Julia J. Inglis

3.0k citations
27 papers · 2.4k indexed · 1 hit paper · h-index 22

Impact in

Papers in

Julia J. Inglis

27 papers receiving 2.4k citations

Hit Papers

Tenascin-C is an endogenous activator of Toll-like receptor 4 that is essential for maintaining inflammation in arthritic joint disease 2009 · 583 citations
5832009202620142020100200300400500

Peers

Julia J. Inglis
Comparison fields: 5 of 108
  • Rheumatology 754
  • Biological Psychiatry 104
  • Immunology and Allergy 244
  • Immunology 804
  • Behavioral Neuroscience 102
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Roland Axmann Germany
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Citations per field
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Citations per year

Countries citing papers authored by Julia J. Inglis

Since Specialization
Citations

This map shows the geographic impact of Julia J. Inglis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Julia J. Inglis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Julia J. Inglis more than expected).

Fields of papers citing papers by Julia J. Inglis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Julia J. Inglis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Julia J. Inglis. The network helps show where Julia J. Inglis may publish in the future.

Co-authors

The 25 scholars most cited alongside Julia J. Inglis, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Julia J. Inglis Line = papers co-authored together Julia J. Inglis links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 201311
2 201153
3 201061
4 201055
5 201014
6 201039
7 201043
8 2010119
9
Tenascin-C is an endogenous activator of Toll-like receptor 4 that is essential for maintaining inflammation in arthritic joint disease
Hit paper breakdown →
2009583
10 200952
11 2009108
12 2008167
13 200897
14 2008159
15 200856
16 200812
17 2007109
18 200760
19 200514
20 200448

About Julia J. Inglis

Julia J. Inglis is a scholar working on Biological Psychiatry, Immunology and Allergy, Physiology, Complementary and Manual Therapy and Rehabilitation, having authored 27 papers that have together received 2.4k indexed citations. Recurring topics across this work include Pain Mechanisms and Treatments (10 papers), Neuropeptides and Animal Physiology (5 papers), Rheumatoid Arthritis Research and Therapies (5 papers), Immunotherapy and Immune Responses (4 papers), Cell Adhesion Molecules Research (4 papers), Exercise and Physiological Responses (3 papers), Inflammatory mediators and NSAID effects (3 papers) and Monoclonal and Polyclonal Antibodies Research (3 papers). The work is most often cited by research in Rheumatology (754 citations), Biological Psychiatry (104 citations), Immunology and Allergy (244 citations), Immunology (804 citations) and Behavioral Neuroscience (102 citations). Julia J. Inglis has collaborated with scholars based in United Kingdom, Australia and United States. Frequent co-authors include Richard Williams, Marc Feldmann, Fiona E. McCann, Gabriel Criado, Fionula M. Brennan, Clare A. Notley, Kay McNamee, Brian M. J. Foxwell, Egle Šimelyte and David W. Essex. Their work appears in journals such as Arthritis Research & Therapy, Neuroscience, Pain, Nature Medicine and Inflammation Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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