Kota Saito

2.7k total citations · 1 hit paper
34 papers, 1.8k citations indexed

About

Kota Saito is a scholar working on Cell Biology, Molecular Biology and Epidemiology. According to data from OpenAlex, Kota Saito has authored 34 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Cell Biology, 21 papers in Molecular Biology and 4 papers in Epidemiology. Recurrent topics in Kota Saito's work include Cellular transport and secretion (21 papers), Endoplasmic Reticulum Stress and Disease (13 papers) and Retinal Development and Disorders (5 papers). Kota Saito is often cited by papers focused on Cellular transport and secretion (21 papers), Endoplasmic Reticulum Stress and Disease (13 papers) and Retinal Development and Disorders (5 papers). Kota Saito collaborates with scholars based in Japan, United States and Spain. Kota Saito's co-authors include Toshiaki Katada, Miharu Maeda, Kenji Kontani, Vivek Malhotra, Randy Schekman, Hiroaki Kajiho, Hiroshi Kurosu, Sheng‐hong Chen, David T. Woodley and Mei Chen and has published in prestigious journals such as Nature, Cell and Nucleic Acids Research.

In The Last Decade

Kota Saito

30 papers receiving 1.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

TANGO1 Facilitates Cargo Loading at Endoplasmic Reticulum Exit Sites

2009 282 citations Kota Saito, Mei Chen et al. Cell profile →

Peers

Countries citing papers authored by Kota Saito

Since Specialization

Citations

This map shows the geographic impact of Kota Saito's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kota Saito with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kota Saito more than expected).

Fields of papers citing papers by Kota Saito

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kota Saito. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kota Saito. The network helps show where Kota Saito may publish in the future.

Co-authorship network of co-authors of Kota Saito

This figure shows the co-authorship network connecting the top 25 collaborators of Kota Saito. A scholar is included among the top collaborators of Kota Saito based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kota Saito. Kota Saito is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Various methods to detect small GTPase activation: from radioisotope-based methods to the Small GTPase ActIvitY ANalysing (SAIYAN) system 2025 ·The Journal of Biochemistry ·Miharu Maeda, Kota Saito	0
2	Towards a unified framework for the function of endoplasmic reticulum exit sites 2025 ·Nature Reviews Molecular Cell Biology ·Hesso Farhan, Ishier Raote, Felix Campelo, Liang Ge, Koret Hirschberg, Alison Forrester, Giulia Zanetti, Jennifer Lippincott‐Schwartz, (unknown), Franck Perez, Kota Saito, Vivek Malhotra	0
3	Disease‐Associated Factors at the Endoplasmic Reticulum–Golgi Interface 2025 ·Traffic ·Miharu Maeda, Masashi Arakawa, Kota Saito	0
4	Small GTPase ActIvitY ANalyzing (SAIYAN) system: A method to detect GTPase activation in living cells 2024 ·The Journal of Cell Biology ·Miharu Maeda, Masashi Arakawa, (unknown), Kota Saito	3
5	Mitotic ER Exit Site Disassembly and Reassembly Are Regulated by the Phosphorylation Status of TANGO1 2020 ·Developmental Cell ·Miharu Maeda, (unknown), Kota Saito	21
6	COPII proteins exhibit distinct subdomains within each ER exit site for executing their functions 2019 ·Scientific Reports ·Miharu Maeda, Kazuo Kurokawa, Toshiaki Katada, Akihiko Nakano, Kota Saito	8
7	The binding of TBK1 to STING requires exocytic membrane traffic from the ER 2018 ·Biochemical and Biophysical Research Communications ·Emari Ogawa, Kojiro Mukai, Kota Saito, Hiroyuki Arai, Tomohiko Taguchi	70
8	Remodeling of ER ‐exit sites initiates a membrane supply pathway for autophagosome biogenesis 2017 ·EMBO Reports ·Liang Ge, Min Zhang, Samuel J. Kenny, Dawei Liu, Miharu Maeda, Kota Saito, (unknown), Ke Xu, Randy Schekman	126
9	CREB3L2-mediated expression of Sec23A/Sec24D is involved in hepatic stellate cell activation through ER-Golgi transport 2017 ·Scientific Reports ·(unknown), Shinichi Fukushima, Kentaro Shinohara, Toshiaki Katada, Kota Saito	22
10	Distinct isoform-specific complexes of TANGO1 cooperatively facilitate collagen secretion from the endoplasmic reticulum 2016 ·Molecular Biology of the Cell ·Miharu Maeda, Kota Saito, Toshiaki Katada	41
11	Dual function of cTAGE5 in collagen export from the endoplasmic reticulum 2016 ·Molecular Biology of the Cell ·(unknown), Miharu Maeda, Kota Saito, Toshiaki Katada	30
12	Mechanisms for exporting large-sized cargoes from the endoplasmic reticulum 2015 ·Cellular and Molecular Life Sciences ·Kota Saito, Toshiaki Katada	37
13	Arl8/ARL-8 functions in apoptotic cell removal by mediating phagolysosome formation inCaenorhabditis elegans 2013 ·Molecular Biology of the Cell ·(unknown), (unknown), (unknown), Keisuke Hashimoto, (unknown), Kota Saito, Masamitsu Fukuyama, Keiko Gengyo‐Ando, Shohei Mitani, Toshiaki Katada, Kenji Kontani	40
14	cTAGE5 mediates collagen secretion through interaction with TANGO1 at endoplasmic reticulum exit sites 2011 ·Molecular Biology of the Cell ·Kota Saito, (unknown), Yūki Ichikawa, Patrik Erlmann, Kenji Kontani, Vivek Malhotra, Toshiaki Katada	125
15	RhoA activation participates in rearrangement of processing bodies and release of nucleated AU-rich mRNAs 2011 ·Nucleic Acids Research ·Shinya Takahashi, (unknown), (unknown), Hiroaki Kajiho, Kota Saito, Kenji Kontani, Hiroshi Nishina, Toshiaki Katada	13
16	The LIM protein Ajuba is required for ciliogenesis and left–right axis determination in medaka 2010 ·Biochemical and Biophysical Research Communications ·Yoko Nagai, Yoichi Asaoka, (unknown), Kota Saito, Haruka Momose, Hiroshi Mitani, Makoto Furutani‐Seiki, Toshiaki Katada, Hiroshi Nishina	9
17	TANGO1 Facilitates Cargo Loading at Endoplasmic Reticulum Exit Sites breakdown → 2009 ·Cell ·Kota Saito, Mei Chen, (unknown), Sheng‐hong Chen, Huilin Zhou, David T. Woodley, (unknown), Randy Schekman, Vivek Malhotra	282
18	Novel Role of the Small GTPase Rheb: Its Implication in Endocytic Pathway Independent of the Activation of Mammalian Target of Rapamycin 2005 ·The Journal of Biochemistry ·Kota Saito	103
19	A Novel Binding Protein Composed of Homophilic Tetramer Exhibits Unique Properties for the Small GTPase Rab5 2002 ·Journal of Biological Chemistry ·Kota Saito, Jun Murai, Hiroaki Kajiho, Kenji Kontani, Hiroshi Kurosu, Toshiaki Katada	81
20	Di-Ras, a Distinct Subgroup of Ras Family GTPases with Unique Biochemical Properties 2002 ·Journal of Biological Chemistry ·Kenji Kontani, Minoru Tada, Tomohiro Ogawa, (unknown), Kota Saito, Yasuhiro Araki, Toshiaki Katada	55

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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