Lotte Leick

2.3k total citations · 1 hit paper
15 papers, 1.9k citations indexed

About

Lotte Leick is a scholar working on Physiology, Molecular Biology and Rehabilitation. According to data from OpenAlex, Lotte Leick has authored 15 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Physiology, 7 papers in Molecular Biology and 5 papers in Rehabilitation. Recurrent topics in Lotte Leick's work include Adipose Tissue and Metabolism (12 papers), Exercise and Physiological Responses (5 papers) and Adipokines, Inflammation, and Metabolic Diseases (4 papers). Lotte Leick is often cited by papers focused on Adipose Tissue and Metabolism (12 papers), Exercise and Physiological Responses (5 papers) and Adipokines, Inflammation, and Metabolic Diseases (4 papers). Lotte Leick collaborates with scholars based in Denmark, Canada and United Kingdom. Lotte Leick's co-authors include Henriette Pilegaard, Bente Klarlund Pedersen, Emma C. Hart, Niels H. Secher, Peter Rasmussen, Helle Adser, Martin V. Pedersen, Patrice Brassard, Jørgen F. P. Wojtaszewski and Juan Hidalgo and has published in prestigious journals such as PLoS ONE, The Journal of Physiology and Journal of Applied Physiology.

In The Last Decade

Lotte Leick

15 papers receiving 1.9k citations

Hit Papers

Evidence for a release of brain‐derived neurotrophic fact... 2009 2026 2014 2020 2009 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Lotte Leick Denmark 15 1.1k 684 330 300 277 15 1.9k
Masataka Suwa Japan 17 648 0.6× 470 0.7× 86 0.3× 185 0.6× 165 0.6× 36 1.2k
Christina Yfanti Greece 16 1.6k 1.5× 878 1.3× 839 2.5× 202 0.7× 528 1.9× 26 2.8k
Chauying J. Jen Taiwan 29 699 0.6× 414 0.6× 224 0.7× 430 1.4× 159 0.6× 64 2.4k
Roger P. Farrar United States 32 918 0.8× 1.3k 1.9× 408 1.2× 579 1.9× 150 0.5× 73 3.1k
Sama F. Sleiman United States 17 634 0.6× 785 1.1× 76 0.2× 321 1.1× 117 0.4× 27 1.8k
Harveen Dhillon United States 18 1.5k 1.4× 651 1.0× 98 0.3× 302 1.0× 546 2.0× 22 3.1k
Peter Rasmussen Denmark 13 589 0.5× 276 0.4× 155 0.5× 504 1.7× 106 0.4× 15 1.8k
Vicente Martínez-Redondo Sweden 15 615 0.6× 675 1.0× 191 0.6× 135 0.5× 144 0.5× 16 1.6k
Helle Adser Denmark 7 474 0.4× 142 0.2× 196 0.6× 403 1.3× 108 0.4× 7 1.3k
Vitaly Ryu United States 25 732 0.7× 218 0.3× 88 0.3× 213 0.7× 232 0.8× 54 1.8k

Countries citing papers authored by Lotte Leick

Since Specialization
Citations

This map shows the geographic impact of Lotte Leick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lotte Leick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lotte Leick more than expected).

Fields of papers citing papers by Lotte Leick

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lotte Leick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lotte Leick. The network helps show where Lotte Leick may publish in the future.

Co-authorship network of co-authors of Lotte Leick

This figure shows the co-authorship network connecting the top 25 collaborators of Lotte Leick. A scholar is included among the top collaborators of Lotte Leick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lotte Leick. Lotte Leick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Harder‐Lauridsen, Nina M., Rikke Krogh‐Madsen, Jens J. Holst, et al.. (2014). Effect of IL-6 on the insulin sensitivity in patients with type 2 diabetes. American Journal of Physiology-Endocrinology and Metabolism. 306(7). E769–E778. 43 indexed citations
2.
Brandauer, Josef, Sara G. Vienberg, Stine Ringholm, et al.. (2013). AMP‐activated protein kinase regulates nicotinamide phosphoribosyl transferase expression in skeletal muscle. The Journal of Physiology. 591(20). 5207–5220. 80 indexed citations
3.
Ringholm, Stine, et al.. (2013). PGC-1α Is Required for Exercise- and Exercise Training-Induced UCP1 Up-Regulation in Mouse White Adipose Tissue. PLoS ONE. 8(5). e64123–e64123. 61 indexed citations
4.
Ringholm, Stine, Lotte Leick, Rasmus Sjørup Biensø, et al.. (2011). Role of PGC-1α in exercise and fasting-induced adaptations in mouse liver. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 301(5). R1501–R1509. 58 indexed citations
5.
Nordsborg, Nikolai Baastrup, Carsten Lundby, Lotte Leick, & Henriette Pilegaard. (2010). Relative Workload Determines Exercise-Induced Increases in PGC-1α mRNA. Medicine & Science in Sports & Exercise. 42(8). 1477–1484. 73 indexed citations
6.
Leick, Lotte, et al.. (2010). PGC-1α is required for training-induced prevention of age-associated decline in mitochondrial enzymes in mouse skeletal muscle. Experimental Gerontology. 45(5). 336–342. 96 indexed citations
7.
Leick, Lotte, Joachim Fentz, Rasmus Sjørup Biensø, et al.. (2010). PGC-1α is required for AICAR-induced expression of GLUT4 and mitochondrial proteins in mouse skeletal muscle. American Journal of Physiology-Endocrinology and Metabolism. 299(3). E456–E465. 85 indexed citations
8.
Leick, Lotte, Ylva Hellsten, Joachim Fentz, et al.. (2009). PGC-1α mediates exercise-induced skeletal muscle VEGF expression in mice. American Journal of Physiology-Endocrinology and Metabolism. 297(1). E92–E103. 102 indexed citations
9.
Adhihetty, Peter J., Giulia Uguccioni, Lotte Leick, et al.. (2009). The role of PGC-1α on mitochondrial function and apoptotic susceptibility in muscle. American Journal of Physiology-Cell Physiology. 297(1). C217–C225. 138 indexed citations
10.
Rasmussen, Peter, Patrice Brassard, Helle Adser, et al.. (2009). Evidence for a release of brain‐derived neurotrophic factor from the brain during exercise. Experimental Physiology. 94(10). 1062–1069. 734 indexed citations breakdown →
11.
Leick, Lotte, et al.. (2009). Endurance exercise induces mRNA expression of oxidative enzymes in human skeletal muscle late in recovery. Scandinavian Journal of Medicine and Science in Sports. 20(4). 593–599. 47 indexed citations
12.
Leick, Lotte, Jørgen F. P. Wojtaszewski, Kristian Kiilerich, et al.. (2007). PGC-1α is not mandatory for exercise- and training-induced adaptive gene responses in mouse skeletal muscle. American Journal of Physiology-Endocrinology and Metabolism. 294(2). E463–E474. 201 indexed citations
13.
Leick, Lotte, Birgitte Lindegaard, Dorthe Stensvold, et al.. (2007). Adipose Tissue Interleukin‐18 mRNA and Plasma Interleukin‐18: Effect of Obesity and Exercise. Obesity. 15(2). 356–363. 57 indexed citations
14.
Plomgaard, Peter, Milena Penkowa, Lotte Leick, et al.. (2006). The mRNA expression profile of metabolic genes relative to MHC isoform pattern in human skeletal muscles. Journal of Applied Physiology. 101(3). 817–825. 36 indexed citations
15.
Joseph, Bertrand, et al.. (2006). Control of gene expression and mitochondrial biogenesis in the muscular adaptation to endurance exercise. Essays in Biochemistry. 42. 13–29. 88 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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