M. E. Jackson

473 total citations
18 papers, 404 citations indexed

About

M. E. Jackson is a scholar working on Molecular Biology, Epidemiology and Genetics. According to data from OpenAlex, M. E. Jackson has authored 18 papers receiving a total of 404 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 7 papers in Epidemiology and 5 papers in Genetics. Recurrent topics in M. E. Jackson's work include Cervical Cancer and HPV Research (7 papers), RNA and protein synthesis mechanisms (5 papers) and Bacterial Genetics and Biotechnology (4 papers). M. E. Jackson is often cited by papers focused on Cervical Cancer and HPV Research (7 papers), RNA and protein synthesis mechanisms (5 papers) and Bacterial Genetics and Biotechnology (4 papers). M. E. Jackson collaborates with scholars based in United Kingdom, Switzerland and Spain. M. E. Jackson's co-authors include Julie M. Pratt, I. Barry Holland, M. Saveria Campo, Guillermo Grindlay, Iain M. Morgan, Neil G. Stoker, William D. Pennie, Kenneth T. Smith, Nigel Mackman and K. T. Smith and has published in prestigious journals such as The EMBO Journal, Journal of Molecular Biology and Oncogene.

In The Last Decade

M. E. Jackson

18 papers receiving 384 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
M. E. Jackson United Kingdom 13 231 199 130 68 57 18 404
S Sawada Japan 12 213 0.9× 67 0.3× 56 0.4× 29 0.4× 141 2.5× 16 479
Twyla Juehne United States 8 131 0.6× 88 0.4× 52 0.4× 93 1.4× 119 2.1× 9 361
Jason Larson United States 7 118 0.5× 72 0.4× 69 0.5× 164 2.4× 45 0.8× 8 382
Dmitri Kamashev Russia 12 442 1.9× 242 1.2× 48 0.4× 63 0.9× 33 0.6× 17 597
J. Pohlner Germany 7 214 0.9× 219 1.1× 50 0.4× 192 2.8× 75 1.3× 8 528
Maria Giuseppina Borri Italy 6 175 0.8× 103 0.5× 53 0.4× 182 2.7× 76 1.3× 7 349
Robert Bever United States 6 285 1.2× 169 0.8× 12 0.1× 27 0.4× 80 1.4× 8 436
Angel C.Y. Yu Canada 8 205 0.9× 183 0.9× 30 0.2× 20 0.3× 41 0.7× 8 437
Heather M. Curry United States 9 182 0.8× 41 0.2× 98 0.8× 28 0.4× 118 2.1× 10 358
Nihal A. Okan United States 11 509 2.2× 191 1.0× 40 0.3× 13 0.2× 63 1.1× 13 654

Countries citing papers authored by M. E. Jackson

Since Specialization
Citations

This map shows the geographic impact of M. E. Jackson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. E. Jackson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. E. Jackson more than expected).

Fields of papers citing papers by M. E. Jackson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. E. Jackson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. E. Jackson. The network helps show where M. E. Jackson may publish in the future.

Co-authorship network of co-authors of M. E. Jackson

This figure shows the co-authorship network connecting the top 25 collaborators of M. E. Jackson. A scholar is included among the top collaborators of M. E. Jackson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. E. Jackson. M. E. Jackson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Morgan, Iain M., et al.. (1998). The BPV-4 co-carcinogen quercetin induces cell cycle arrest and up-regulates transcription from the LCR of BPV-4. Oncogene. 16(21). 2739–2746. 20 indexed citations
2.
Scobie, Linda, M. Saveria Campo, & M. E. Jackson. (1997). The role of exogenous p53 and E6 oncoproteins in in vitro transformation by bovine papillomavirus type 4 (BPV-4): significance of the absence of an E6 ORF in the BPV-4 genome.. Journal of General Virology. 78(11). 3001–3008. 9 indexed citations
3.
Jackson, M. E., Vincent O’Brien, Iain M. Morgan, Guillermo Grindlay, & M. Saveria Campo. (1996). Bovine papillomavirus type 4. International Journal of Oncology. 9(6). 1189–99. 11 indexed citations
4.
Coggins, Lesley W., Linda Scobie, M. E. Jackson, & M. Saveria Campo. (1995). Assignment of the bovine p53 gene (TP53) to Chromosome 19q15 by fluorescence in situ hybridization. Mammalian Genome. 6(9). 687–688. 5 indexed citations
5.
Jackson, M. E. & M. Saveria Campo. (1995). Both viral E2 protein and the cellular factor PEBP2 regulate transcription via E2 consensus sites within the bovine papillomavirus type 4 long control region. Journal of Virology. 69(10). 6038–6046. 25 indexed citations
6.
Jackson, M. E., et al.. (1994). An Element Binding a C/EBP-related Transcription Factor Contributes to Negative Regulation of the Bovine Papillomavirus Type 4 Long Control Region. Journal of General Virology. 75(11). 3047–3056. 15 indexed citations
7.
Jackson, M. E., et al.. (1991). The B subgroup bovine papillomaviruses lack an identifiable E6 open reading frame. Molecular Carcinogenesis. 4(5). 382–387. 41 indexed citations
8.
Jackson, M. E. & M. Saveria Campo. (1991). Positive and negative E2-independent regulatory elements in the long control region of bovine papillomavirus type 4. Journal of General Virology. 72(4). 877–883. 12 indexed citations
9.
Jackson, M. E.. (1991). Commentary negative regulation of eukaryotic transcription. Journal of Cell Science. 100(1). 1–7. 33 indexed citations
10.
Smith, K. T., et al.. (1990). Cooperation Between Bovine Papillomavirus Type 4 and ras in the Morphological Transformation of Primary Bovine Fibroblasts. Journal of General Virology. 71(12). 3041–3046. 25 indexed citations
11.
Jackson, M. E. & Julie M. Pratt. (1988). Analysis of the membrane‐binding domain of penicillin‐binding protein 5 of Escherichia coli. Molecular Microbiology. 2(5). 563–568. 21 indexed citations
12.
Mackman, Nigel, et al.. (1987). Role of SecA and SecY in protein export as revealed by studies of TonA assembly into the outer membrane of Escherichia coli. Journal of Molecular Biology. 198(4). 693–703. 19 indexed citations
13.
Jackson, M. E. & Julie M. Pratt. (1987). An 18 amino acid amphiphilic helix forms the membrane‐anchoring domain of the Escherichia coli penicillin‐binding protein 5. Molecular Microbiology. 1(3). 23–28. 41 indexed citations
14.
Pratt, Julie M., M. E. Jackson, & I. Barry Holland. (1986). The C terminus of penicillin-binding protein 5 is essential for localisation to the E. coli inner membrane.. The EMBO Journal. 5(9). 2399–2405. 57 indexed citations
15.
Jackson, M. E., Julie M. Pratt, & I. Barry Holland. (1986). Intermediates in the assembly of the TonA polypeptide into the outer membrane of Escherichia coli K12. Journal of Molecular Biology. 189(3). 477–486. 19 indexed citations
16.
Jackson, M. E., Julie M. Pratt, Neil G. Stoker, & I. Barry Holland. (1985). An inner membrane protein N-terminal signal sequence is able to promote efficient localisation of an outer membrane protein in Escherichia coli.. The EMBO Journal. 4(9). 2377–2383. 29 indexed citations
17.
Jackson, M. E., Julie M. Pratt, & I. Barry Holland. (1983). Enhanced polypeptide synthesis programmed by linear DNA fragments in cell‐free extracts lacking exonuclease V. FEBS Letters. 163(2). 221–224. 6 indexed citations
18.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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