M Ui

14.0k total citations · 7 hit papers
99 papers, 12.1k citations indexed

About

M Ui is a scholar working on Molecular Biology, Surgery and Genetics. According to data from OpenAlex, M Ui has authored 99 papers receiving a total of 12.1k indexed citations (citations by other indexed papers that have themselves been cited), including 75 papers in Molecular Biology, 29 papers in Surgery and 21 papers in Genetics. Recurrent topics in M Ui's work include Pancreatic function and diabetes (29 papers), Receptor Mechanisms and Signaling (22 papers) and Diabetes and associated disorders (16 papers). M Ui is often cited by papers focused on Pancreatic function and diabetes (29 papers), Receptor Mechanisms and Signaling (22 papers) and Diabetes and associated disorders (16 papers). M Ui collaborates with scholars based in Japan, United States and China. M Ui's co-authors include Toshiaki Katada, Toshihiko Murayama, Fumikazu Okajima, Osamu Hazeki, Taro Okada, Gary Bokoch, Alfred G. Gilman, T. Nakamura, Masayuki Oinuma and John K. Northup and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The EMBO Journal.

In The Last Decade

M Ui

98 papers receiving 11.5k citations

Hit Papers

Essential role of phospha... 1982 2026 1996 2011 1994 1982 1982 1985 1984 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Essential role of phosphatidylinositol 3-kinase in insulin-induced glucose transport and antilipolysis in rat adipocytes. Studies with a selective inhibitor wortmannin.
    1994 820 citations Taro Okada, Osamu Hazeki et al. Journal of Biological Chemistry profile →
  2. Direct modification of the membrane adenylate cyclase system by islet-activating protein due to ADP-ribosylation of a membrane protein.
    1982 673 citations Toshiaki Katada, M Ui Proceedings of the National Academy of Sciences profile →
  3. ADP ribosylation of the specific membrane protein of C6 cells by islet-activating protein associated with modification of adenylate cyclase activity.
    1982 526 citations Toshiaki Katada, M Ui Journal of Biological Chemistry profile →
  4. Simultaneous inhibitions of inositol phospholipid breakdown, arachidonic acid release, and histamine secretion in mast cells by islet-activating protein, pertussis toxin. A possible involvement of the toxin-specific substrate in the Ca2+-mobilizing receptor-mediated biosignaling system.
    1985 448 citations T. Nakamura, M Ui Journal of Biological Chemistry profile →
  5. Purification and properties of the inhibitory guanine nucleotide-binding regulatory component of adenylate cyclase.
    1984 427 citations Toshiaki Katada, M Ui et al. Journal of Biological Chemistry profile →
  6. The inhibitory guanine nucleotide-binding regulatory component of adenylate cyclase. Properties and function of the purified protein.
    1984 355 citations Toshiaki Katada, M Ui et al. Journal of Biological Chemistry profile →
  7. The inhibitory guanine nucleotide-binding regulatory component of adenylate cyclase. Subunit dissociation and guanine nucleotide-dependent hormonal inhibition.
    1984 294 citations Toshiaki Katada, M Ui et al. Journal of Biological Chemistry profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
M Ui 9.2k 2.5k 2.2k 1.5k 1.4k 99 12.1k
Paul C. Sternweis 12.7k 1.4× 3.4k 1.4× 3.5k 1.6× 1.3k 0.9× 848 0.6× 103 15.2k
Michio Ui 6.2k 0.7× 1.5k 0.6× 1.6k 0.7× 1.4k 1.0× 958 0.7× 180 9.4k
Toshiaki Katada 13.1k 1.4× 3.5k 1.4× 3.7k 1.7× 1.7k 1.2× 1.5k 1.1× 300 18.1k
Karl H. Jakobs 7.7k 0.8× 2.1k 0.8× 2.0k 0.9× 1.3k 0.9× 664 0.5× 175 10.0k
Werner Schlegel 4.9k 0.5× 1.6k 0.6× 801 0.4× 1.1k 0.7× 1.4k 1.0× 206 8.6k
Richard T. Premont 11.4k 1.2× 5.2k 2.1× 2.9k 1.3× 1.4k 1.0× 1.1k 0.8× 166 15.3k
Fumikazu Okajima 7.1k 0.8× 924 0.4× 1.8k 0.8× 1.6k 1.1× 1.2k 0.9× 197 9.9k
Shamshad Cockcroft 8.4k 0.9× 1.2k 0.5× 4.9k 2.2× 2.0k 1.3× 1.4k 1.0× 190 11.9k
Kazuyoshi Yonezawa 9.3k 1.0× 893 0.4× 2.0k 0.9× 1.3k 0.9× 1.3k 0.9× 97 11.9k
Peter Gierschik 6.9k 0.8× 1.6k 0.7× 1.6k 0.7× 923 0.6× 592 0.4× 173 9.7k

Countries citing papers authored by M Ui

Since Specialization
Citations

This map shows the geographic impact of M Ui's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M Ui with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M Ui more than expected).

Fields of papers citing papers by M Ui

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M Ui. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M Ui. The network helps show where M Ui may publish in the future.

Co-authorship network of co-authors of M Ui

This figure shows the co-authorship network connecting the top 25 collaborators of M Ui. A scholar is included among the top collaborators of M Ui based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M Ui. M Ui is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sasaki, Takehiko, Kaoru Hazeki, Osamu Hazeki, M Ui, & Toshiaki Katada. (1995). Permissive effect of ceramide on growth factor-induced cell proliferation. Biochemical Journal. 311(3). 829–834. 26 indexed citations
2.
Ishii, Isao & M Ui. (1994). Possible Involvement of GTP-Binding Proteins in 1α,25-Dihydroxyvitamin D3 Induction of Tissue Transglutaminase in Mouse Peritoneal Macrophages. Biochemical and Biophysical Research Communications. 203(3). 1773–1780. 6 indexed citations
3.
Kanai, Fumihiko, K. Ito, M. Todaka, et al.. (1993). Insulin-Stimulated GLUT4 Translocation Is Relevant to the Phosphorylation of IRS-1 and the Activity of PI3 Kinase. Biochemical and Biophysical Research Communications. 195(2). 762–768. 255 indexed citations
4.
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Ito, Hiroyuki, Robert Tung, T Sugimoto, et al.. (1992). On the mechanism of G protein beta gamma subunit activation of the muscarinic K+ channel in guinea pig atrial cell membrane. Comparison with the ATP-sensitive K+ channel.. The Journal of General Physiology. 99(6). 961–983. 159 indexed citations
6.
Ito, Hiroyuki, T Sugimoto, Ichiro Kobayashi, et al.. (1991). On the mechanism of basal and agonist-induced activation of the G protein-gated muscarinic K+ channel in atrial myocytes of guinea pig heart.. The Journal of General Physiology. 98(3). 517–533. 62 indexed citations
7.
Katada, Toshiaki & M Ui. (1988). Unique Properties of a New GTP-binding Protein with a Molecular Mass of 24,000 Daltons Purified from Porcine Brain Membranes. Cold Spring Harbor Symposia on Quantitative Biology. 53(0). 255–261. 3 indexed citations
8.
Oinuma, Masayuki, Toshiaki Katada, & M Ui. (1987). A new GTP-binding protein in differentiated human leukemic (HL-60) cells serving as the specific substrate of islet-activating protein, pertussis toxin.. Journal of Biological Chemistry. 262(17). 8347–8353. 80 indexed citations
9.
Katada, Toshiaki, Masayuki Oinuma, & M Ui. (1986). Mechanisms for inhibition of the catalytic activity of adenylate cyclase by the guanine nucleotide-binding proteins serving as the substrate of islet-activating protein, pertussis toxin.. Journal of Biological Chemistry. 261(11). 5215–5221. 195 indexed citations
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Nakamura, T. & M Ui. (1985). Simultaneous inhibitions of inositol phospholipid breakdown, arachidonic acid release, and histamine secretion in mast cells by islet-activating protein, pertussis toxin. A possible involvement of the toxin-specific substrate in the Ca2+-mobilizing receptor-mediated biosignaling system.. Journal of Biological Chemistry. 260(6). 3584–3593. 448 indexed citations breakdown →
13.
14.
Kurose, Hitoshi, Toshiaki Katada, Tomoko Amano, & M Ui. (1983). Specific uncoupling by islet-activating protein, pertussis toxin, of negative signal transduction via alpha-adrenergic, cholinergic, and opiate receptors in neuroblastoma x glioma hybrid cells.. Journal of Biological Chemistry. 258(8). 4870–4875. 440 indexed citations
15.
Katada, Toshiaki & M Ui. (1982). Direct modification of the membrane adenylate cyclase system by islet-activating protein due to ADP-ribosylation of a membrane protein.. Proceedings of the National Academy of Sciences. 79(10). 3129–3133. 673 indexed citations breakdown →
16.
Fujimoto, K., Takemasa Sakaguchi, & M Ui. (1981). Adrenergic mechanisms in the hyperglycaemia and hyperinsulinaemia of diabetic KK mice. Diabetologia. 20(5). 568–72. 14 indexed citations
17.
Hata, Shun-ichi, Futoshi Okada, Miyuki Honma, & M Ui. (1980). Plasma cyclic 3′,5′-guanosine monophosphate and cyclic 3′,5′-adenosine monophosphate response to methacholine in man. Cellular and Molecular Life Sciences. 36(9). 1127–1128. 2 indexed citations
18.
19.
Katada, Toshiaki & M Ui. (1976). Accelerated turnover of blood glucose in pertussis-sensitized rats due to combined actions of endogenous insulin and adrenergic beta-stimulation. Biochimica et Biophysica Acta (BBA) - General Subjects. 421(1). 57–69. 25 indexed citations
20.
Ui, M. (1975). [Adrenergic control of blood sugar level--in vivo studies on the mechanism of metabolic regulations (author's transl)].. PubMed. 47(9). 779–807. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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