Jürgen Deka

727 citations
9 papers · 586 · h-index 8

Impact in

Papers in

    • Wnt/β-catenin signaling in development and cancer 4
    • Cancer-related gene regulation 2
    • Melanoma and MAPK Pathways 1
    • Cancer Cells and Metastasis 2
    • Cancer-related Molecular Pathways 1

Jürgen Deka

9 papers receiving 579 citations

Peers

Jürgen Deka
Comparison fields: 5 of 64
  • Immunology and Allergy 119
  • Cancer Research 111
  • Molecular Biology 408
  • Immunology 124
  • Cell Biology 90
Replace Christa Prange with:
Christa Prange United States
M Fukumoto Japan
Kyle Draheim United States
Anne Vehlow Germany
C. Botta Italy
Karina Petat‐Dutter Germany
Jane R. Giblin United States
Lucinda F. Reynolds United States
Kristina Holmqvist Sweden
Egle Balciunaite United States
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Citations per field
00.5×4.8×
Christa Prange · 1×
Citations per year

Countries citing papers authored by Jürgen Deka

Since Specialization
Citations

This map shows the geographic impact of Jürgen Deka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jürgen Deka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jürgen Deka more than expected).

Fields of papers citing papers by Jürgen Deka

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jürgen Deka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jürgen Deka. The network helps show where Jürgen Deka may publish in the future.

Co-authors

The 25 scholars most cited alongside Jürgen Deka, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jürgen Deka Line = papers co-authored together Jürgen Deka links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 2010102
2 200399
3 200283
4 200975
5 199872
6 200466
7 201544
8 199940
9 20195

About Jürgen Deka

Jürgen Deka is a scholar working on Molecular Biology, Oncology, Immunology and Allergy, Pathology and Forensic Medicine and Radiology, Nuclear Medicine and Imaging, having authored 9 papers that have together received 586 indexed citations. Recurring topics across this work include Wnt/β-catenin signaling in development and cancer (4 papers), Cell Adhesion Molecules Research (3 papers), Genetic factors in colorectal cancer (2 papers), Cancer Cells and Metastasis (2 papers), Cancer-related gene regulation (2 papers), Monoclonal and Polyclonal Antibodies Research (2 papers), Cancer-related Molecular Pathways (1 paper) and Melanoma and MAPK Pathways (1 paper). The work is most often cited by research in Immunology and Allergy (119 citations), Cancer Research (111 citations), Molecular Biology (408 citations), Immunology (124 citations) and Cell Biology (90 citations). Jürgen Deka has collaborated with scholars based in United Kingdom, Switzerland and Germany. Frequent co-authors include Ann Ager, Anne J. Ridley, Aleksandar Ívetic, Oliver Müller, Jürgen Kuhlmann, Michel Aguet, Sven Eyckerman, Oliver Florey, Monica Belich and V. S. Lang. Their work appears in journals such as Journal of Biological Chemistry, European Journal of Biochemistry, Frontiers in Immunology, EBioMedicine and Oncogene.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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