Kristopher Nielsen
About
In The Last Decade
Kristopher Nielsen
25 papers receiving 402 citations
Peers
Comparison fields: 5 of 74
- Genetics 234
- Molecular Biology 104
- Philosophy 87
- Cognitive Neuroscience 82
- Rheumatology 47
Countries citing papers authored by Kristopher Nielsen
This map shows the geographic impact of Kristopher Nielsen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kristopher Nielsen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kristopher Nielsen more than expected).
Fields of papers citing papers by Kristopher Nielsen
This network shows the impact of papers produced by Kristopher Nielsen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kristopher Nielsen. The network helps show where Kristopher Nielsen may publish in the future.
Co-authorship network of co-authors of Kristopher Nielsen
This figure shows the co-authorship network connecting the top 25 collaborators of Kristopher Nielsen. A scholar is included among the top collaborators of Kristopher Nielsen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kristopher Nielsen. Kristopher Nielsen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 25 | |
| 4 | 7 | |
| 5 | 7 | |
| 6 | From Engel to Enactivism: Contextualizing the Biopsychosocial Model | 3 |
| 7 | 2 | |
| 8 | 13 | |
| 9 | 8 | |
| 10 | 22 | |
| 11 | Simple mutations in the coding region of three acyl-CoA dehydrogenase genes may lead to skipping of the constitutive exon due to disruption of potential splice enhancer sequences or mRNA secondary structures | 1 |
| 12 | 2 | |
| 13 | 53 | |
| 14 | 39 | |
| 15 | 15 | |
| 16 | 60 | |
| 17 | 17 | |
| 18 | A chromosomal survey of an institution for the mentally retarded. Study of 476 karyotypes with banding techniques and clinical assessment of patients with chromosome anomalies. | 18 |
| 19 | 8 | |
| 20 | 30 |
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.