Kathrin L. Engisch

752 citations
31 papers · 602 indexed · h-index 14

Kathrin L. Engisch

30 papers receiving 594 citations

Peers

Kathrin L. Engisch
Comparison fields: 5 of 72
  • Cellular and Molecular Neuroscience 296
  • Cell Biology 197
  • Molecular Biology 423
  • Physiology 18
  • Aging 5
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Julia Preobraschenski Germany
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Kathrin L. Engisch relative to Julia Preobraschenski Germany Julia Preobraschenski's profile →
Citations per field
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Citations per year

Countries citing papers authored by Kathrin L. Engisch

Since Specialization
Citations

This map shows the geographic impact of Kathrin L. Engisch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kathrin L. Engisch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kathrin L. Engisch more than expected).

Fields of papers citing papers by Kathrin L. Engisch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kathrin L. Engisch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kathrin L. Engisch. The network helps show where Kathrin L. Engisch may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Kathrin L. Engisch, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Kathrin L. Engisch Line = papers co-authored together Kathrin L. Engisch links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20252
2 20231
3 20226
4 20223
5 202013
6 20115
7 200813
8 200617
9 200547
10 20059
11 200528
12 200442
13 200331
14 19991
15 1998127
16 199612
17 199033
18
Human pharmacokinetics, excretion, and metabolism of the anthracycline antibiotic menogaril (7-OMEN, NSC 269148) and their correlation with clinical toxicities.
198652
19 19867
20
Human Pharmacokinetics and Correlation With Clinical Toxicities of the Anthracycline Antibiotic Menogaril (7-Omen)
19853

About Kathrin L. Engisch

Kathrin L. Engisch is a scholar working on Cellular and Molecular Neuroscience, Cell Biology and Molecular Biology, having authored 31 papers that have together received 602 indexed citations. Recurring topics across this work include Ion channel regulation and function (15 papers), Neuroscience and Neuropharmacology Research (14 papers), Neuroscience and Neural Engineering (8 papers), Cellular transport and secretion (7 papers), Nicotinic Acetylcholine Receptors Study (5 papers), Cancer therapeutics and mechanisms (3 papers), Neural dynamics and brain function (3 papers) and Photoreceptor and optogenetics research (3 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (296 citations), Cell Biology (197 citations) and Molecular Biology (423 citations). Kathrin L. Engisch has collaborated with scholars based in United States, Mexico and Canada. Frequent co-authors include Martha C. Nowycky, Mark M. Rich, Xueyong Wang, Gerald D. Fischbach, Gary L. Wenk, Martin J. Pinter, Timothy C. Cope, Qingbo Wang, Merrill J. Egorin and David A. Van Echo. Their work appears in journals such as Journal of Neuroscience, The Journal of Physiology and Journal of Neurophysiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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