Kenneth Takeda
- Molecular Biology
- Cellular and Molecular Neuroscience top 5%
- Cell Biology top 5%
- Immunology and Allergy top 2%
- Immunology
- Co-authors
- Philippe RondéMireille GaireGrégory GiannoneJacques HaiechYves MélyBernard BucherH. StoeckelJean‐Luc Galzi
- Topics
- Ion channel regulation and function (10 papers)Receptor Mechanisms and Signaling (9 papers)Cell Adhesion Molecules Research (8 papers)
- Journals
- Journal of Biological ChemistryThe Journal of Physical Chemistry BThe Journal of Physiology
- Partner nations
- FranceUnited StatesSwitzerland
In The Last Decade
Kenneth Takeda
30 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 96
- Molecular Biology 660
- Cellular and Molecular Neuroscience 324
- Cell Biology 282
- Immunology and Allergy 230
- Immunology 105
Countries citing papers authored by Kenneth Takeda
This map shows the geographic impact of Kenneth Takeda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kenneth Takeda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kenneth Takeda more than expected).
Fields of papers citing papers by Kenneth Takeda
This network shows the impact of papers produced by Kenneth Takeda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kenneth Takeda. The network helps show where Kenneth Takeda may publish in the future.
Co-authorship network of co-authors of Kenneth Takeda
This figure shows the co-authorship network connecting the top 25 collaborators of Kenneth Takeda. A scholar is included among the top collaborators of Kenneth Takeda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kenneth Takeda. Kenneth Takeda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 111 | |
| 3 | 52 | |
| 4 | 29 | |
| 5 | 63 | |
| 6 | 124 | |
| 7 | 12 | |
| 8 | 88 | |
| 9 | 104 | |
| 10 | 20 | |
| 11 | 44 | |
| 12 | 11 | |
| 13 | 22 | |
| 14 | 35 | |
| 15 | 9 | |
| 16 | 6 | |
| 17 | 9 | |
| 18 | 24 | |
| 19 | 45 | |
| 20 | 12 |
About Kenneth Takeda
Kenneth Takeda is a scholar working on Immunology and Allergy, Cellular and Molecular Neuroscience and Cell Biology, having authored 30 papers that have together received 1.1k indexed citations. Recurring topics across this work include Ion channel regulation and function (10 papers), Receptor Mechanisms and Signaling (9 papers) and Cell Adhesion Molecules Research (8 papers). The work is most often cited by research in Immunology and Allergy (230 citations), Cellular and Molecular Neuroscience (324 citations) and Cell Biology (282 citations). Kenneth Takeda has collaborated with scholars based in France, United States and Switzerland. Frequent co-authors include Philippe Rondé, Mireille Gaire, Grégory Giannone, Jacques Haiech, Yves Mély, Bernard Bucher, H. Stoeckel, Jean‐Luc Galzi, Thérèse B. Deramaudt and Jean‐Gaël Barbara. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Physical Chemistry B and The Journal of Physiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.