Kaichun Wei

1.1k total citations
17 papers, 877 citations indexed

About

Kaichun Wei is a scholar working on Molecular Biology, Physiology and Genetics. According to data from OpenAlex, Kaichun Wei has authored 17 papers receiving a total of 877 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Physiology and 6 papers in Genetics. Recurrent topics in Kaichun Wei's work include DNA Repair Mechanisms (6 papers), Genetic Mapping and Diversity in Plants and Animals (6 papers) and Genetic factors in colorectal cancer (5 papers). Kaichun Wei is often cited by papers focused on DNA Repair Mechanisms (6 papers), Genetic Mapping and Diversity in Plants and Animals (6 papers) and Genetic factors in colorectal cancer (5 papers). Kaichun Wei collaborates with scholars based in Japan, United States and Spain. Kaichun Wei's co-authors include Winfried Edelmann, Tchaiko Parris, Raju Kucherlapati, Thomas A. Kunkel, Allan Clark, Matthew D. Scharff, Ziqiang Li, Alberto Martín, Caroline J. Woo and Philip D. Bardwell and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Kaichun Wei

16 papers receiving 866 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kaichun Wei Japan 12 658 262 174 145 127 17 877
Marcin M. Gorski Italy 11 887 1.3× 108 0.4× 219 1.3× 70 0.5× 64 0.5× 15 1.1k
Brenda K. Shell United States 10 1.0k 1.6× 144 0.5× 265 1.5× 60 0.4× 159 1.3× 10 1.2k
Helena M. Yoder United States 7 1.1k 1.6× 217 0.8× 169 1.0× 59 0.4× 80 0.6× 7 1.2k
Huajun Yan United States 11 872 1.3× 112 0.4× 88 0.5× 94 0.6× 54 0.4× 13 1.0k
Emmanuel Tubacher France 8 465 0.7× 111 0.4× 170 1.0× 73 0.5× 131 1.0× 11 708
Sonja Schaetzlein United States 11 706 1.1× 66 0.3× 115 0.7× 111 0.8× 88 0.7× 15 1.1k
Shujuan J. Xia United States 13 583 0.9× 102 0.4× 170 1.0× 34 0.2× 57 0.4× 17 818
M.H. Goyns United Kingdom 12 322 0.5× 151 0.6× 81 0.5× 45 0.3× 117 0.9× 40 724
Wiljo J. F. de Leeuw Netherlands 9 405 0.6× 247 0.9× 236 1.4× 25 0.2× 136 1.1× 15 828
Paula Gutierrez‐Martinez Spain 10 763 1.2× 37 0.1× 138 0.8× 67 0.5× 106 0.8× 12 1.0k

Countries citing papers authored by Kaichun Wei

Since Specialization
Citations

This map shows the geographic impact of Kaichun Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kaichun Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kaichun Wei more than expected).

Fields of papers citing papers by Kaichun Wei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kaichun Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kaichun Wei. The network helps show where Kaichun Wei may publish in the future.

Co-authorship network of co-authors of Kaichun Wei

This figure shows the co-authorship network connecting the top 25 collaborators of Kaichun Wei. A scholar is included among the top collaborators of Kaichun Wei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kaichun Wei. Kaichun Wei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Chen, Chun-Chin, Elena Avdievich, Yongwei Zhang, et al.. (2017). EXO1 suppresses double-strand break induced homologous recombination between diverged sequences in mammalian cells. DNA repair. 57. 98–106. 12 indexed citations
2.
Yanez, Diana A., Berta Terré, Lluís Palenzuela, et al.. (2015). EXO1 is critical for embryogenesis and the DNA damage response in mice with a hypomorphicNbs1allele. Nucleic Acids Research. 43(15). 7371–7387. 14 indexed citations
3.
Schaetzlein, Sonja, Richard Chahwan, Elena Avdievich, et al.. (2013). MammalianExo1encodes both structural and catalytic functions that play distinct roles in essential biological processes. Proceedings of the National Academy of Sciences. 110(27). E2470–9. 55 indexed citations
4.
Schaetzlein, Sonja, Zhenyu Ju, André Lechel, et al.. (2007). Exonuclease-1 Deletion Impairs DNA Damage Signaling and Prolongs Lifespan of Telomere-Dysfunctional Mice. Cell. 131(1). 190–190. 2 indexed citations
5.
Kose, Hiroyuki, Tohru Sakai, Shin‐ichi Tsukumo, et al.. (2007). Maturational arrest of thymocyte development is caused by a deletion in the receptor-like protein tyrosine phosphatase κ gene in LEC rats. Genomics. 89(6). 673–677. 19 indexed citations
6.
Schaetzlein, Sonja, Zhenyu Ju, André Lechel, et al.. (2007). Exonuclease-1 Deletion Impairs DNA Damage Signaling and Prolongs Lifespan of Telomere-Dysfunctional Mice. Cell. 130(5). 863–877. 115 indexed citations
7.
Bardwell, Philip D., Caroline J. Woo, Kaichun Wei, et al.. (2004). Altered somatic hypermutation and reduced class-switch recombination in exonuclease 1–mutant mice. Nature Immunology. 5(2). 224–229. 202 indexed citations
8.
Wei, Kaichun, Allan Clark, Michael Kane, et al.. (2003). Inactivation of Exonuclease 1 in mice results in DNA mismatch repair defects, increased cancer susceptibility, and male and female sterility. Genes & Development. 17(5). 603–614. 261 indexed citations
9.
Wei, Kaichun, Raju Kucherlapati, & Winfried Edelmann. (2002). Mouse models for human DNA mismatch-repair gene defects. Trends in Molecular Medicine. 8(7). 346–353. 100 indexed citations
10.
Yamada, Takahisa, Takeshi Miyake, Kenkichi Sugiura, et al.. (2001). Identification of Epistatic Interactions Involved in Non-Insulin-Dependent Diabetes Mellitus in the Otsuka Long-Evans Tokushima Fatty Rat.. EXPERIMENTAL ANIMALS. 50(2). 115–123. 2 indexed citations
11.
Wei, Suwen, Kaichun Wei, Daniel H. Moralejo, et al.. (2000). Genetic Evidence for Obesity Loci Involved in the Regulation of Body Fat Distribution in Obese Type 2 Diabetes Rat, OLETF. Genomics. 70(1). 19–25. 24 indexed citations
12.
Sugiura, Kenkichi, Takeshi Miyake, Yukio Taniguchi, et al.. (1999). Identification of novel non-insulin-dependent diabetes mellitus susceptibility loci in the Otsuka Long-Evans Tokushima Fatty rat by MQM-mapping method. Mammalian Genome. 10(12). 1126–1131. 26 indexed citations
13.
Moralejo, Daniel H., Suwen Wei, Kaichun Wei, et al.. (1999). Identification of quantitative trait loci for non-insulin-dependent diabetes mellitus that interact with body weight in the Otsuka Long-Evans Tokushima Fatty rat.. PubMed. 110(6). 545–58. 24 indexed citations
14.
15.
Moralejo, Daniel H., Kenichi Sogawa, Kaichun Wei, et al.. (1998). Mapping of the Gene for Rat Telomerase Protein Component 1(Tlp1) to Chromosome 15.. EXPERIMENTAL ANIMALS. 47(2). 141–142.
16.
Moralejo, Daniel H., Min Zhu, Suwen Wei, et al.. (1998). Identification of a Quantitative Trait Locus Influencing Plasma Insulin Levels after 70% Pancreatectomy Using the OLETF Rat.. Journal of Veterinary Medical Science. 60(10). 1157–1160. 1 indexed citations
17.
Moralejo, Daniel H., Suwen Wei, Kaichun Wei, Takahisa Yamada, & Kozo Matsumoto. (1998). X-Linked Locus Is Responsible for Non-Insulin-Dependent Diabetes Mellitus in the OLETF Rat.. Journal of Veterinary Medical Science. 60(3). 373–375. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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