Karl Agger
- Molecular Biology top 2%
- Genetics top 5%
- Cancer Research top 5%
- Oncology top 10%
- Immunology top 10%
- Co-authors
- Kristian HelinPaul A. CloosJens Peter ChristensenJesper Frank ChristensenJuri RappsilberDiego PasiniKlaus HansenSimon Rose
- Topics
- Epigenetics and DNA Methylation (20 papers)Cancer-related gene regulation (10 papers)Histone Deacetylase Inhibitors Research (8 papers)
- Partner nations
- DenmarkItalyUnited Kingdom
In The Last Decade
Karl Agger
24 papers receiving 4.3k citations
Hit Papers
Peers
Comparison fields: 5 of 111
- Molecular Biology 3.9k
- Genetics 551
- Cancer Research 484
- Oncology 328
- Immunology 246
Countries citing papers authored by Karl Agger
This map shows the geographic impact of Karl Agger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Karl Agger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Karl Agger more than expected).
Fields of papers citing papers by Karl Agger
This network shows the impact of papers produced by Karl Agger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Karl Agger. The network helps show where Karl Agger may publish in the future.
Co-authorship network of co-authors of Karl Agger
This figure shows the co-authorship network connecting the top 25 collaborators of Karl Agger. A scholar is included among the top collaborators of Karl Agger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Karl Agger. Karl Agger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 6 | |
| 3 | 1 | |
| 4 | 2 | |
| 5 | 19 | |
| 6 | 84 | |
| 7 | 187 | |
| 8 | 58 | |
| 9 | 359 | |
| 10 | 72 | |
| 11 | 333 | |
| 12 | 244 | |
| 13 | 36 | |
| 14 | Erasing the methyl mark: histone demethylases at the center of cellular differentiation and diseasebreakdown → | 521 |
| 15 | 13 | |
| 16 | 416 | |
| 17 | UTX and JMJD3 are histone H3K27 demethylases involved in HOX gene regulation and developmentbreakdown → | 1027 |
| 18 | The putative oncogene GASC1 demethylates tri- and dimethylated lysine 9 on histone H3breakdown → | 568 |
| 19 | 96 | |
| 20 | 35 |
About Karl Agger
Karl Agger is a scholar working on Aging, Molecular Biology and Genetics, having authored 25 papers that have together received 4.4k indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (20 papers), Cancer-related gene regulation (10 papers) and Histone Deacetylase Inhibitors Research (8 papers). The work is most often cited by research in Molecular Biology (3.9k citations), Aging (91 citations) and Cancer Research (484 citations). Karl Agger has collaborated with scholars based in Denmark, Italy and United Kingdom. Frequent co-authors include Kristian Helin, Paul A. Cloos, Jens Peter Christensen, Jesper Frank Christensen, Juri Rappsilber, Diego Pasini, Klaus Hansen, Simon Rose, Anna Elisabetta Salcini and Jonas W. Højfeldt. Their work appears in journals such as Nature, Cell and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.