K S Kosik

3.1k total citations · 1 hit paper
16 papers, 2.1k citations indexed

About

K S Kosik is a scholar working on Cell Biology, Physiology and Molecular Biology. According to data from OpenAlex, K S Kosik has authored 16 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Cell Biology, 8 papers in Physiology and 7 papers in Molecular Biology. Recurrent topics in K S Kosik's work include Alzheimer's disease research and treatments (6 papers), Microtubule and mitosis dynamics (6 papers) and Dementia and Cognitive Impairment Research (3 papers). K S Kosik is often cited by papers focused on Alzheimer's disease research and treatments (6 papers), Microtubule and mitosis dynamics (6 papers) and Dementia and Cognitive Impairment Research (3 papers). K S Kosik collaborates with scholars based in United States, Belgium and Colombia. K S Kosik's co-authors include D. J. Selkoe, Catharine Joachim, Nobuyuki Nukina, Lawrence K. Duffy, Carmela R. Abraham, Steven M. Greenberg, Wei Qiao Qiu, Edward H. Koo, Anil Amaratunga and Richard E. Fine and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Neurology.

In The Last Decade

K S Kosik

16 papers receiving 2.1k citations

Hit Papers

Microtubule-associated protein tau (tau) is a major antig... 1986 2026 1999 2012 1986 250 500 750 1000

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
K S Kosik United States 11 1.4k 1.1k 536 484 250 16 2.1k
Lucia Pastorino United States 23 1.1k 0.8× 1.4k 1.3× 315 0.6× 596 1.2× 354 1.4× 31 2.3k
Inge Grundke-Iqbal United States 8 983 0.7× 844 0.8× 249 0.5× 387 0.8× 247 1.0× 8 1.6k
Melvin L. Billingsley United States 22 868 0.6× 1.0k 1.0× 302 0.6× 694 1.4× 202 0.8× 46 2.2k
Sabiha Khatoon United States 16 1.4k 1.1× 907 0.8× 279 0.5× 430 0.9× 386 1.5× 36 2.1k
Tsuneo Yamazaki Japan 23 1.7k 1.3× 1.0k 0.9× 460 0.9× 419 0.9× 292 1.2× 53 2.5k
Graham Gibb United Kingdom 18 1.1k 0.8× 810 0.8× 250 0.5× 442 0.9× 239 1.0× 29 1.6k
Paul Fraser Canada 15 1.4k 1.0× 879 0.8× 481 0.9× 420 0.9× 348 1.4× 18 1.8k
Malika Hamdane France 29 1.4k 1.0× 1.2k 1.1× 303 0.6× 698 1.4× 364 1.5× 57 2.7k
Karsten Stamer Germany 4 1.1k 0.8× 864 0.8× 504 0.9× 491 1.0× 214 0.9× 7 1.7k
Pascal Kienlen‐Campard Belgium 27 1.4k 1.1× 1.3k 1.2× 231 0.4× 503 1.0× 320 1.3× 68 2.3k

Countries citing papers authored by K S Kosik

Since Specialization
Citations

This map shows the geographic impact of K S Kosik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K S Kosik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K S Kosik more than expected).

Fields of papers citing papers by K S Kosik

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K S Kosik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K S Kosik. The network helps show where K S Kosik may publish in the future.

Co-authorship network of co-authors of K S Kosik

This figure shows the co-authorship network connecting the top 25 collaborators of K S Kosik. A scholar is included among the top collaborators of K S Kosik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K S Kosik. K S Kosik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Fox‐Fuller, Joshua T., Alice Cronin‐Golomb, Jennifer R. Gatchel, et al.. (2019). Biological and Cognitive Markers of Presenilin1 E280A Autosomal Dominant Alzheimer's Disease: A Comprehensive Review of the Colombian Kindred. The Journal of Prevention of Alzheimer s Disease. 6(2). 112–120. 32 indexed citations
2.
Chen, Kewei, Yakeel T. Quiroz, Laura Jakimovich, et al.. (2012). O1‐01‐04: Age‐associated trajectories of biomarkers in early‐onset Alzheimer's disease, for the Alzheimer's Prevention Initiative. Alzheimer s & Dementia. 8(4S_Part_2). 1 indexed citations
3.
Kumar, Pravir, Rashmi K. Ambasta, Kenneth M. Rosen, et al.. (2007). CHIP and HSPs interact with β-APP in a proteasome-dependent manner and influence Aβ metabolism. Human Molecular Genetics. 16(7). 848–864. 129 indexed citations
4.
Neumann, Peter J., et al.. (2001). Measuring Alzheimer’s disease progression with transition probabilities. Neurology. 57(6). 957–964. 90 indexed citations
5.
Kosik, K S. (1998). Neurolab: learning how the nervous system adapts to microgravity.. PubMed. 1(5). 36–8. 2 indexed citations
6.
Flanagan, Lisa A., Casey Cunningham, Jianjun Chen, et al.. (1997). The structure of divalent cation-induced aggregates of PIP2 and their alteration by gelsolin and tau. Biophysical Journal. 73(3). 1440–1447. 72 indexed citations
7.
Leclerc, Nicole, Peter W. Baas, Craig C. Garner, & K S Kosik. (1996). Juvenile and mature MAP2 isoforms induce distinct patterns of process outgrowth.. Molecular Biology of the Cell. 7(3). 443–455. 38 indexed citations
8.
Kosik, K S, et al.. (1996). When Someone You Love Has Alzheimer's: The Caregiver's Journey. Medical Entomology and Zoology. 2 indexed citations
9.
Greenberg, Steven M., Edward H. Koo, D. J. Selkoe, Wei Qiao Qiu, & K S Kosik. (1994). Secreted beta-amyloid precursor protein stimulates mitogen-activated protein kinase and enhances tau phosphorylation.. Proceedings of the National Academy of Sciences. 91(15). 7104–7108. 125 indexed citations
10.
Amaratunga, Anil, Peter J. Morin, K S Kosik, & Richard E. Fine. (1993). Inhibition of kinesin synthesis and rapid anterograde axonal transport in vivo by an antisense oligonucleotide. Journal of Biological Chemistry. 268(23). 17427–17430. 59 indexed citations
11.
Lee, V M, et al.. (1991). Microtubule destabilization and neurofilament phosphorylation precede dendritic sprouting after close axotomy of lamprey central neurons.. Proceedings of the National Academy of Sciences. 88(11). 5016–5020. 41 indexed citations
12.
Kosik, K S, et al.. (1987). Type II CaS /calmodulin-dependent kinase phosphorylates tau protein in the region of the mouse repeat. Fed. Proc., Fed. Am. Soc. Exp. Biol.; (United States). 1 indexed citations
13.
Nukina, Nobuyuki, K S Kosik, & D. J. Selkoe. (1987). Recognition of Alzheimer paired helical filaments by monoclonal neurofilament antibodies is due to crossreaction with tau protein.. Proceedings of the National Academy of Sciences. 84(10). 3415–3419. 178 indexed citations
14.
Kosik, K S, Catharine Joachim, & D. J. Selkoe. (1986). Microtubule-associated protein tau (tau) is a major antigenic component of paired helical filaments in Alzheimer disease.. Proceedings of the National Academy of Sciences. 83(11). 4044–4048. 1142 indexed citations breakdown →
15.
Kosik, K S, et al.. (1984). Microtubule-associated protein 2: monoclonal antibodies demonstrate the selective incorporation of certain epitopes into Alzheimer neurofibrillary tangles.. Proceedings of the National Academy of Sciences. 81(24). 7941–7945. 187 indexed citations
16.
Furie, Bruce & K S Kosik. (1981). Reply. Annals of Neurology. 9(6). 620–620. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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