Claudio Cantù

1.9k total citations
47 papers, 1.2k citations indexed

About

Claudio Cantù is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Claudio Cantù has authored 47 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 42 papers in Molecular Biology, 6 papers in Oncology and 6 papers in Genetics. Recurrent topics in Claudio Cantù's work include Wnt/β-catenin signaling in development and cancer (20 papers), Cancer-related gene regulation (16 papers) and RNA Research and Splicing (12 papers). Claudio Cantù is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (20 papers), Cancer-related gene regulation (16 papers) and RNA Research and Splicing (12 papers). Claudio Cantù collaborates with scholars based in Sweden, Switzerland and Italy. Claudio Cantù's co-authors include Konrad Basler, George Hausmann, Tomáš Valenta, Dario Zimmerli, Michel Aguet, Andreas E. Moor, Nikolaos Doumpas, Matthias B. Moor, Colm E. Nestor and Antonio Lentini and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Claudio Cantù

42 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Claudio Cantù Sweden 22 946 274 209 145 113 47 1.2k
Huifei Liu United States 12 807 0.9× 119 0.4× 135 0.6× 176 1.2× 203 1.8× 29 1.2k
Lucia Micale Italy 20 652 0.7× 117 0.4× 279 1.3× 125 0.9× 217 1.9× 59 1.1k
Reinhard Grausenburger Austria 14 702 0.7× 181 0.7× 92 0.4× 111 0.8× 126 1.1× 25 1.1k
W. Patrick Devine United States 11 658 0.7× 231 0.8× 72 0.3× 163 1.1× 112 1.0× 26 1.2k
D. Spencer Currle United States 11 677 0.7× 356 1.3× 195 0.9× 167 1.2× 46 0.4× 17 1.1k
Jixiang Ding United States 18 1.4k 1.4× 146 0.5× 415 2.0× 117 0.8× 150 1.3× 34 1.7k
Seung Tae Baek South Korea 17 966 1.0× 166 0.6× 196 0.9× 193 1.3× 77 0.7× 29 1.4k
Li‐Wei Chang United States 20 823 0.9× 107 0.4× 119 0.6× 275 1.9× 107 0.9× 31 1.3k
Mirella Tanori Italy 19 708 0.7× 218 0.8× 109 0.5× 98 0.7× 42 0.4× 45 1.2k
Marta Rosário Germany 14 1.2k 1.3× 313 1.1× 132 0.6× 106 0.7× 296 2.6× 26 1.6k

Countries citing papers authored by Claudio Cantù

Since Specialization
Citations

This map shows the geographic impact of Claudio Cantù's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Claudio Cantù with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Claudio Cantù more than expected).

Fields of papers citing papers by Claudio Cantù

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Claudio Cantù. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Claudio Cantù. The network helps show where Claudio Cantù may publish in the future.

Co-authorship network of co-authors of Claudio Cantù

This figure shows the co-authorship network connecting the top 25 collaborators of Claudio Cantù. A scholar is included among the top collaborators of Claudio Cantù based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Claudio Cantù. Claudio Cantù is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rovelli, Grazia, Francesca Gullo, Sergio Ottolenghi, et al.. (2025). SOX2 and NR2F1 coordinate the gene expression program of the early postnatal visual thalamus. Biology Open. 14(8).
2.
3.
Chakraborty, Chaitali, et al.. (2025). Wnt signaling activation induces CTCF binding and loop formation at cis-regulatory elements of target genes. Genome Research. 35(8). 1701–1716. 1 indexed citations
4.
5.
Moparthi, Lavanya, et al.. (2024). The tumor suppressor p53 is a negative regulator of the carcinoma-associated transcription factor FOXQ1. Journal of Biological Chemistry. 300(4). 107126–107126. 4 indexed citations
6.
Pagella, Pierfrancesco, et al.. (2024). An unexpected role of Nogo-A as regulator of tooth enamel formation. International Journal of Oral Science. 16(1). 60–60.
7.
Pastori, Valentina, et al.. (2024). Transcriptional repression of the oncofetal LIN28B gene by the transcription factor SOX6. Scientific Reports. 14(1). 10287–10287. 1 indexed citations
8.
Debbache, Julien, Phil F. Cheng, Mitchell P. Levesque, et al.. (2024). TGFβ signaling sensitizes MEKi-resistant human melanoma to targeted therapy-induced apoptosis. Cell Death and Disease. 15(12). 925–925. 2 indexed citations
9.
Kugelberg, Unn, et al.. (2023). Stress‐sensitive dynamics of miRNAs and Elba1 in Drosophila embryogenesis. Molecular Systems Biology. 19(5). e11148–e11148. 1 indexed citations
10.
Pagella, Pierfrancesco, et al.. (2023). The time-resolved genomic impact of Wnt/β-catenin signaling. Cell Systems. 14(7). 563–581.e7. 7 indexed citations
11.
Jauregi‐Miguel, Amaia, et al.. (2023). Single-cell response to Wnt signaling activation reveals uncoupling of Wnt target gene expression. Experimental Cell Research. 429(2). 113646–113646. 2 indexed citations
12.
Moparthi, Lavanya, et al.. (2022). The oncogenic transcription factor FOXQ1 is a differential regulator of Wnt target genes. Journal of Cell Science. 135(19). 11 indexed citations
13.
Barchiesi, Riccardo, Xu Li, Esi Domi, et al.. (2022). An epigenetic mechanism for over-consolidation of fear memories. Molecular Psychiatry. 27(12). 4893–4904. 10 indexed citations
14.
Baggiolini, Arianna, Damian Dalcher, Luigi Lerra, et al.. (2021). Epigenetic control of melanoma cell invasiveness by the stem cell factor SALL4. Nature Communications. 12(1). 5056–5056. 21 indexed citations
15.
Saxena, Meera, Ravi Kiran Reddy Kalathur, Natalia Rubinstein, et al.. (2020). A Pygopus 2-Histone Interaction Is Critical for Cancer Cell Dedifferentiation and Progression in Malignant Breast Cancer. Cancer Research. 80(17). 3631–3648. 12 indexed citations
16.
Fugazza, Cristina, M. Marini, Maria Franca Marongiu, et al.. (2020). The Coup-TFII orphan nuclear receptor is an activator of the γ-globin gene. Haematologica. 106(2). 474–482. 7 indexed citations
17.
Cantù, Claudio, Lasse D. Jensen, Christiane König, et al.. (2019). Pharmacophore-guided discovery of CDC25 inhibitors causing cell cycle arrest and tumor regression. Scientific Reports. 9(1). 1335–1335. 22 indexed citations
18.
Cantù, Claudio, Anastasia Felker, Dario Zimmerli, et al.. (2018). Mutations in Bcl9 and Pygo genes cause congenital heart defects by tissue-specific perturbation of Wnt/β-catenin signaling. Genes & Development. 32(21-22). 1443–1458. 33 indexed citations
19.
Azim, Kasum, Guillaume Marcy, Andrea Rivera, et al.. (2017). Pharmacogenomic identification of small molecules for lineage specific manipulation of subventricular zone germinal activity. PLoS Biology. 15(3). e2000698–e2000698. 41 indexed citations
20.
Moor, Andreas E., Pascale Anderle, Claudio Cantù, et al.. (2015). BCL9/9L-β-catenin Signaling is Associated With Poor Outcome in Colorectal Cancer. EBioMedicine. 2(12). 1932–1943. 44 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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