Lukas Geisler

689 total citations
17 papers, 535 citations indexed

About

Lukas Geisler is a scholar working on Epidemiology, Oncology and Molecular Biology. According to data from OpenAlex, Lukas Geisler has authored 17 papers receiving a total of 535 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Epidemiology, 6 papers in Oncology and 5 papers in Molecular Biology. Recurrent topics in Lukas Geisler's work include Liver Disease Diagnosis and Treatment (7 papers), Neuroendocrine Tumor Research Advances (6 papers) and Lung Cancer Research Studies (6 papers). Lukas Geisler is often cited by papers focused on Liver Disease Diagnosis and Treatment (7 papers), Neuroendocrine Tumor Research Advances (6 papers) and Lung Cancer Research Studies (6 papers). Lukas Geisler collaborates with scholars based in Germany, United States and Netherlands. Lukas Geisler's co-authors include Alexander Wree, Ariel E. Feldstein, Hal M. Hoffman, Bettina G. Papouchado, María Eugenia Inzaugarat, Matthew D. McGeough, Casey D. Johnson, Carla A. Peña, Akiko Eguchi and Susanne Schuster and has published in prestigious journals such as PLoS ONE, Hepatology and International Journal of Molecular Sciences.

In The Last Decade

Lukas Geisler

16 papers receiving 531 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Lukas Geisler Germany 9 249 216 144 97 87 17 535
Kengo Kawai Japan 11 342 1.4× 130 0.6× 181 1.3× 87 0.9× 60 0.7× 26 584
Carlos Sanz‐García Spain 10 297 1.2× 218 1.0× 89 0.6× 55 0.6× 132 1.5× 20 542
Jennifer Demieville United States 13 225 0.9× 117 0.5× 182 1.3× 143 1.5× 143 1.6× 16 523
Nobuko Serizawa Japan 7 205 0.8× 145 0.7× 182 1.3× 73 0.8× 173 2.0× 18 535
Xiaobo Cai China 11 179 0.7× 120 0.6× 173 1.2× 80 0.8× 51 0.6× 24 377
Maria Ruart Spain 7 258 1.0× 124 0.6× 189 1.3× 81 0.8× 51 0.6× 9 478
Alexander Weymann United States 10 149 0.6× 144 0.7× 152 1.1× 169 1.7× 45 0.5× 13 527
Koichiro Ohashi Japan 7 150 0.6× 142 0.7× 90 0.6× 60 0.6× 52 0.6× 23 394
Milica Stefanović Spain 9 121 0.5× 146 0.7× 96 0.7× 104 1.1× 147 1.7× 26 454
Marilena Fazi Italy 15 197 0.8× 230 1.1× 119 0.8× 228 2.4× 66 0.8× 36 703

Countries citing papers authored by Lukas Geisler

Since Specialization
Citations

This map shows the geographic impact of Lukas Geisler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lukas Geisler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lukas Geisler more than expected).

Fields of papers citing papers by Lukas Geisler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lukas Geisler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lukas Geisler. The network helps show where Lukas Geisler may publish in the future.

Co-authorship network of co-authors of Lukas Geisler

This figure shows the co-authorship network connecting the top 25 collaborators of Lukas Geisler. A scholar is included among the top collaborators of Lukas Geisler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lukas Geisler. Lukas Geisler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Kohlhepp, Marlene, Jana Hundertmark, Lukas Geisler, et al.. (2025). THU-037 Lycorine reduces liver fibrosis by inactivating hepatic stellate cells (HSCs) in mouse models of metabolic dysfunction-associated steatohepatitis (MASH). Journal of Hepatology. 82. S263–S264.
2.
Calcagno, David, Susanne Gaul, Avinash Toomu, et al.. (2022). NOD‐like receptor protein 3 activation causes spontaneous inflammation and fibrosis that mimics human NASH. Hepatology. 76(3). 727–741. 44 indexed citations
3.
Kaufmann, Benedikt, María Eugenia Inzaugarat, Theresa Maria Holtmann, et al.. (2022). Interleukin‐18 signaling promotes activation of hepatic stellate cells in mouse liver fibrosis. Hepatology. 77(6). 1968–1982. 54 indexed citations
4.
Geisler, Lukas, Joeri Lambrecht, Henning Jann, et al.. (2022). Inflammatory Cytokines Associated with Diagnosis, Tumor Grade and Prognosis in Patients with Neuroendocrine Tumors. Journal of Clinical Medicine. 11(20). 6191–6191. 5 indexed citations
5.
Geisler, Lukas, Raphael Mohr, Joeri Lambrecht, et al.. (2021). The Role of miRNA in the Pathophysiology of Neuroendocrine Tumors. International Journal of Molecular Sciences. 22(16). 8569–8569. 13 indexed citations
6.
Detjen, Katharina, Lukas Geisler, Pamela Riemer, et al.. (2021). Elevated Flt3L Predicts Long-Term Survival in Patients with High-Grade Gastroenteropancreatic Neuroendocrine Neoplasms. Cancers. 13(17). 4463–4463. 6 indexed citations
8.
Mohr, Raphael, Burcin Özdirik, Joeri Lambrecht, et al.. (2021). From Liver Cirrhosis to Cancer: The Role of Micro-RNAs in Hepatocarcinogenesis. International Journal of Molecular Sciences. 22(3). 1492–1492. 21 indexed citations
9.
Holtmann, Theresa Maria, María Eugenia Inzaugarat, Lukas Geisler, et al.. (2021). Bile Acids Activate NLRP3 Inflammasome, Promoting Murine Liver Inflammation or Fibrosis in a Cell Type-Specific Manner. Cells. 10(10). 2618–2618. 32 indexed citations
10.
Mohr, Raphael, Lukas Geisler, Alexander Wree, et al.. (2020). Serum levels of miR-223 but not miR-21 are decreased in patients with neuroendocrine tumors. PLoS ONE. 15(12). e0244504–e0244504. 4 indexed citations
11.
Özdirik, Burcin, Lukas Geisler, Raphael Mohr, et al.. (2020). Soluble Urokinase Plasminogen Activator Receptor (suPAR) Concentrations Are Elevated in Patients with Neuroendocrine Malignancies. Journal of Clinical Medicine. 9(6). 1647–1647. 10 indexed citations
12.
Özdirik, Burcin, Raphael Mohr, Lukas Geisler, et al.. (2020). Analysis of miR-29 Serum Levels in Patients with Neuroendocrine Tumors—Results from an Exploratory Study. Journal of Clinical Medicine. 9(9). 2881–2881. 8 indexed citations
13.
Gaul, Susanne, Lukas Geisler, Matthew D. McGeough, et al.. (2020). ASK1 inhibition reduces cell death and hepatic fibrosis in an Nlrp3 mutant liver injury model. JCI Insight. 5(2). 58 indexed citations
14.
Heymann, Felix, Christian Preisinger, Anjali A. Roeth, et al.. (2019). Polypropylene mesh implantation for hernia repair causes myeloid cell–driven persistent inflammation. JCI Insight. 4(2). 53 indexed citations
15.
Wree, Alexander, Lukas Geisler, & Frank Tacke. (2019). Mikrobiom & NASH – enge Komplizen in der Progression von Fettlebererkrankungen. Zeitschrift für Gastroenterologie. 57(7). 871–882. 5 indexed citations
16.
Inzaugarat, María Eugenia, Theresa Maria Holtmann, Mick Frissen, et al.. (2018). Different bile acids display distinct ability to trigger Nlrp3 inflammasome activation in a cell-dependent manner contributing to cholestatic liver injury and fibrosis. Journal of Hepatology. 68. S451–S451. 1 indexed citations
17.
Wree, Alexander, Matthew D. McGeough, María Eugenia Inzaugarat, et al.. (2017). NLRP3 inflammasome driven liver injury and fibrosis: Roles of IL‐17 and TNF in mice. Hepatology. 67(2). 736–749. 220 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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