of female Sprague-Dawley rats after repeated exposure to atrazine. SHILAP Revista de lepidopterología. 6. 1416708–1416708.

Corona, Rebeca, et al.. (2024). Early and transitory hypoactivity and olfactory alterations after chronic atrazine exposure in female Sprague-Dawley rats. NeuroToxicology. 101. 68–81. 4 indexed citations

Giòrdano, Magda, et al.. (2023). Contribution and functional connectivity between cerebrum and cerebellum on sub-lexical and lexical-semantic processing of verbs. PLoS ONE. 18(9). e0291558–e0291558. 2 indexed citations

Giòrdano, Magda, et al.. (2022). Chronic atrazine exposure increases the expression of genes associated with GABAergic and glutamatergic systems in the brain of male albino rat. SHILAP Revista de lepidopterología. 4. 933300–933300. 5 indexed citations

Giòrdano, Magda, et al.. (2020). Brain alterations in GABA, glutamate and glutamine markers after chronic atrazine exposure in the male albino rat. Archives of Toxicology. 94(9). 3217–3230. 19 indexed citations

Rodríguez, Verónica M., et al.. (2016). Risk-taking, locomotor activity and dopamine levels in the nucleus accumbens and medial prefrontal cortex in male rats treated prenatally with alcohol. Pharmacology Biochemistry and Behavior. 153. 88–96. 18 indexed citations

Giòrdano, Magda, et al.. (2014). The herbicide glyphosate causes behavioral changes and alterations in dopaminergic markers in male Sprague-Dawley rat. NeuroToxicology. 46. 79–91. 60 indexed citations

Bardullas, Ulises, Magda Giòrdano, & Verónica M. Rodríguez. (2013). Atrazine is primarily responsible for the toxicity of long-term exposure to a combination of atrazine and inorganic arsenic in the nigrostriatal system of the albino rat. Neurotoxicology and Teratology. 40. 59–66. 25 indexed citations

Giòrdano, Magda, et al.. (2009). Spatial memory: Theoretical basis and comparative review on experimental methods in rodents. Behavioural Brain Research. 203(2). 151–164. 155 indexed citations

10.

Peña‐Ortega, Fernando, et al.. (2009). Beta‐amyloid protein (25–35) disrupts hippocampal network activity: Role of Fyn‐kinase. Hippocampus. 20(1). 78–96. 64 indexed citations

11.

Bardullas, Ulises, et al.. (2008). Chronic low-level arsenic exposure causes gender-specific alterations in locomotor activity, dopaminergic systems, and thioredoxin expression in mice. Toxicology and Applied Pharmacology. 239(2). 169–177. 93 indexed citations

12.

Aguilar, Manuel B., et al.. (2008). Intranigral transplants of a GABAergic cell line produce long-term alleviation of established motor seizures. Behavioural Brain Research. 193(1). 17–27. 24 indexed citations

13.

Freed, William J., et al.. (2006). Intranigral transplants of immortalized GABAergic cells decrease the expression of kainic acid-induced seizures in the rat. Behavioural Brain Research. 171(1). 109–115. 35 indexed citations

14.

Gutiérrez‐Ospina, Gabriel, et al.. (2002). Insulin-like growth factor I partly prevents axon elimination in the neonate rat optic nerve. Neuroscience Letters. 325(3). 207–210. 10 indexed citations

15.

Giòrdano, Magda, et al.. (2001). Combined mesencephalic and hypothalamic transplants reverse lesion-induced sexual behavior deficits in the male rat. Behavioural Brain Research. 120(1). 97–104. 5 indexed citations

16.

Giòrdano, Magda, et al.. (1993). Immortalized GABAergic Cell Lines Derived from Rat Striatum Using a Temperature-Sensitive Allele of the SV40 Large T Antigen. Experimental Neurology. 124(2). 395–400. 51 indexed citations

17.

Giòrdano, Magda, et al.. (1990). Behavioral effects of neural transplants into the intact striatum. Pharmacology Biochemistry and Behavior. 37(1). 135–148. 14 indexed citations

18.

Sanberg, Paul R., et al.. (1990). Tyrosine hydroxylase-positive fibers and neurons in transplanted striatal tissue in rats with quinolinic acid lesions of the striatum. Brain Research Bulletin. 25(6). 889–894. 6 indexed citations

19.

Norman, Andrew B., Magda Giòrdano, & Paul R. Sanberg. (1989). Fetal striatal tissue grafts into excitotoxin-lesioned striatum: Pharmacological and behavioral aspects. Pharmacology Biochemistry and Behavior. 34(1). 139–147. 23 indexed citations

20.

Norman, Andrew B., et al.. (1988). Striatal tissue transplants attenuate apomorphine-induced rotational behavior in rats with unilateral kainic acid lesions. Neuropharmacology. 27(3). 333–336. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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