Klaus‐Dieter Palitzsch

469 citations
18 papers · 353 indexed · h-index 9

Klaus‐Dieter Palitzsch

16 papers receiving 339 citations

Peers

Klaus‐Dieter Palitzsch
Comparison fields: 5 of 75
  • Hepatology 100
  • Epidemiology 126
  • Cardiology and Cardiovascular Medicine 71
  • Equine 5
  • Endocrinology, Diabetes and Metabolism 44
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Countries citing papers authored by Klaus‐Dieter Palitzsch

Since Specialization
Citations

This map shows the geographic impact of Klaus‐Dieter Palitzsch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Klaus‐Dieter Palitzsch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Klaus‐Dieter Palitzsch more than expected).

Fields of papers citing papers by Klaus‐Dieter Palitzsch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Klaus‐Dieter Palitzsch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Klaus‐Dieter Palitzsch. The network helps show where Klaus‐Dieter Palitzsch may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Klaus‐Dieter Palitzsch, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Klaus‐Dieter Palitzsch Line = papers co-authored together Klaus‐Dieter Palitzsch links everyone, so they are left out of the graph.

All Works

18 of 18 papers shown
#Work
1 20150
2 20122
3 20060
4
Correlation between iron status and genetic hemochromatosis (codon C282Y) in a large German population.
20044
5 200220
6 20017
7 200129
8 20014
9 200158
10 20018
11 199929
12 199945
13 199927
14 199926
15 19973
16 199451
17 199439
18
Metronidazole Inhibits Leukocyte-Endothelial in Rat Mesenteric Venules Cell Adhesion
19941

About Klaus‐Dieter Palitzsch

Klaus‐Dieter Palitzsch is a scholar working on Hepatology, Endocrinology, Diabetes and Metabolism, Cardiology and Cardiovascular Medicine, Pharmaceutical Science and Nephrology, having authored 18 papers that have together received 353 indexed citations. Recurring topics across this work include Heart Rate Variability and Autonomic Control (4 papers), Cardiovascular Syncope and Autonomic Disorders (3 papers), Diabetes and associated disorders (3 papers), Diabetes Treatment and Management (3 papers), Hepatitis C virus research (3 papers), Metabolism, Diabetes, and Cancer (3 papers), Liver Disease Diagnosis and Treatment (2 papers) and Adipose Tissue and Metabolism (2 papers). The work is most often cited by research in Hepatology (100 citations), Epidemiology (126 citations), Cardiology and Cardiovascular Medicine (71 citations), Equine (5 citations) and Endocrinology, Diabetes and Metabolism (44 citations). Klaus‐Dieter Palitzsch has collaborated with scholars based in Germany and United States. Frequent co-authors include Jürgen Schölmerich, Holger Arndt, Klaus Schlottmann, Axel Holstege, Wolfgang Jilg, E Frick, Matthew B. Grisham, D. Neil Granger, Cornelius Keyl and Karin Hochmuth. Their work appears in journals such as Anesthesia & Analgesia, Journal of Medical Virology, Diabetes Care, European Journal of Gastroenterology & Hepatology and Journal of Parenteral and Enteral Nutrition.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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