Kazuo Terai

612 total citations
11 papers, 525 citations indexed

About

Kazuo Terai is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Surgery. According to data from OpenAlex, Kazuo Terai has authored 11 papers receiving a total of 525 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cardiology and Cardiovascular Medicine, 4 papers in Molecular Biology and 3 papers in Surgery. Recurrent topics in Kazuo Terai's work include Cardiac Fibrosis and Remodeling (3 papers), Cardiac Ischemia and Reperfusion (2 papers) and Cardiac Imaging and Diagnostics (2 papers). Kazuo Terai is often cited by papers focused on Cardiac Fibrosis and Remodeling (3 papers), Cardiac Ischemia and Reperfusion (2 papers) and Cardiac Imaging and Diagnostics (2 papers). Kazuo Terai collaborates with scholars based in Japan. Kazuo Terai's co-authors include Hisao Hirota, Shoko Sugiyama, Ichiro Kawase, Tadashi Kuroda, Masatsugu Hori, Yoshimune Hiramoto, Yuichi Oshima, Keiko Yamauchi‐Takihara, Masahiro Izumi and Yoshikazu Nakaoka and has published in prestigious journals such as Circulation, Circulation Research and Molecular and Cellular Biology.

In The Last Decade

Kazuo Terai

11 papers receiving 518 citations

Peers

Kazuo Terai
Nakon Aroonsakool United States
Traian S. Lupu United States
Eto Y Japan
Olga Zhelyabovska United States
Melissa Bevard United States
Nicole E. Hastings United States
Kazuo Terai
Citations per year, relative to Kazuo Terai Kazuo Terai (= 1×) peers Shoko Sugiyama

Countries citing papers authored by Kazuo Terai

Since Specialization
Citations

This map shows the geographic impact of Kazuo Terai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kazuo Terai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kazuo Terai more than expected).

Fields of papers citing papers by Kazuo Terai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kazuo Terai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kazuo Terai. The network helps show where Kazuo Terai may publish in the future.

Co-authorship network of co-authors of Kazuo Terai

This figure shows the co-authorship network connecting the top 25 collaborators of Kazuo Terai. A scholar is included among the top collaborators of Kazuo Terai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kazuo Terai. Kazuo Terai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Izumi, Masahiro, Mitsuru Masaki, Yoshimune Hiramoto, et al.. (2006). Cross-talk between bone morphogenetic protein 2 and leukemia inhibitory factor through ERK 1/2 and Smad1 in protection against doxorubicin-induced injury of cardiomyocytes. Journal of Molecular and Cellular Cardiology. 40(2). 224–233. 21 indexed citations
2.
Kuroda, Tadashi, Hisao Hirota, Mitsuru Masaki, et al.. (2005). Sildenafil as Adjunct Therapy to High-Dose Epoprostenol in a Patient with Pulmonary Veno-Occlusive Disease. Heart Lung and Circulation. 15(2). 139–142. 22 indexed citations
3.
Masaki, Mitsuru, Masahiro Izumi, Tadashi Kuroda, et al.. (2005). Smad1 Protects Cardiomyocytes From Ischemia-Reperfusion Injury. Journal of Cardiac Failure. 11(9). S271–S271. 1 indexed citations
4.
Masaki, Mitsuru, Masahiro Izumi, Yuichi Oshima, et al.. (2005). Smad1 Protects Cardiomyocytes From Ischemia-Reperfusion Injury. Circulation. 111(21). 2752–2759. 54 indexed citations
5.
Terai, Kazuo, Yoshimune Hiramoto, Shoko Sugiyama, et al.. (2005). AMP-Activated Protein Kinase Protects Cardiomyocytes against Hypoxic Injury through Attenuation of Endoplasmic Reticulum Stress. Molecular and Cellular Biology. 25(21). 9554–9575. 310 indexed citations
6.
Masaki, Mitsuru, Tadashi Kuroda, Naoki Hosen, et al.. (2004). Solitary right ventricle metastasis by renal cell carcinoma. Journal of the American Society of Echocardiography. 17(4). 397–398. 14 indexed citations
7.
Ito, Hiroshi, Kazuo Terai, Katsuomi Iwakura, Ichiro Kawase, & Kenshi Fujii. (2004). Hemodynamics of microvascular dysfunction in patients with anterior wall acute myocardial infarction. The American Journal of Cardiology. 94(2). 209–212. 12 indexed citations
9.
Takeda, Yoshihiro, Etsuo Tsuchikane, Tomoko Kobayashi, et al.. (2003). Effect of plaque debulking before stent implantation on in-stent neointimal proliferation: A serial 3-dimensional intravascular ultrasound study. American Heart Journal. 146(1). 175–182. 9 indexed citations
10.
Nagai, Hiroyuki, Yuichi Oshima, Hisao Hirota, et al.. (2003). Specific Cardiomyopathy Caused by Multisystemic Lipid Storage in Jordans' Anomaly. Internal Medicine. 42(7). 587–590. 3 indexed citations
11.
Nakaoka, Yoshikazu, Keigo Nishida, Yasushi Fujio, et al.. (2003). Activation of gp130 Transduces Hypertrophic Signal Through Interaction of Scaffolding/Docking Protein Gab1 With Tyrosine Phosphatase SHP2 in Cardiomyocytes. Circulation Research. 93(3). 221–229. 78 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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