M. Eugenia Delgado

1.6k total citations
26 papers, 1.2k citations indexed

About

M. Eugenia Delgado is a scholar working on Molecular Biology, Epidemiology and Pathology and Forensic Medicine. According to data from OpenAlex, M. Eugenia Delgado has authored 26 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 7 papers in Epidemiology and 5 papers in Pathology and Forensic Medicine. Recurrent topics in M. Eugenia Delgado's work include Cell death mechanisms and regulation (9 papers), Autophagy in Disease and Therapy (5 papers) and Mitochondrial Function and Pathology (4 papers). M. Eugenia Delgado is often cited by papers focused on Cell death mechanisms and regulation (9 papers), Autophagy in Disease and Therapy (5 papers) and Mitochondrial Function and Pathology (4 papers). M. Eugenia Delgado collaborates with scholars based in Spain, Germany and Ireland. M. Eugenia Delgado's co-authors include Michael H. Gordon, María Pilar Almajano, Thomas Brunner, Markus Rehm, Thomas Grabinger, Maike A. Laussmann, Paloma Morales, Ana I. Haza, Rosa Carbó and Martina Haller and has published in prestigious journals such as Journal of Biological Chemistry, Molecular Cell and Gastroenterology.

In The Last Decade

M. Eugenia Delgado

26 papers receiving 1.2k citations

Peers

M. Eugenia Delgado
Sridhar Radhakrishnan United States
Jeong‐Heon Ko South Korea
Jae In Jung South Korea
Susan J. Zunino United States
Sridhar Radhakrishnan United States
M. Eugenia Delgado
Citations per year, relative to M. Eugenia Delgado M. Eugenia Delgado (= 1×) peers Sridhar Radhakrishnan

Countries citing papers authored by M. Eugenia Delgado

Since Specialization
Citations

This map shows the geographic impact of M. Eugenia Delgado's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. Eugenia Delgado with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. Eugenia Delgado more than expected).

Fields of papers citing papers by M. Eugenia Delgado

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. Eugenia Delgado. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. Eugenia Delgado. The network helps show where M. Eugenia Delgado may publish in the future.

Co-authorship network of co-authors of M. Eugenia Delgado

This figure shows the co-authorship network connecting the top 25 collaborators of M. Eugenia Delgado. A scholar is included among the top collaborators of M. Eugenia Delgado based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. Eugenia Delgado. M. Eugenia Delgado is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Delgado, M. Eugenia, Salvador Naranjo‐Suarez, Eulàlia Belloc, et al.. (2024). CPEB4 modulates liver cancer progression by translationally regulating hepcidin expression and sensitivity to ferroptosis. JHEP Reports. 7(3). 101296–101296. 3 indexed citations
2.
Lambrecht, R, M. Eugenia Delgado, Anna Pia Plazzo, et al.. (2023). Liver receptor homolog-1 (NR5A2) orchestrates hepatic inflammation and TNF-induced cell death. Cell Reports. 42(12). 113513–113513. 10 indexed citations
3.
Hellwig, Christian T., M. Eugenia Delgado, Josip Skoko, et al.. (2021). Proteasome inhibition triggers the formation of TRAIL receptor 2 platforms for caspase-8 activation that accumulate in the cytosol. Cell Death and Differentiation. 29(1). 147–155. 9 indexed citations
4.
Delgado, M. Eugenia, et al.. (2021). Metabolic Reprogramming of Liver Fibrosis. Cells. 10(12). 3604–3604. 46 indexed citations
5.
Huang, Juan, Truong San Phan, Christian Schmidt, et al.. (2019). The orphan nuclear receptor LRH-1/NR5a2 critically regulates T cell functions. Science Advances. 5(7). eaav9732–eaav9732. 18 indexed citations
6.
Delgado, M. Eugenia & Thomas Brunner. (2019). The many faces of tumor necrosis factor signaling in the intestinal epithelium. Genes and Immunity. 20(8). 609–626. 36 indexed citations
7.
Delp, Johannes, M. Eugenia Delgado, Yan Niu, et al.. (2019). Thiazolides promote G1 cell cycle arrest in colorectal cancer cells by targeting the mitochondrial respiratory chain. Oncogene. 39(11). 2345–2357. 31 indexed citations
8.
Grabinger, Thomas, M. Eugenia Delgado, Feodora Ivanova Kostadinova, et al.. (2016). Inhibitor of Apoptosis Protein-1 Regulates Tumor Necrosis Factor–Mediated Destruction of Intestinal Epithelial Cells. Gastroenterology. 152(4). 867–879. 57 indexed citations
9.
Würstle, Maximilian L., et al.. (2015). An Analysis of the Truncated Bid- and ROS-dependent Spatial Propagation of Mitochondrial Permeabilization Waves during Apoptosis. Journal of Biological Chemistry. 291(9). 4603–4613. 8 indexed citations
10.
Ichim, Gabriel, Jonathan Lopez, Shafiq U. Ahmed, et al.. (2015). Limited Mitochondrial Permeabilization Causes DNA Damage and Genomic Instability in the Absence of Cell Death. Molecular Cell. 57(5). 860–872. 336 indexed citations
11.
Delgado, M. Eugenia, Lydia Dyck, Maike A. Laussmann, & Markus Rehm. (2014). Modulation of apoptosis sensitivity through the interplay with autophagic and proteasomal degradation pathways. Cell Death and Disease. 5(1). e1011–e1011. 43 indexed citations
12.
Delgado, M. Eugenia, et al.. (2013). Determining the contributions of caspase-2, caspase-8 and effector caspases to intracellular VDVADase activities during apoptosis initiation and execution. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1833(10). 2279–2292. 18 indexed citations
13.
Flanagan, Lorna, et al.. (2011). Dimerization of Smac is crucial for its mitochondrial retention by XIAP subsequent to mitochondrial outer membrane permeabilization. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1813(5). 819–826. 5 indexed citations
14.
Laussmann, Maike A., Egle Passante, Heiko Düßmann, et al.. (2011). Proteasome inhibition can induce an autophagy-dependent apical activation of caspase-8. Cell Death and Differentiation. 18(10). 1584–1597. 115 indexed citations
15.
Morales, Paloma, et al.. (2009). Antiapoptotic effects of dietary antioxidants towards N‐nitrosopiperidine and N‐nitrosodibutylamine‐induced apoptosis in HL‐60 and HepG2 cells. Journal of Applied Toxicology. 29(5). 403–413. 23 indexed citations
16.
Delgado, M. Eugenia, Ana I. Haza, Ana Isabel García García, & Paloma Morales. (2009). Myricetin, quercetin, (+)-catechin and (−)-epicatechin protect against N-nitrosamines-induced DNA damage in human hepatoma cells. Toxicology in Vitro. 23(7). 1292–1297. 25 indexed citations
17.
García, Ana Isabel García, et al.. (2008). Organosulfur compounds alone or in combination with vitamin C protect towards N-nitrosopiperidine- and N-nitrosodibutylamine-induced oxidative DNA damage in HepG2 cells. Chemico-Biological Interactions. 173(1). 9–18. 16 indexed citations
18.
19.
Almajano, María Pilar, Rosa Carbó, M. Eugenia Delgado, & Michael H. Gordon. (2007). Effect of pH on the Antimicrobial Activity and Oxidative Stability of Oil‐in‐Water Emulsions Containing Caffeic Acid. Journal of Food Science. 72(5). C258–63. 80 indexed citations
20.
Almajano, María Pilar, M. Eugenia Delgado, & Michael H. Gordon. (2006). Albumin causes a synergistic increase in the antioxidant activity of green tea catechins in oil-in-water emulsions. Food Chemistry. 102(4). 1375–1382. 68 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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