Kaisa Granfors
- Rheumatology top 0.2%
- Immunology top 2%
- Genetics top 1%
- Hematology top 1%
- Molecular Biology top 10%
- Co-authors
- Auli ToivanenSirpa JalkanenPaavo ToivanenR. Lahesmaa-RantalaMatti K. ViljanenR Merilahti-PaloO. Mäki‐IkolaMarjatta Leirisalo‐Repo
- Topics
- Spondyloarthritis Studies and Treatments (59 papers)Yersinia bacterium, plague, ectoparasites research (59 papers)Rheumatoid Arthritis Research and Therapies (55 papers)
- Cited by
- RheumatologyHematologyImmunology
- Partner nations
- FinlandUnited StatesGermany
In The Last Decade
Kaisa Granfors
181 papers receiving 5.0k citations
Peers
Comparison fields: 5 of 110
- Rheumatology 2.4k
- Immunology 1.5k
- Genetics 1.4k
- Hematology 882
- Molecular Biology 840
Countries citing papers authored by Kaisa Granfors
This map shows the geographic impact of Kaisa Granfors's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kaisa Granfors with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kaisa Granfors more than expected).
Fields of papers citing papers by Kaisa Granfors
This network shows the impact of papers produced by Kaisa Granfors. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kaisa Granfors. The network helps show where Kaisa Granfors may publish in the future.
Co-authorship network of co-authors of Kaisa Granfors
This figure shows the co-authorship network connecting the top 25 collaborators of Kaisa Granfors. A scholar is included among the top collaborators of Kaisa Granfors based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kaisa Granfors. Kaisa Granfors is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 9 | |
| 3 | 15 | |
| 4 | 45 | |
| 5 | 35 | |
| 6 | 78 | |
| 7 | 36 | |
| 8 | 60 | |
| 9 | 16 | |
| 10 | 21 | |
| 11 | 59 | |
| 12 | 13 | |
| 13 | 60 | |
| 14 | 23 | |
| 15 | 37 | |
| 16 | 7 | |
| 17 | 10 | |
| 18 | 21 | |
| 19 | 16 | |
| 20 | Evaluation of ELISA in the diagnosis of Yersinia enterocolitica diarrhoea in children. | 4 |
About Kaisa Granfors
Kaisa Granfors is a scholar working on Rheumatology, Pharmacology and Genetics, having authored 183 papers that have together received 5.4k indexed citations. Recurring topics across this work include Spondyloarthritis Studies and Treatments (59 papers), Yersinia bacterium, plague, ectoparasites research (59 papers) and Rheumatoid Arthritis Research and Therapies (55 papers). The work is most often cited by research in Rheumatology (2.4k citations), Hematology (882 citations) and Immunology (1.5k citations). Kaisa Granfors has collaborated with scholars based in Finland, United States and Germany. Frequent co-authors include Auli Toivanen, Sirpa Jalkanen, Paavo Toivanen, R. Lahesmaa-Rantala, Matti K. Viljanen, R Merilahti-Palo, O. Mäki‐Ikola, Marjatta Leirisalo‐Repo, R. Saario and Juha Kirveskari. Their work appears in journals such as Nature, New England Journal of Medicine and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.