Peter J. Lockyer

1.8k total citations
22 papers, 1.5k citations indexed

About

Peter J. Lockyer is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Peter J. Lockyer has authored 22 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 12 papers in Cell Biology and 5 papers in Cellular and Molecular Neuroscience. Recurrent topics in Peter J. Lockyer's work include Protein Kinase Regulation and GTPase Signaling (14 papers), Cellular transport and secretion (11 papers) and Cell death mechanisms and regulation (6 papers). Peter J. Lockyer is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (14 papers), Cellular transport and secretion (11 papers) and Cell death mechanisms and regulation (6 papers). Peter J. Lockyer collaborates with scholars based in United Kingdom, United States and Belgium. Peter J. Lockyer's co-authors include Peter J. Cullen, Sabine Kupzig, Simon Walker, Mark R. Philips, Trever G. Bivona, Simon J. Cook, Ian M. Ahearn, Ignacío Pérez de Castro, Theresa M. Grana and Vi K. Chiu and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Nature Reviews Molecular Cell Biology.

In The Last Decade

Peter J. Lockyer

22 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Peter J. Lockyer United Kingdom 17 1.1k 440 194 190 166 22 1.5k
Silvia Carrasco Spain 11 1.0k 0.9× 353 0.8× 184 0.9× 191 1.0× 99 0.6× 12 1.5k
Sascha E. Dho Canada 15 1.4k 1.3× 458 1.0× 198 1.0× 95 0.5× 189 1.1× 21 1.8k
Junhui Bian United States 13 1.3k 1.1× 364 0.8× 202 1.0× 110 0.6× 264 1.6× 16 1.5k
Priam Villalonga Spain 19 1.2k 1.0× 337 0.8× 172 0.9× 103 0.5× 359 2.2× 28 1.6k
Miranda van Triest Netherlands 11 1.1k 1.0× 283 0.6× 233 1.2× 118 0.6× 120 0.7× 11 1.5k
Stephen Gutowski United States 16 1.7k 1.5× 738 1.7× 245 1.3× 174 0.9× 131 0.8× 45 2.1k
Maree C. Faux Australia 22 1.8k 1.6× 376 0.9× 358 1.8× 96 0.5× 292 1.8× 42 2.2k
Ron Kriz United States 17 1.1k 1.0× 271 0.6× 162 0.8× 191 1.0× 170 1.0× 23 1.5k
Tammy M. Seasholtz United States 17 1.4k 1.2× 363 0.8× 206 1.1× 130 0.7× 114 0.7× 25 1.8k
Mélanie Robitaille Canada 22 1.3k 1.2× 352 0.8× 396 2.0× 57 0.3× 164 1.0× 44 1.6k

Countries citing papers authored by Peter J. Lockyer

Since Specialization
Citations

This map shows the geographic impact of Peter J. Lockyer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter J. Lockyer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter J. Lockyer more than expected).

Fields of papers citing papers by Peter J. Lockyer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter J. Lockyer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter J. Lockyer. The network helps show where Peter J. Lockyer may publish in the future.

Co-authorship network of co-authors of Peter J. Lockyer

This figure shows the co-authorship network connecting the top 25 collaborators of Peter J. Lockyer. A scholar is included among the top collaborators of Peter J. Lockyer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter J. Lockyer. Peter J. Lockyer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Dai, Yanfeng, et al.. (2011). Ca2+-dependent Monomer and Dimer Formation Switches CAPRI Protein between Ras GTPase-activating Protein (GAP) and RapGAP Activities. Journal of Biological Chemistry. 286(22). 19905–19916. 21 indexed citations
2.
Kupzig, Sabine, Simon Walker, Qing Liu, et al.. (2006). GAP1 Family Members Constitute Bifunctional Ras and Rap GTPase-activating Proteins. Journal of Biological Chemistry. 281(15). 9891–9900. 67 indexed citations
3.
Cook, Simon J. & Peter J. Lockyer. (2005). Recent advances in Ca2+-dependent Ras regulation and cell proliferation. Cell Calcium. 39(2). 101–112. 67 indexed citations
4.
Liu, Qing, Simon Walker, Dingcheng Gao, et al.. (2005). CAPRI and RASAL impose different modes of information processing on Ras due to contrasting temporal filtering of Ca2+. The Journal of Cell Biology. 170(2). 183–190. 57 indexed citations
5.
Walker, Simon, Sabine Kupzig, Takashi Tsuboi, et al.. (2004). Identification of a Ras GTPase‐activating protein regulated by receptor‐mediated Ca2+ oscillations. The EMBO Journal. 23(8). 1749–1760. 67 indexed citations
6.
Walker, Simon, Peter J. Lockyer, & Peter J. Cullen. (2003). The Ras binary switch: an ideal processor for decoding complex Ca2+ signals?. Biochemical Society Transactions. 31(5). 966–969. 23 indexed citations
7.
Walker, Simon, Peter J. Cullen, James A. Taylor, & Peter J. Lockyer. (2003). Control of Ras cycling by Ca2+. FEBS Letters. 546(1). 6–10. 38 indexed citations
8.
Bivona, Trever G., Ignacío Pérez de Castro, Ian M. Ahearn, et al.. (2003). Phospholipase Cγ activates Ras on the Golgi apparatus by means of RasGRP1. Nature. 424(6949). 694–698. 357 indexed citations
9.
Cullen, Peter J. & Peter J. Lockyer. (2002). Integration of calcium and RAS signalling. Nature Reviews Molecular Cell Biology. 3(5). 339–348. 313 indexed citations
10.
Walker, Simon, et al.. (2002). Analyzing the Role of the Putative Inositol 1,3,4,5-Tetrakisphosphate Receptor GAP1IP4BP in Intracellular Ca2+ Homeostasis. Journal of Biological Chemistry. 277(50). 48779–48785. 13 indexed citations
11.
Lockyer, Peter J., Sabine Kupzig, & Peter J. Cullen. (2001). CAPRI regulates Ca2+-dependent inactivation of the Ras-MAPK pathway. Current Biology. 11(12). 981–986. 88 indexed citations
12.
Lockyer, Peter J., et al.. (2001). Curcumin, a Molecule That Inhibits the Ca2+-ATPase of Sarcoplasmic Reticulum but Increases the Rate of Accumulation of Ca2+. Journal of Biological Chemistry. 276(50). 46905–46911. 81 indexed citations
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15.
Cozier, Gyles E., Peter J. Lockyer, Sabine Kupzig, et al.. (2000). GAP1IP4BP Contains a Novel Group I Pleckstrin Homology Domain That Directs Constitutive Plasma Membrane Association. Journal of Biological Chemistry. 275(36). 28261–28268. 71 indexed citations
16.
Lockyer, Peter J., Stefan Wennström, Sabine Kupzig, et al.. (1999). Identification of the Ras GTPase-activating protein GAP1m as a phosphatidylinositol-3,4,5-trisphosphate-binding protein in vivo. Current Biology. 9(5). 265–269. 64 indexed citations
17.
Lockyer, Peter J., et al.. (1999). Tissue-Specific Expression and Endogenous Subcellular Distribution of the Inositol 1,3,4,5-Tetrakisphosphate-Binding Proteins GAP1IP4BPand GAP1m. Biochemical and Biophysical Research Communications. 255(2). 421–426. 12 indexed citations
18.
Bottomley, Joanna, et al.. (1998). Structural and Functional Analysis of the Putative Inositol 1,3,4,5-Tetrakisphosphate Receptors GAP1IP4BPand GAP1m. Biochemical and Biophysical Research Communications. 250(1). 143–149. 25 indexed citations
19.
Lockyer, Peter J., et al.. (1998). Cloning and expression of an insect Ca2+-ATPase from Heliothis virescens. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1369(1). 14–18. 7 indexed citations
20.
Lockyer, Peter J., Joanna Bottomley, Venkateswarlu Kanamarlapudi, et al.. (1997). Distinct subcellular localisations of the putative inositol 1,3,4,5-tetrakisphosphate receptors GAP1 IP4BP and GAP1 m result from the GAP1 IP4BP PH domain directing plasma membrane targeting. Current Biology. 7(12). 1007–1010. 78 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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