Lotte Leick

2.3k total citations · 1 hit paper
15 papers, 1.9k citations indexed

About

Lotte Leick is a scholar working on Physiology, Molecular Biology and Rehabilitation. According to data from OpenAlex, Lotte Leick has authored 15 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Physiology, 7 papers in Molecular Biology and 5 papers in Rehabilitation. Recurrent topics in Lotte Leick's work include Adipose Tissue and Metabolism (12 papers), Exercise and Physiological Responses (5 papers) and Adipokines, Inflammation, and Metabolic Diseases (4 papers). Lotte Leick is often cited by papers focused on Adipose Tissue and Metabolism (12 papers), Exercise and Physiological Responses (5 papers) and Adipokines, Inflammation, and Metabolic Diseases (4 papers). Lotte Leick collaborates with scholars based in Denmark, Canada and United Kingdom. Lotte Leick's co-authors include Henriette Pilegaard, Bente Klarlund Pedersen, Emma C. Hart, Niels H. Secher, Peter Rasmussen, Helle Adser, Martin V. Pedersen, Patrice Brassard, Jørgen F. P. Wojtaszewski and Juan Hidalgo and has published in prestigious journals such as PLoS ONE, The Journal of Physiology and Journal of Applied Physiology.

In The Last Decade

Lotte Leick

15 papers receiving 1.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Evidence for a release of brain‐derived neurotrophic factor from the brain during exercise

2009 734 citations Peter Rasmussen, Patrice Brassard et al. Experimental Physiology profile →

Peers

Countries citing papers authored by Lotte Leick

Since Specialization

Citations

This map shows the geographic impact of Lotte Leick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lotte Leick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lotte Leick more than expected).

Fields of papers citing papers by Lotte Leick

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lotte Leick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lotte Leick. The network helps show where Lotte Leick may publish in the future.

Co-authorship network of co-authors of Lotte Leick

This figure shows the co-authorship network connecting the top 25 collaborators of Lotte Leick. A scholar is included among the top collaborators of Lotte Leick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lotte Leick. Lotte Leick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

#	Work	Indexed citations
1	Effect of IL-6 on the insulin sensitivity in patients with type 2 diabetes 2014 ·American Journal of Physiology-Endocrinology and Metabolism ·Nina M. Harder‐Lauridsen, Rikke Krogh‐Madsen, Jens J. Holst, Peter Plomgaard, Lotte Leick, Bente Klarlund Pedersen, Christian P. Fischer	43
2	AMP‐activated protein kinase regulates nicotinamide phosphoribosyl transferase expression in skeletal muscle 2013 ·The Journal of Physiology ·Josef Brandauer, Sara G. Vienberg, (unknown), Stine Ringholm, Steve Risis, (unknown), Jonas M. Kristensen, Christian Frøsig, Lotte Leick, Joachim Fentz, Sebastian B. Jørgensen, Bente Kiens, Jørgen F. P. Wojtaszewski, Erik A. Richter, Juleen R. Zierath, Laurie J. Goodyear, Henriette Pilegaard, Jonas T. Treebak	80
3	PGC-1α Is Required for Exercise- and Exercise Training-Induced UCP1 Up-Regulation in Mouse White Adipose Tissue 2013 ·PLoS ONE ·Stine Ringholm, Jakob G. Knudsen, Lotte Leick, (unknown), (unknown), Henriette Pilegaard	61
4	Role of PGC-1α in exercise and fasting-induced adaptations in mouse liver 2011 ·American Journal of Physiology-Regulatory, Integrative and Comparative Physiology ·(unknown), Stine Ringholm, Lotte Leick, Rasmus Sjørup Biensø, Kristian Kiilerich, (unknown), (unknown), Jørgen F. P. Wojtaszewski, Juan Hidalgo, Per Amstrup Pedersen, Henriette Pilegaard	58
5	Relative Workload Determines Exercise-Induced Increases in PGC-1α mRNA 2010 ·Medicine & Science in Sports & Exercise ·Nikolai Baastrup Nordsborg, Carsten Lundby, Lotte Leick, Henriette Pilegaard	73
6	PGC-1α is required for training-induced prevention of age-associated decline in mitochondrial enzymes in mouse skeletal muscle 2010 ·Experimental Gerontology ·Lotte Leick, (unknown), (unknown), Henriette Pilegaard	96
7	PGC-1α is required for AICAR-induced expression of GLUT4 and mitochondrial proteins in mouse skeletal muscle 2010 ·American Journal of Physiology-Endocrinology and Metabolism ·Lotte Leick, Joachim Fentz, Rasmus Sjørup Biensø, Jakob G. Knudsen, Jacob Jeppesen, Bente Kiens, Jørgen F. P. Wojtaszewski, Henriette Pilegaard	85
8	PGC-1α mediates exercise-induced skeletal muscle VEGF expression in mice 2009 ·American Journal of Physiology-Endocrinology and Metabolism ·Lotte Leick, Ylva Hellsten, Joachim Fentz, (unknown), Jørgen F. P. Wojtaszewski, Juan Hidalgo, Henriette Pilegaard	102
9	The role of PGC-1α on mitochondrial function and apoptotic susceptibility in muscle 2009 ·American Journal of Physiology-Cell Physiology ·Peter J. Adhihetty, Giulia Uguccioni, Lotte Leick, Juan Hidalgo, Henriette Pilegaard, David A. Hood	138
10	Evidence for a release of brain‐derived neurotrophic factor from the brain during exercise breakdown → 2009 ·Experimental Physiology ·Peter Rasmussen, Patrice Brassard, Helle Adser, Martin V. Pedersen, Lotte Leick, Emma C. Hart, Niels H. Secher, Bente Klarlund Pedersen, Henriette Pilegaard	734
11	Endurance exercise induces mRNA expression of oxidative enzymes in human skeletal muscle late in recovery 2009 ·Scandinavian Journal of Medicine and Science in Sports ·Lotte Leick, Peter Plomgaard, Lars Grønlykke, (unknown), Jørgen F. P. Wojtaszewski, Henriette Pilegaard	47
12	PGC-1α is not mandatory for exercise- and training-induced adaptive gene responses in mouse skeletal muscle 2007 ·American Journal of Physiology-Endocrinology and Metabolism ·Lotte Leick, Jørgen F. P. Wojtaszewski, (unknown), Kristian Kiilerich, Gemma Comes, Ylva Hellsten, Juan Hidalgo, Henriette Pilegaard	201
13	Adipose Tissue Interleukin‐18 mRNA and Plasma Interleukin‐18: Effect of Obesity and Exercise 2007 ·Obesity ·Lotte Leick, Birgitte Lindegaard, Dorthe Stensvold, Peter Plomgaard, Bengt Saltin, Henriette Pilegaard	57
14	The mRNA expression profile of metabolic genes relative to MHC isoform pattern in human skeletal muscles 2006 ·Journal of Applied Physiology ·Peter Plomgaard, Milena Penkowa, Lotte Leick, Bente Klarlund Pedersen, Bengt Saltin, Henriette Pilegaard	36
15	Control of gene expression and mitochondrial biogenesis in the muscular adaptation to endurance exercise 2006 ·Essays in Biochemistry ·Bertrand Joseph, Henriette Pilegaard, (unknown), Lotte Leick, David A. Hood	88

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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